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What does high pitched tinnitus sound like, why do i hear ringing in my ears at night - For You

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Tinnitus (pronounced tih-NITE-us or TIN-ih-tus) is sound in the head with no external source. Almost everyone has had tinnitus for a short time after being exposed to extremely loud noise. While there's no cure for chronic tinnitus, it often becomes less noticeable and more manageable over time. Sound waves travel through the ear canal to the middle and inner ear, where hair cells in part of the cochlea help transform sound waves into electrical signals that then travel to the brain's auditory cortex via the auditory nerve. Most people who seek medical help for tinnitus experience it as subjective, constant sound, and most have some degree of hearing loss.
Tinnitus can arise anywhere along the auditory pathway, from the outer ear through the middle and inner ear to the brain's auditory cortex, where it's thought to be encoded (in a sense, imprinted). Most tinnitus is "sensorineural," meaning that it's due to hearing loss at the cochlea or cochlear nerve level. Tinnitus that's continuous, steady, and high-pitched (the most common type) generally indicates a problem in the auditory system and requires hearing tests conducted by an audiologist.
Your general health can affect the severity and impact of tinnitus, so this is also a good time to take stock of your diet, physical activity, sleep, and stress level — and take steps to improve them.
In addition to treating associated problems (such as depression or insomnia), there are several strategies that can help make tinnitus less bothersome. There is no FDA-approved drug treatment for tinnitus, and controlled trials have not found any drug, supplement, or herb to be any more effective than a placebo.
The most effective approaches are behavioral strategies and sound-generating devices, often used in combination. Not all insurance companies cover tinnitus treatments in the same way, so be sure to check your coverage.
For many, it's a ringing sound, while for others, it's whistling, buzzing, chirping, hissing, humming, roaring, or even shrieking.
Things that cause hearing loss (and tinnitus) include loud noise, medications that damage the nerves in the ear (ototoxic drugs), impacted earwax, middle ear problems (such as infections and vascular tumors), and aging. One of the most common causes of tinnitus is damage to the hair cells in the cochlea (see "Auditory pathways and tinnitus").
She or he will take a medical history, give you a physical examination, and do a series of tests to try to find the source of the problem.
Pulsatile tinnitus calls for a thorough evaluation by an otolaryngologist (commonly called an ear, nose, and throat specialist, or ENT) or neurotologist, especially if the noise is frequent or constant. You may also be able to reduce the impact of tinnitus by treating depression, anxiety, insomnia, and pain with medications or psychotherapy. No single approach works for everyone, and you may need to try various combinations of techniques before you find what works for you.
CBT uses techniques such as cognitive restructuring and relaxation to change the way patients think about and respond to tinnitus.

Masking devices, worn like hearing aids, generate low-level white noise (a high-pitched hiss, for example) that can reduce the perception of tinnitus and sometimes also produce residual inhibition — less noticeable tinnitus for a short time after the masker is turned off. Other treatments that have been studied for tinnitus include transcutaneous electrical stimulation of parts of the inner ear by way of electrodes placed on the skin or acupuncture needles, and stimulation of the brain using a powerful magnetic field (a technique called repetitive transcranial magnetic stimulation, or rTMS). Some medications (especially aspirin and other nonsteroidal anti-inflammatory drugs taken in high doses) can cause tinnitus that goes away when the drug is discontinued.
For example, if you have a heart murmur, you may hear a whooshing sound with every heartbeat; your clinician can also hear that sound through a stethoscope. This stimulates abnormal activity in the neurons, which results in the illusion of sound, or tinnitus.
Tinnitus can also be a symptom of Mnire's disease, a disorder of the balance mechanism in the inner ear.
Our bodies normally produce sounds (called somatic sounds) that we usually don't notice because we are listening to external sounds. She or he will also ask you to describe the noise you're hearing (including its pitch and sound quality, and whether it's constant or periodic, steady or pulsatile) and the times and places in which you hear it.
If you have age-related hearing loss, a hearing aid can often make tinnitus less noticeable by amplifying outside sounds.
The aim is to habituate the auditory system to the tinnitus signals, making them less noticeable or less bothersome. Many people can hear their heartbeat — a phenomenon called pulsatile tinnitus — especially as they grow older, because blood flow tends to be more turbulent in arteries whose walls have stiffened with age. The main components of TRT are individual counseling (to explain the auditory system, how tinnitus develops, and how TRT can help) and sound therapy.
Individual studies have reported improvements in as many as 80% of patients with high-pitched tinnitus.
In two small trials, rTMS compared with a sham procedure helped improve the perception of tinnitus in a few patients. Pulsatile tinnitus may be more noticeable at night, when you're lying in bed, because more blood is reaching your head, and there are fewer external sounds to mask the tinnitus.
The resulting electrical noise takes the form of tinnitus — a sound that is high-pitched if hearing loss is in the high-frequency range and low-pitched if it's in the low-frequency range. Tinnitus can be a side effect of many medications, especially when taken at higher doses (see "Some drugs that can cause or worsen tinnitus").
A 2010 review of six studies by the Cochrane Collaboration (an international group of health authorities who evaluate randomized trials) found that after CBT, the sound was no less loud, but it was significantly less bothersome, and patients' quality of life improved. A device is inserted in the ear to generate low-level noise and environmental sounds that match the pitch, volume, and quality of the patient's tinnitus. In a Cochrane review of the one randomized trial that followed Jastreboff's protocol and met the organization's standards, TRT was much more effective in reducing tinnitus severity and disability than a technique called masking (see below).
Many people worry that tinnitus is a sign that they are going deaf or have another serious medical problem, but it rarely is.
If you notice any new pulsatile tinnitus, you should consult a clinician, because in rare cases it is a sign of a tumor or blood vessel damage.

This kind of tinnitus resembles phantom limb pain in an amputee — the brain is producing abnormal nerve signals to compensate for missing input. The first is that, when you kill in the hair cells in your ear using a high noise stimulus (like a rock concert), your brain areas which process sound will remap to make up the difference. The other hypothesis is that losing hearing decreases the inhibition you normally have in your auditory cortex (because it's not getting any sound stimulus to inhibit), and therefore you get more excitatory signals, more firing, and phantom perception of sound. They exposed them to 123 decibels of sound at greater than 4 kilohertz, for more than 7 hours. The net result of this was when they tested the rats to see if they reacted to tones a week later, they still had their low pitch hearing, but nothing in the high range.
They were just fine for lower tones, but for higher tones they had to play the tones at a much higher volume to get any effects.So ok, the rats are deaf in these ranges. They also looked at patterns of activity in the auditory cortex of the animals, and found that the deafened rats had undergone cortical reorganization, with a lot more of their brain taking over the low tones, and less available for the high tones. Both rats did respond, so the hearing lesioned rats are NOT deaf to those tones (which is important, usually even when we have hearing loss like this, it's not total). And the interesting thing is that the deafened animals actually responded MORE to the test sound.
It looks like from the other studies the tone should be high pitched, if the excited neuron firing which they detected was in the areas they thought it was.
In one test, the rats were encouraged to prefer the light side of two chambers (they usually prefer the dark) in response to a specific, higher than 4KHz tone. It doesn't actually prove it, but I think it's is probably as close as they are going to get. So if, after hearing loss, you have less GABA signaling, you have excitatory signaling that is uninhibited, and you might get tinnitus. When the authors gave drugs to the deafened rats that increased GABA signaling and tested them on conditioned place preference again, the rats began to prefer the dark side, suggesting they weren't hearing the high pitched tones in their head anymore.
It's an interesting study, and it looks like, though the brain does remap itself in response to hearing loss, the cause of tinnitus lies in the LOSS of inhibition to auditory cortex neurons. It also lines up with reports in humans, who usually report ringing in their ears at high pitches, rather than low.
There are relatively few people who suffer from CHRONIC tinnitus, but for those who do it can really get you down.
It's possible that these drugs could be tried to treat chronic tinnitus in humans (though no proof on that one yet) and finally get rid of that awful ringing in your ears.Yang S, Weiner BD, Zhang LS, Cho SJ, & Bao S (2011).

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