Diabetes has skyrocketed since the 1980’s and is expected to continue growing rapidly into the future.  Until recently, this increase was attributed almost exclusively to poor dietary habits, limited physical activity, and obesity. Findings like those described above suggest that diabetics, pre-diabetics, and non-diabetics may benefit from reducing levels of this electromagnetic pollution like dirty electricity in their homes.  Doing so may help them better regulate their blood sugar levels.
It may surprise you to hear that the biggest health threat to the US may be electropollution. Kids are increasingly experiencing ADHD, asthma, diabetes, learning disabilities, headaches, sleep disturbances and mood. Learn to easily read a normal blood sugar levels chart, A good grasp and understanding of normal blood sugar levels chart is necessary whether you have diabetes or not. Diabetes blood sugar levels chart: what is a normal blood, Keep in mind that the blood glucose level before a meal for a non diabetic person and a person with prediabetes may be very similar.
Blood glucose levels chart and a normal blood sugar range, A blood glucose levels chart is a handy thing to have access to for deciphering those glucose numbers on a blood test. Normal blood sugar levels chart – buzzle, You must have noticed that the normal blood glucose levels for men and women are slightly different. What is normal blood sugar level – healthiack, The blood sugar concentration or blood glucose level is the amount of glucose (sugar) present in the blood of a human or an animal. Normal blood sugar levels chart mmol – medhelp, Common questions and answers about normal blood sugar levels chart mmol. Home Journals Books Conferences News About Us Jobs Open Journal of Endocrine and Metabolic Diseases Vol.
Following a diet designed to keep blood sugar from rising after meals helped diabetic people keep their disease under control. People with type 2 diabetes who ate what is known as a low-glycemic-index diet for six months had greater blood sugar control and fewer heart disease risk factors than those who followed another eating plan. Both diets were high in fiber and low in saturated fat, and both derived about 40% of their calories from carbohydrates. But the low-glycemic-index diet emphasized carbohydrates that had less impact on blood sugar levels, such as beans, pasta, nuts, and certain whole grains. The basic premise behind the low-glycemic-index diet is that all carbs are not created equal. Specifically, the glycemic index measures how much a 50-gram portion of a carbohydrate raises blood sugar levels compared to pure glucose, which has a glycemic index score of 100. Typically, foods that score higher than 70 are considered high-glycemic-index (GI) foods; those that score 55 and under are considered low-GI foods. Many highly refined foods, including white bread, corn flakes, and instant potatoes have high GI scores; unprocessed, high-fiber foods tend to have lower GI scores. Foods such as carrots and potatoes can either be high-GI or low-GI foods, depending on several factors, including how long they are stored and how they are cooked or processed. Research suggests that the GI response to a given food also varies from person to person and can even vary within the same person from day to day. You can eat low GI foods to control blood sugar levels, and still not losing weight, because low GI does not mean low in calories. The HbA1c test (also called glycosylated haemoglobin level) is a laboratory blood test which measures your average blood glucose over the previous weeks and gives an indication of your longer-term blood glucose control.
Most diabetes specialists and GPs have a lot of confidence in this test and will use it to help show people with type 1 or type 2 diabetes how they are going with their blood glucose management. When the body processes sugar, glucose in the bloodstream naturally attaches to haemoglobin.  The amount of glucose that combines with this protein is directly proportional to the total amount of sugar that is in your system at that time.
Because red blood cells in the human body survive for 8-12 weeks before renewal, measuring glycated haemoglobin (or HbA1c) can be used to reflect average blood glucose levels over that duration, providing a useful longer-term gauge of blood glucose control.  If your blood sugar levels have been high in recent weeks, your HbA1c will also be greater. A major study, the UK Prospective Diabetes (UKPDS) Study* published in 2000, managed to quantify many of the benefits of reducing a high HbA1c level by just 1%. If you are pre-diabetic or diabetic, regular self-monitoring of blood glucose helps you understand how certain foods or food combinations effect your blood glucose level – for the better or worse. If you are struggling with pre-diabetes or diabetes and you would like information about how to manage your blood glucose and HbA1c levels within optimal range, make an appointment to see me and we can work together to start you on your journey to wellness with Real Food and healthy lifestyle choices. Breast Cancer is not, in most cases, fast moving or aggressive.  There are better ways than mammograms to monitor the health of your breasts and things you can do to improve or maintain healthy breast tissues.
If you have been diagnosed with breast cancer, take a breath - you have time to consider all your options.
Sleep Well is a wireless blood glucose monitoring device concept, designed aiming to eliminate the sleepless nights of parents with the tension of getting their kids glucose level down to the extreme level. Design(dot)fr is a place to discover and share web content about design, architecture and decorative art. Diabetes can occur in mother before pregnancy - pregestational diabetes or during pregnancy - gestational diabetes.
The possibility of life-threatening structural anomalies is the most concerned issue in a case of maternal diabetes. However, there is no association of birth defects in offspring born to a diabetic father or in mother who develop diabetes after first trimester of pregnancy.
Hyperglycemia disturbs the development of embryo by decreasing levels of arachidonic acid, myoinositol and accumulation of sorbitol and trace metals. Any newborn weighing more than 4 kg irrespective of gender and age of gestation are macrosomic or large babies.
Birth related injuries (Shoulder dystocia & Brachial plexus injury), asphyxia and increases the need of cesarean deliveries in mother.
It has been seen that impaired glucose control in later 2 trimesters of pregnancy co-relate more with fetal obesity.
Strict blood glucose control in 2nd and 3rd trimester may reduce the incidence of large babies to near normal. Episodic maternal hyperglycemia promotes fetal catabolic state where excessive oxygen utilization occurs.
Therefore, seeking medical attention to attain a normal glycemic state throughout the pregnancy is critical to prevent hazards to both baby and mother.
A birth of a baby with down syndrome is a period of anxiety and breakdown to family members and most to the parents.
Now, there is evidence that exposure to dirty electricity may also be linked to increased blood sugar levels and by extension – diabetes. Magda Havas, a researcher and professor at Trent University in Canada, found that plasma glucose levels of two Type 1 diabetics and two Type 2 diabetics responded directly to the amount of dirty electricity in their environment.  The Type 1 diabetics required less insulin in environments with low levels of dirty electricity. Havas has also analyzed blood sugar data for diabetics in a long-term care facility (Canada) and a clinic (Japan) in which special plug-in filters were installed in outlets to reduce dirty electricity in the buildings.  Of the five diabetics in the long-term care facility, for whom data were available, two (both Type 1 diabetics) had significantly lower fasting plasma glucose levels after the filters were installed. The same is true with potatoes and the riper the fruit or vegetable, the higher the GI score. Eating only low GI will also not help you to reverse Insulin resistance and type 2 diabetes.
By taking the Manna Blood Sugar Support supplement with each meal, you can lower the GI of the food you ate by up to 43%, meaning that you can stay satisfied for longer due to stable blood sugar levels.


The test is used as a regular monitoring tool if you have been diagnosed with diabetes. It may also be used as one of several screening measures in the general population to look for elevated blood glucose levels, which are suggestive of diabetes. Association of glycaemia with macrovascular and microvascular complications of type 2 diabetes (UKDPS 35): prospective observational study. The system comprises a portable and lightweight monitor with an ability to take wireless signal that parents keep with them and a wearable bracelet monitor that remains fastened on the child’s hand to transmit the health condition to the monitor. High blood sugar level in mother is not just harmful to her but also to the baby developing in her womb.
Compared to a mother with normal blood glucose level, a mother with diabetes before pregnancy has 4 to 8 times higher risk of major fetal anomalies. A study done in 1992 showed a clear preponderance of babies weighing more than 4.5 kg in mothers with diabetes compared to non-diabetic mothers.
The girth of abdomen increases significantly due to deposition of fat due to high glucose in fetal circulation. Increased mortality and morbidity has been seen in Infants born to Diabetic Mothers (IDM) in 1st 28 days of life. Polycythemia: Fetal hypoxia stimulates secretion of erythropoietin from kidney, a hormone which has role in hemoglobin synthesis. Hypoglycemia: Low blood sugar level in newborn can occur due to high insulin level causing symptoms like seizure, coma and brain damage during early life.
Jaundice or Hyperbilirubinemia: Prematurity and Polycythemia are the main factors contributing to jaundice in IDM.
Respiratory Distress Syndrome (RDS): Can result from prematurity, surfactant deficiency, fluid retention in lungs (transient tachypnea) and cardiomyopathy. Pediatrician receives the delivery, fetal bradycardia with maternal hypertension was the scenario.
Are these health issues being worsened by the explosion of electronics in our homes and schools?  Seminal research by Dr. Red blood cells have a lifespan of about six weeks and so the test gives a good indication of what your overall blood glucose levels have been throughout that time.
This monitor allows the parents to check their kid’s health without even leaving her bed and can be set to trigger an alarm when the blood sugar level of the child crosses a particular height.
According to studies, the defects mostly involve the brain and spinal cord, followed by heart, genitourinary system and limb defects. Increased Glycated hemoglobin (HbA1c) which can be measured by laboratory blood testing has seen to be highly co-related with development of teratogenesis.
This results in a condition called Polycythemia where there is increased level of hemoglobin in blood. Hence, blood sugar level monitoring is necessary during newborn period and is done as per standard protocol. I often write my views online as well as share few important topics for medical students, doctors and specially parents. Havas has distinguished between true Type 1 and Type 2 diabetes and a third type of diabetes that she calls Type 3 diabetes.  The blood sugar and blood viscosity of Type 3 diabetics are affected by dirty electricity in their environment, while this does not appear to be the case for true Type 1 and Type 2 diabetics. Moreover, parents can easily review their child’s nightly activities during the middle of the night at their bedside convenient. Increased glucose level in mother leads to increased insulin secretion in fetus which leads to storage of excess nutrients. Sam Milham suggests that “dirty electricity” and other electrical energy may be disproportionally affecting children. If an emergency situation takes place, they can rush to their child’s room with the monitor to perform the aid that has to be taken based on the information displayed. The pain killer effect of aspirin is best known for its effects on the two cyclooxygenase enzymes (COX1 & COX2), but, recently, aspirin could specifically inhibit the protein I-kappa-?-kinase beta (IKK-beta). This kinase is used for its role in the cascade of signals that activate the nuclear factor kappa-b (NF-kappa-B) family of cellular genes which regulate inflammatory and immune responses. Now, it turns out that IKK-beta also works in another pathway to contribute to insulin resistance by interfering with insulin signaling. Objective: In view of the recent rodent data demonstrating a potentially important role of IKK? in mediating insulin resistance and the ability of salicylates to inhibit IKK? activity, we decided to examine the role of different doses of aspirin (low, moderate and high) in experimentally induced diabetic rats.
Groups from the fourth to the six consist of 20 diabetic induced rats and further subdivided  into rats taking either aspirin alone in different doses (low, moderate or high) or aspirin and insulin. At the end of the protocol, fasting blood sugar level (FBS), glycosylated hemoglobin (HBA1c%), total serum proteins, C-peptide, lipid profile and C-reactive proteins were measured. Results: Different doses of aspirin showed that moderate and to a greater extent high dose aspirin administration to diabetic rats have greater impact on fasting blood glucose levels whether treated with insulin or not. Again, HBA1c% in diabetic rats treated with insulin and receiving HDA was lower than diabetic rats treated with insulin only or even taking LDA in addition. On the contrary, different doses of aspirin (LDA, MDA&HDA) administration to diabetic rats have no any influence on HBA1c% as compared to normal non-diabetic rats. TGs in diabetic rats receiving MDA alone was elevated as compared to normal non-diabetic rats.
Again, moderate and HDA in diabetic rats not taking insulin had high TGs level as compared to diabetic rats treated with insulin only. Conclusion: The study concluded that the inflammatory pathways hold a substantial part in insulin resistance in type 2 DM. The influence of salicylate compounds on insulin sensitivity is multifactorial especially in high doses, and involves both beneficial and deleterious effects depending on the species and experimental model studied. Introduction Diabetes mellitus (DM) is a disorder in which blood sugar levels are abnormally high because either absolute or relative insulin deficiency. In type 1 diabetes (IDDM), more than 90% of the insulin-producing cells of the pancreas are permanently destroyed. In type 2 diabetes (NIDDM), the pancreas continues to produce insulin, sometimes even at higher than normal levels.
However, the body develops resistance to the effects of insulin, so there is not enough insulin to meet the body’s needs. Type 2 DM may occur in children and adolescents, but usually begins in people older than 30 years and becomes progressively more common with age. Obesity is the chief risk factor for developing type 2 DM and 80% - 90% of people with this disease are obese. Certain diseases and drugs are known to affect the body uses insulin and can lead to type 2 DM. High levels of corticosteroids and pregnancy are the most common causes of altered insulin use.
If a person with DM keeps blood sugar levels tightly controlled, complications are less likely to develop. Potentiation of oral antidiabetic agents may be caused partially by displacement from serum proteins. Now, it turns out that IKK-beta also works in another pathway to contribute to insulin resistance by interfering with insulin signaling [1-7].


Evidence that these effects were mediated by salicylate inhibition of IKK? activity, as opposed to inhibition of cyclooxygenases, was obtained by demonstrating that heterozygous deletion of IKK? protected mice against the development of insulin resistance during high-fat feeding or lipid infusion [9].
While early studies suggested a neutral effect of aspirin on glucose metabolism in diabetic patients [10-15], more recent clinical trials have demonstrated a detrimental effect of aspirin therapy on insulin sensitivity [16-18]. The blood glucose levels (by using standard diagnostic kits) were recorded to monitor the degree of diabetes.
Statistical Analysis Data analysis was performed using SPSS software, version 16. Difference between the experimental groups were evaluated by Kruskal-Wallis, followed by the Mann-Whitney test (for significant variables), for multiple comparisons on ranks. Data were ranked by ordering them from lowest to highest and assigning them, in order, the integer values from 1 to the sample size.
Ties were resolved by assigning tied values the mean of the ranks they would have received if there were no ties. Accordingly, the mean rank score was calculated for each group by dividing the sum of the ranks by the sample size of that group. Fasting Blood Sugar Level in normal and diabetic rats without or with administration of insulin alone or insulin and different doses of aspirin. HemoglobinA1c% (HBA1c%) Level in normal and diabetic rats without or with administration of insulin and aspirin in different doses. Serum Triglycerides (TGs) Level (mgldl) Tables 4(a), (b) and figure 7 showed that diabetic rats taking MDA had a significant higher serum TGs level as compared to normal non-diabetic rats (P Tables 4(a), (b) and figure 8 showed that treated diabetic rats with insulin and taking moderate dose aspirin had significant higher serum TGs levels as compared to either normal non-diabetic rats or treated diabetic rats taking insulin (P Other parameters showed no significant changes between the different groups studied. Discussion Although the hypoglycemic potential of salicylate therapy was demonstrated more than 100 years ago [21] and again recently [22], clinical utility in diabetes treatment has been limited by nausea, vomiting, tinnitus and deafness associated with high dose therapy [23-25].
Triglycerides (TGs)) Level in normal and diabetic rats without orwith administration of insulin and aspirin in different doses. Again, HBA1c% (glycosylated hemoglobin%) in diabetic rats treated with insulin and receiving HDA was lower than diabetic rats treated with insulin only or even taking LDA in addition. On the contrary, different doses of aspirin (LDA, MDA & HDA) administration to diabetic rats have no any influence on HBA1c% as compared to normal non-diabetic rats.
Specifically, TNF-? can decrease glucose uptake and utilization of peripheral tissues by targeting insulin signaling pathways and glucose transport 4 (GLUT 4). NF-k? is a pro-inflammatory marker that controls the production of a host inflammatory markers and mediators, including TNF-?, IL-6 and C-reactive proteins [33-35]. TNF-? induces activation of the IKK? kinases which regulates NF-k? transcriptional activity.
Aspirin is supposed to inhibit NF-k? expression and also reduced TNF-? level and improved insulin resistance [32]. These results are in accordance with Yaun hypothesis [8] supporting it as opposed to the inhibition of the cyclooxygenases pathway [36,37] as evidenced by the lack of any effect of different aspirin doses on C-reactive proteins level in diabetic rats in our study. Decreased hepatic insulin sensitivity may lead to increased hepatic gluconeogenesis, post-prandial hyperinsulinemia and increased formation of triglycerides (TGs) [32]. High dose aspirin, through its effect on peripheral insulin resistance as evidenced in many studies as well as in our study was supposed to improve insulin resistance in the liver of diabetic rats and lower, rather than augmenting TGs level. Actually, our study showed an elevated levels of TGs in diabetic rats receiving MDA alone as compared to normal non-diabetic rats, Again, moderate and HDA in diabetic rats not taking insulin had high TGs level as compared to diabetic rats treated with insulin only.
These findings suggest that the effects of salicylates may depend on the experimental model and possibly on the species studied [40]. This could not preclude patients from taking low dose aspirin to prevent cardiovascular disease [41], but more basal and clinical studies are needed before recommending higher dosage of salicylates for the treatment of type 2 diabetes itself. Acknowledgements This study was sponsored by Institute of Scientific Research and Revival of Islamic Heritage with grant no. Moataz AbdelFattah, Professor of Medical Statistics and Clinical Epidemiology, Medical Research Institute, Alexandria University, Egypt for his statistical advice throughout preparation of this manuscript. Schmitz-Peiffer, et al., “Alterations in the Expression and Cellular Localization of Protein Kinase C Isozymes Epsilon and Theta Are Associated with Insulin Resistance in Skeletal Muscle of the High Fat Fed Rat,” Diabetes, Vol. Spiegelman, “Adipose Expression of Tumor Necrosis Factor: Direct Role in Obesity-Linked Insulin Resistance,” Science, Vol. Shulman, “Cellular Mechanisms of Insulin Resistance,” The Journal of Clinical Investigation, Vol.
Kim, et al., “Prevention of Fat-Induced Insulin Resistance by Salicylate,” The Journal of Clinical Investigation, Vol. Waldhausl, “Acetyl-Salicylic Impairs Insulin-Mediated Glucose Utilization and Reduces Insulin Clearance in Healthy and Non-Insulin-Dependent Diabetic Man,” Diabetologia, Vol. Shoelson, “Reversal of Obesityand Diet-Induced Insulin Resistance with Salicylates or Targeted Disruption of IKK?,” Science, Vol.
Shulman, “Mechanism by Which High-Dose Aspirin Improves Glucose Metabolism in Type 2 Diabetes,” The Journal of Clinical Investigation, Vol. Alberti, “Inhibition by Salicylate of Gluconeogenesis in the Isolated Perfused Rat Liver,” Clinical and Experimental Pharmacology and Physiology, Vol.
Sperling, “Indomethacin and Salicylate Decreaseepinephrine-Induced Glycogenolysis,” Metabolism, Vol. Burt, “NIDDM as a Disease of the Innate Immune System: Association of Acute-Phase Reactants and Interleukin-6 with Metabolic Syndrome X,” Diabetologia, Vol. Jacobs, “Association of Inflammation with Worsening Homa-Insulin Resistance,” Diabetologia, Vol. Glassm, “Macrophages, Inflammation, and Insulin Resistance,” Annual Review of Physiology, Vol.
Dixitm, “Obesity Increases the Production of Proinflammatory Mediators from Adipose Tissue T Cells and Compromises TCR Repertoire Diversity: Implications for Systemic Inflammation and Insulin Resistance,” The Journal of Immunology, Vol. Wang, “Effect of Aspirin on the Expression of Hepatocyte NF-?B and Serum TNF-? in Streptozotocin-Induced Type 2 Diabetic Rats,” Journal of Korean Medical Science, Vol. Shoelson, “Local and Systemic Insulin Resistance Resulting from Hepatic Activation of Ikk? and NF-?B,” Nature Medicine, Vol. Karin, “IKK? Links Inflammation to Obesity-Induced Insulin Resistance,” Nature Medicine, Vol. Zhang, “Feed-Forward Signaling of TNF-? and NF-?B via IKK? Pathway Contributes to Insulin Resistance and Coronary Arteriolar Dysfunction in Type 2 Diabetic Mice,” American Journal of Physiology Heart and Circulatory Physiology, Vol. Drzewoski, “Reduced Sensitivity of Plateletsfrom type 2 Diabetic Patients to Acetylsalicylic Acid (Aspirin)—Its Relation to Metabolic Control,” Thrombosis Research, Vol.
Bocksch, “Prevalence of Aspirinresistance in Patients with Type 2 Diabetes,” ActaDiabetologica, Vol. Brodows, “Aspirin Causes Tissue Insensitivity to Insulin in Normal Man,” The Journal of Clinical Endocrinology and Metabolism, Vol. Torella, “Influence of Acetylsalicylic Acid on Glucose Turnover in Normalman,” Diabetes and Metabolism, Vol.



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Comments

  1. 24.04.2014 at 10:42:59


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  3. 24.04.2014 at 22:55:25


    The mortality rate of diabetic ketoacidosis.

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