Pohlandt F 1994 Prevention of postnatal bone demineralization in very low-birth-weight infants by individually monitored supplementation with calcium and phosphorus. This journal is a member of and subscribes to the principles of the Committee on Publication Ethics. Chronic Kidney Disease (CKD) is characterised by a gradual and permanent loss of kidney function that worsens as it progresses from stages 1 to 5. Renal anaemia is secondary to chronic kidney disease (CKD) and it appears early in the course of CKD, worsening as it progresses.
Diagnosis of Renal AnaemiaDiagnosing renal anaemia is critical in establishing early and appropriate treatment in patients.
Treament for Renal AmaemiaThe priority in the treatment of anaemia is to correct the underlying factors that caused the anaemia and the specific treatment strategy depends on the cause of the anaemia. There are a number of treatment guidelines available which provide information on the management of anaemia and CKD related anaemia. National Kidney Foundation The National Kidney Foundation, a major voluntary nonprofit health organization, is dedicated to preventing kidney and urinary tract diseases, improving the health and well-being of individuals and families affected by kidney disease and increasing the availability of all organs for transplantation.
Official journal of the International Society of Nephrology NPG publishes journals and online databases across the life, physical and applied sciences and, most recently, clinical medicine.
The Renal Association (UK) The Renal Association is the professional body for United Kingdom nephrologists (renal physicians, or kidney doctors) and renal scientists in the UK.
European Renal Association - European Dialysis and Transplant Association (ERA-EDTA)The ERA-EDTA is one of the fastest growing Medical Association whose purpose is to encourage and to report advances in the field of clinical nephrology, dialysis, renal transplantation and related subjects.
This decrease in PP did not result from a decreasing phosphate supplementation, because UP did not decrease. EPG Patient Direct is not in a position to verify this information and cannot be held responsible for the accuracy of the content contained. Anaemia is defined as a reduction of the number of circulating red blood cells to below a certain threshold level. Treatment of renal anaemia can significantly improve overall health in patients with chronic kidney disease (CKD). Content encompasses daily news from award-winning journalists, expert opinion and practical methodology, and more high impact research and reviews than any science publisher.
Plasma and urine phosphate concentrations (PP, UP) determined on the same day together with the corresponding creatinine concentrations (PCrea, UCrea) built up a data set. Therefore we concluded that the renal phosphate threshold decreases with increasing pm age.


Every infant contributed, if available, only one value to every calculation in every 2-wk period. It is also described as a low haemoglobin concentration or a low volume of packed red cells2. It is also involved in the promotion and dissemination of research and education relating to the specialty. Relationship between hemoglobin level and quality of life in anemic patients with chronic kidney disease receiving epoetin alfa.
The following investigation was carried out to study this hypothesis.Top of pageMETHODSThe case notes of 62 VLBW infants admitted to our intensive care unit from October 1984 to December 1987 were reviewed retrospectively. Wiley, New York, pp 31–43 Bijvoet OLM 1977 Kidney function in calcium and phosphate metabolism. The Association was founded in 1950, when its activities were restricted to holding scientific meetings.
All urinary measurements of phosphate and creatinine (morning spot urinary specimens) were recorded in cases where plasma concentrations of the same morning were available. Academic Press, New York, pp 50–83 Senterre J, Salle B 1988 Renal aspects of calcium and phosphorus metabolism in preterm infants. Data sets of infants younger than 3 d of age were omitted because of possible transitional changes during this early time after birth. For each data set the pm age (gestational age plus postnatal age) of the individual child was calculated.
The impact of anemia on cardiomyopathy, morbidity, and and mortality in end-stage renal disease.
Data sets obtained from 29 to 36 wk of pm age (study period) were included in the study.Measured data. Phosphate, creatinine, and calcium were measured in plasma and urine by standard methods during the daily routine(2–4). Levin A, Thompson CR, Ethier J, Carlisle EJ, Tobe S, Mendelssohn D, Burgess E, Jindal K, Barrett B, Singer J, Djurdjev O. The method of calculating censored median values and their 95% confidence interval was taken from life table analysis (Kaplan Meier)(5, 6). Left ventricular mass index increase in early renal disease: impact of decline in hemoglobin. In life table analysis it happens sometimes that a patient, who survived a certain time, moves away and his outcome is not known.


This time can be used as censored survival time for estimation of the survival probability of the whole population over a period of time. The effects of higher hemoglobin levels on mortality and hospitalization in hemodialysis patients.
Perlman RL, Finkelstein FO, Liu L, Roys E, Kiser M, Eisele G, Burrows-Hudson S, Messana JM, Levin N, Rajagopalan S, Port FK, Wolfe RA, Saran R. Median postnatal age was 21 (3-82) d (Table 1), and 395 data sets were included in the study.
Quality of life in chronic kidney disease (CKD): a cross-sectional analysis in the Renal Research Institute-CKD study. We concluded that the renal phosphate threshold decreases with increasing pm age.The kidney plays a key role in regulating PP(7, 8). As shown inTable 4 it was now possible to include the data for 31-52 of the 62 infants studied in each 2-wk period.
In the first place, renal function might be impaired by exogenous factors like repeated episodes of ischemia and hypoxemia or nephrotoxic drugs. For example, all but three of our infants received antibiotic therapy with aminoglycosides, which might have affected renal tubular function in spite of regular drug level monitoring. The effect of aminoglycosides on the renal phosphate threshold in preterm infants has not yet been investigated. Second, it is well known that PTH does have a phosphaturic effect by decreasing renal phosphate threshold. Therefore an increasing secondary hyperparathyroidism is unlikely the cause of this postnatal change in the phosphate threshold.
Finally there is an imbalance in maturation of glomerular and tubular function in the preterm kidney.
In VLBW infants, GFR is related to pm age and increases exponentially from 26 to 33 wk of pm age(14). By usingβ2-microglobulin as a marker for tubulus maturation, glomerular functional development precedes tubular functional development until the 34th pm wk(15).



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