Type 2 diabetes and circulation problems 8th,dam hm km,herbal treatment for diabetes 2 kost,management of type 2 diabetes mellitus ppt harrison - How to DIY

When the body cannot regulate the blood sugar level, this will lead to high glucose (sugar) concentration in the blood.
Autonomic neuropathy, on the other hand, is affecting nerves on areas that we do not control consciously.
Diabetes could hinder an individual’s normal blood circulation due to the narrowing of arteries. Diabetics are prone to fungal and bacterial infections because of changes in their diet and medications. Basic proper foot care could greatly help prevent the onset of diabetic foot pain and other foot problems.
The condition of gangrene is a serious one, primarily marked by loss of adequate supply of blood to the tissues of the body, ultimately resulting in its necrosis or death. Injury to the tissues, infection (mostly of bacterial origin) and presence of any underlying health ailment that inflicts damage to the blood carrying vessel structure form the three key underlying mechanisms that are responsible for initiation and development of irreversible tissue damage and life threatening condition of gangrene.
As already explained, one of the causes of gangrene in the toes and fingers is linked with damaged blood vessels. High level of blood glucose associated with diabetes mellitus also damages the nerves (known as peripheral neuropathy), especially the nerves in the lower extremities. The dullness or lack of sensation puts diabetic individuals at an increased risk of injuring the skin of their fingers or feet without having any realization (it mostly goes unnoticed in the feet region). The sore or foot ulcer formed takes an extended time to heal because of both, poor circulation of blood through the injured area as well as lesser number of defence cells. Bacterial infection also forms another causative factor that makes the diabetic individual more prone to developing gangrene. Such type of infection is marked by noticeable skin discolouration and dryness along with the formation of skin blisters. The weakened immune system (seen in diabetes) further raises the risk of incurring serious infection, which later develops, into gangrene. Patients with diabetes not only have to monitor their glucose levels but also take extra attention to taking care of their feet.
This is why appointments for diabetes care are not complete without a thorough foot examination. To best address the condition, the guidelines further note that a multi-disciplinary team, including infectious diseases specialists, podiatrists, surgeons and orthopedists be at hand to diagnose and treat the infection.
Diabetic foot infections are an increasingly common problem, but proper care can save limbs and, ultimately, lives, suggest new guidelines released by the IDSA. Poor treatment of infected foot wounds in people with diabetes can lead to lower extremity amputation, and about 50 percent of patients who have foot amputations die within five years – a worse mortality rate than for most cancers. Because people with diabetes often have poor circulation and little or no feeling in their feet, a sore caused by a rubbing shoe or a cut can go unnoticed and worsen. The guidelines emphasize the importance of rapid and appropriate therapy for treating infected wounds on the feet, typically including surgical removal (debridement) of dead tissue, appropriate antibiotic therapy and, if necessary, removing pressure on the wound and improving blood flow to the area. Because treatment of diabetic foot infections can be complicated, the best approach is to involve a multidisciplinary team that can assess and address various aspects of the problem, suggest the guidelines, which are a revision and update of IDSA's 2004 diabetic foot infections guidelines.
Research since the previous guidelines shows that many foot infections are treated improperly, including prescribing the wrong antibiotic or not addressing underlying conditions such as peripheral arterial disease. The new guidelines include 10 common questions with extensive, evidence-based answers, which the panel that wrote the guidelines determined were most likely to help a health care provider treating a patient with diabetes who has a foot wound. When a foot sore is infected, imaging the foot is usually necessary to determine if the bone is infected. The voluntary guidelines (which can be found at the IDSA website) are not intended to take the place of a doctor's judgment, but rather to support the decision-making process, which must be individualized according to each patient's circumstances.
According to a University of Texas study, Crazy Ants may become the dominant invasive ant species displacing Fire Ants in the near future. Scientists have successfully placed tiny synthetic motors in live human cells through nanotechnology. Over the past year I have interacted with hundreds of people with diabetes, and have come to learn one very important lesson that has changed my view of diabetes altogether. While insulin resistance is a condition that is most commonly associated with type 2 diabetes, an increasing body of evidence is now shedding light on the fact that insulin resistance is a common thread that underlies many health conditions previously unassociated with blood sugar, including (but not limited to) heart disease, diabetes, atherosclerosis, the metabolic syndrome, obesity and cancer.
What that means is simple: insulin resistance significantly increases your risk for the development of a collection of health conditions that can significantly reduce your quality of life and decrease your life expectancy.
Insulin is a hormone which is released by the pancreas in response to rising blood glucose. Without insulin, cells in the liver, muscle, and fat have a difficult time vacuuming up glucose from the blood.
Insulin resistance is a condition in which multiple tissues in the human body become resistant to the effects of insulin. As a result of insulin resistance, the pancreas is forced to secrete increasing amounts of insulin, resulting in a condition known as hyperinsulinemia. Hyperinsulinemia is a dangerous condition for many tissues, simply because elevated insulin concentrations in the blood act as potent signals for cell growth. There is a significant amount of confusion about what actually causes insulin resistance, and in my life as a type 1 diabetic as well as my career as a research scientist, I have come to realize that life-saving information about insulin resistance is poorly understood. There exists a large wall between what the research world understands about insulin resistance and what the general public understands about insulin resistance. This wall exists for a number of reasons, and is heavily influenced by economic forces that profit on lifelong health conditions like diabetes.


Researchers debate the causal mechanisms of insulin resistance tirelessly, day after day, and travel thousands of miles to attend large conferences to flex their scientific muscles. Despite this, however, researchers in the laboratory environment can induce insulin resistance in laboratory animals an in humans incredibly easily, using one simple technique.
Insulin resistance is caused by lipid overload, resulting from either a high fat diet or insufficient fat “burning” through movement.
Visit almost any laboratory on the planet that studies insulin resistance in animals or in humans and you’ll notice one simple technique that achieves insulin resistance in a repeatable fashion – eating a diet high in fat. In some studies, researchers use a diet high in fat and high in sucrose (table sugar), to ensure that both the muscle and the liver become extremely insulin resistant. A high intake of dietary fat causes lipid overload and insulin resistance in the muscle and liver.
I am often asked whether there is any research to back up the claim that fatty acids cause insulin resistance.
Saturated fatty acids are derived mainly from animal sources, and have direct negative effects on the muscle and liver.
When excess fat accumulates it the muscle and liver tissue, the ability of glucose to enter both tissues is significantly compromised. Too often, the blame is placed on carbohydrates as the cause of insulin resistance despite the fact that the evidence clearly supports that excessive fat consumption causes excessive fat storage. I’ve talked extensively about the difference between REAL and FAKE carbohydrates, and gone into detail about the effects they have on tissues throughout your body. To save you from having to scroll through previous articles, let’s walk through the difference between REAL and FAKE carbohydrates once again.
REAL carbohydrates come from mainly fruits and vegetables, and can be eaten in their whole, natural state with minimal cooking or processing.
REAL carbohydrates have untold health benefits, and are absolutely required for optimal athletic performance, athletic recovery and preventing against lipid overload. In the past decade, a large body of evidence has begun to uncover the potent effects of refined carbohydrates on decreased cardiovascular health, diabetes health, liver health and unwanted weight gain.
Exercise is without doubt the most effective method of increasing insulin sensitivity, and is considered the gold-standard method of decreasing diabetes risk(20–27).
Think of exercise as being the signal that increases the appetite of your muscle tissue to accept incoming glucose in the blood. Exercise acts in both the short term and the long term, and leads to significant increases in the ability of the muscle tissue to respond to glucose in the blood.
I am happy to see that exercise is now being prescribed as a treatment for insulin resistant individuals, regardless of whether they have diabetes or not. It is important to recognize that insulin resistance affects everyone, even those who show no symptoms of high blood sugar. Diagnosed with type 1 diabetes at the age of 22, I have spent over a decade learning the fundamentals of nutrition at the doctorate level.
That's why I've spent over 10 years developing a rock solid system that can minimize blood glucose variability and insulin resistance. Death of cell and tissue may target any part of the body, however, it has been typically observed in the extremities, such as the toes, fingers and hands. In both, type 1 and type 2 diabetes, the raised level of sugar is capable of causing damage to the blood vessels and reducing blood supply. Owing to the nerve damage, the transmission of sensation, particularly those of pain to the brain gets impaired.
A notorious bacterial organism, Clostridium perfringes is often linked with gas gangrene; after it attacks the site of injury or sometimes, surgical wound.
Diabetes complicates the microvascular circulation which decreases blood circulation and slows down healing of the infected area.
But about half of lower extremity amputations that aren't caused by trauma can be prevented through proper care of foot infections, note the new IDSA diabetic foot infections guidelines, which are being published today in the journal Clinical Infectious Diseases.
Many patients with foot infections initially receive only antibiotic therapy, which is often insufficient in the absence of proper wound care and surgical interventions, the guidelines note. The first step is to determine if the wound is infected, which the guidelines note is likely if there are at least two of the following signs: redness, warmth, tenderness, pain or swelling. It is also important to perform a culture of the wound to determine the bacteria causing the infection, which will then help guide which antibiotic should be used for treatment.
This realization came to me early on in my career as a nutrition and fitness coach for people with diabetes, and continues to hold true. When you consume carbohydrates, the glucose that enters the bloodstream knocks on the door of the beta cells in the pancreas as a signal to make insulin.
These tissues are capable to vacuuming up only a small percentage (5-10%) of the glucose in circulation without the help of insulin. Unfortunately, excellent research does no good if the information is not put in the hands of those who need it. You may have heard the phrase “there is no money in the cure.” This is a true statement, and this practice is what keeps life-saving information out of the hands of those who need it.
They propose every mechanism you can imagine, and blame every tissue you can think of, from the pancreas to the muscle to the liver to the brain to your blood.


There is a considerable amount of research that justifies this notion, and this evidence clearly points to the fact that excess fatty acids are a potent cause of both muscular and liver insulin resistance. REAL carbohydrates are found mainly in plant foods, and come pre-packaged with a host of vitamins, minerals, antioxidants, fiber and water. They are “refined” products that have been processed, manufactured and changed from their original and whole state.
Forcing the muscle to contract and elongate thousands of times in a short period of time increases it’s fat burning capabilities and also increases its willingness to store more glucose. In many cases, those with prediabetes can stave off the transition to diabetes by adopting a regular exercise regimen to increase insulin sensitivity regularly. Massao Hirabara S, de Oliveira Carvalho CR, Mendonca JR, Piltcher Haber E, Fernandes LC, Curi R. In the absence of a continuous supply of nutrient and oxygen rich blood, the cells within the body begin to fail in carrying out their normal function and finally give up. As a result of restricted blood circulation to the extremities, such as the feet, the area becomes deprived of those cells (white blood cells) which aid in fighting off any infection. Guidelines released by the Infectious Diseases Society of America (IDSA) suggests that most amputations can be prevented with proper care.
About half of ulcers are not infected and therefore should not be treated with antibiotics, the guidelines note.
Because of the complexity of diabetic foot infections, the guidelines suggest these patients are best served by a multidisciplinary team, including infectious diseases specialists, podiatrists, surgeons and orthopedists.
When insulin is present, the amount of glucose that can be transported into tissues significantly increases, allowing tissues to be properly fed, and keeping blood glucose concentrations in the normal range. More tissue growth often results in increased fatness, increased cell replication rates and a significant increase in the risk for cancer.
Think of REAL carbohydrates as the types of foods that you would find if you were walking in the woods by yourself.
FAKE carbohydrates have been modified from their original state in order to make them edible and often times to make them taste sweet. Free fatty acid-induced insulin resistance is associated with activation of protein kinase C theta and alterations in the insulin signaling cascade. Mechanism by which fatty acids inhibit insulin activation of insulin receptor substrate-1 (IRS-1)-associated phosphatidylinositol 3-kinase activity in muscle.
Saturated fatty acid-induced insulin resistance is associated with mitochondrial dysfunction in skeletal muscle cells.
Palmitate acutely raises glycogen synthesis in rat soleus muscle by a mechanism that requires its metabolization (Randle cycle). Increased lipid availability impairs insulin-stimulated ATP synthesis in human skeletal muscle.
Mechanisms underlying skeletal muscle insulin resistance induced by fatty acids: importance of the mitochondrial function. Rice and noodle consumption is associated with insulin resistance and hyperglycaemia in an Asian population. Resistance exercise enhances the molecular signaling of mitochondrial biogenesis induced by endurance exercise in human skeletal muscle. Skeletal muscle and beyond: the role of exercise as a mediator of systemic mitochondrial biogenesis.
Effects of 7 days of exercise training on insulin sensitivity and responsiveness in type 2 diabetes mellitus. Effects of exercise on glycemic control and body mass in type 2 diabetes mellitus: A meta-analysis of controlled clinical trials.
Skeletal muscle lipid content and insulin resistance: evidence for a paradox in endurance-trained athletes.
Exercise as a therapeutic intervention for the prevention and treatment of insulin resistance. Swelling and inflammation resulting from the infection causes the local temperature of the area involved to be slightly elevated, and also leads to pain. People with infections do need antibiotic therapy and those with a severe infection should be hospitalized immediately. In rural areas, doctors may be able to use telemedicine to consult with the appropriate experts, Dr. FAKE carbohydrates include grains, cereals, pastas, rice, bread products and artificial sweeteners.
Dead and infected tissue must be surgically removed, which, if the infection is extensive, can mean amputation of the toe, foot, or even part of the leg. Nearly 80 percent of all nontraumatic amputations occur in people with diabetes – and 85 percent of those begin with a foot ulcer.



Alternative medicine for juvenile diabetes 2014
Diabetes patient food list in tamil 2014
30.11.2015 Cures For Diabetes


Comments to Type 2 diabetes and circulation problems 8th

  1. Insulin to overcome the insulin resistance journal Annals of Medicine the consequences.
  2. Arzu_18 on 30.11.2015
  3. Life for the rest of their fibre.
  4. dagi on 30.11.2015
  5. From being made with some of the nutrient-dense showed adding more coconut oil.
  6. KazbeK_666 on 30.11.2015