Treatment of postprandial hyperglycemia in type 2 diabetes diet,gc gruppe umsatz 2014,how to treat diabetes in hamsters,diabetes treatment medications used - Plans Download

T2DM likely develops as a result of polygenic defects that predispose affected individuals to the disease. The complex interplay between these defects contributes to the ongoing progression of T2DM, although the disease itself results mainly from the first 3 defects: impaired insulin secretion from b-cells, impaired insulin action in muscle, and increased hepatic glucose production.
Initially, an increase in insulin resistance and impairments in b-cell function and the incretin effect interact, over time resulting in a relative insulin deficiency as well as excessive glucagon production (leading to overproduction of endogenous glucose in the liver). EVERY 1% reduction in HBA 1C REDUCED RISK* 1% Deaths from diabetes Heart attacks Microvascular complications Peripheral vascular disorders UKPDS 35.
Follow up Elke diabetes patient verdient 3 tot (maximaal) 6 maandelijks een HbA1c bepaling Elke diabetes patient verdient 3 tot (maximaal) 6 maandelijks een HbA1c bepaling Jaarlijks obligatoir nazicht oftalmologisch Jaarlijks obligatoir nazicht oftalmologisch Jaarlijks nazicht andere cv risicofactoren Jaarlijks nazicht andere cv risicofactoren Jaarlijks nazicht microalbuminurie Jaarlijks nazicht microalbuminurie Elke raadpleging nazicht voeten !!!!! Insulin Resistance Syndrome + Obesity + Insulin resistance + Hyperinsulinaemia (initially) + Type 2 diabetes or impaired glucose tolerance + Dyslipidaemia + ?? Blood pressure + Atherosclerosis DeFronzo, Ferrannini.
Therapie is dynamisch doorheen de ziekte NHANES=National Health and Nutritional Examination Survey.


Environmental factors such as a sedentary lifestyle and a high-fat diet can exacerbate defects in both insulin secretion from pancreatic b-cells and insulin action in muscle and adipose tissues. American Association of Clinical Endocrinologists Medical Guidelines for Clinical Practice for developing a diabetes mellitus comprehensive care plan.
Contributions of insulin-resistance and insulin-secretory defects to the pathogenesis of type 2 diabetes mellitus.
Clinical review 135: The importance of beta-cell failure in the development and progression of type 2 diabetes.
Acarbose treatment and the risk of cardiovascular disease and hypertension in patients with impaired glucose tolerance: the STOP-NIDDM trial. Intense metabolic control by means of insulin in patients with diabetes mellitus and acute myocardial infarction (DIGAMI 2): effects on mortality and morbidity. Effect of intensive blood-glucose control with metformin on complications in overweight patients with type 2 diabetes (UKPDS 34).


Intensive blood-glucose control with sulphonylureas or insulin compared with conventional treatment and risk of complications in patients with type 2 diabetes (UKPDS 33).
Om het te downloaden, raad, alsjeblieft, deze presentatie aan je vrienden in de sociale netwerken. From the triumvirate to the ominous octet: a new paradigm for the treatment of type 2 diabetes mellitus.
Both glucotoxicity and lipotoxicity worsen b-cell secretion, which in turn aggravates hyperglycemia.



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