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Autism is a disorder of neural development characterized by impaired social interaction, by impaired verbal and non-verbal communication, and by restricted, repetitive, or stereotyped behavior. A widely accepted explanation for an apparent world-wide, rapid acceleration in the prevalence of Autism Spectrum Disorder (ASD) has remained elusive despite intense scientific investigation intent on discovering the etiology of this disorder.
Previous research carried out by Harvard University psychiatrist and Pulitzer Prize winning author Dr.
We propose a scientific study designed to expose an association, if any such association exists, between maternal recall of such an ETA event as described in Dr.
This research project will attempt to document any correlations among volunteer mothers of children diagnosed with ASD who give a history of spontaneous recall (i.e.
Funding that will enable this study to commence is hereby requested in the amount of $100,000.
Could such a research protocol as that suggested above result in the acquisition of new information which might help to elucidate a completely unanticipated reason for the apparent acceleration in the prevalence of ASD world-wide? In considering ASD, we must acknowledge that we are confronting a relatively recently recognized variation in normal human development, the prevalence of which appears to be accelerating at a truly alarming rate, and the implications of which, for the future of humanity, could be nothing less than profound.
Again, from Wikipedia:[“Autism has a strong genetic basis, although the genetics of autism are complex and it is unclear whether ASD is explained more by rare mutations with major effects, or by rare multigene interactions of common genetic variants.
We can see that ASD is a disorder of relatively recent description that appears to be exhibiting a rapid increase in prevalence in our society and probably involves new (uninherited) genetic mutations. Each of these elements suggest that a link between ASD and the ETA phenomenon, as described in Dr. For decades UFO researchers have been “leaning into the wind” as they have battled to increase our awareness and understanding of the UFO and other, possibly related phenomena.
The apparent rapid acceleration in the prevalence of ASD among our population may be one important manifestation of an extraterrestrial presence and intervention impacting our world.
We could begin this process by designing and funding a research protocol, with the help of professional epidemiologists, which will address the important question of whether or not the ETA research previously accomplished by Dr. Over the last 5 years, scientists have identified a number of rare gene changes, or mutations, associated with autism.


This is extremely unlikely because, currently, our society remains trapped in a conundrum in which valid questions that appear to lend any credence to the Extraterrestrial Hypothesis will not be addressed by the wider scientific community. Mack) long enough to inform ourselves about the UFO phenomenon and its associated Extraterrestrial Hypothesis currently recognize and acknowledge the existence of a great deal of evidence which robustly supports the conclusion that several phenomena surrounding the Extraterrestrial Hypothesis are real.
Children born with ASD are suspected to have a heritable, but in most cases not inherited, change (mutation) in their genetic material (DNA). Complexity arises due to interactions among multiple genes, the environment, and epigenetic factors which do not change DNA but are heritable and influence gene expression.
It is imperative that we also understand and acknowledge that the prevalence of this disorder appears to be increasing coincidentally with numerous reports, well documented by Dr. A total lack of research funding, denial, ridicule, and at times intimidation have presented nearly insurmountable obstacles to their attempts to increase our understanding of these extraordinary, but still largely discounted phenomena. Mack’s abduction research, and the subsequent birth to that mother of a child that has been diagnosed with ASD. Statistical comparison and analysis will be made between those data and equivalent data sets obtained from otherwise matched mothers of children diagnosed with ASD but lacking a history of either spontaneous or hypnosis-aided recall of an ETA event.
However, the broad majority of our society remain intransigently resistant to affirming and accepting this now obvious conclusion. The important question is – if the prevalence of ASD is in fact increasing in our society, then what is causing these new genetic mutations to occur? Therefore, a complete epidemiologic investigation designed to ferret-out the root cause of the new genetic mutation(s) responsible for the apparent world-wide increase in the prevalence of ASD should include an aim to define what role, if any, the ETA phenomenon might be playing in the origin and explosive evolution of ASD. Scientific freedom to investigate the UFO, ETA, and other possibly related phenomena has been severely and unwisely restricted.
We can then be set free to discuss, to share information, and to move toward finding answers to myriad questions concerning who these visitors are, how they are appearing here in our reality, why they are here, how they may have previously influenced human civilization, how they view their relationship to humanity, and, most importantly, how we might establish a less frightening, peaceful, and perhaps mutually beneficial relationship with them. Such an approach could demonstrate a presently unanticipated and unrealized association between mothers with a history of ETA and the rapidly increasing prevalence of ASD world-wide. Additionally, discovery of a positive correlation would serve to alert mankind to a covert and ongoing program, conceived and enacted by extraterrestrials, that is apparently centered on the wholesale genetic modification of humanity.


Any question framed by the Extraterrestrial Hypothesis, and there are potentially many such questions, simply cannot and will not be addressed by otherwise qualified members of established academia.
Our society therefore remains mired in a present world view in which not only are we disallowed from submitting research grant proposals such as the “Hypothetical Research Grant Proposal” above, but we remain socially constrained from even asking such (possibly) relevant questions, even among friends and family.
Leo Sprinkle may have an identifiable and causative association with the apparent explosive and alarming escalation in the world-wide prevalence of ASD.
Scientific research intending to investigate and to potentially expose a heretofore unrecognized connection between Dr.
Typically, autism cannot be traced to a Mendelian (single-gene) mutation or to a single chromosome abnormality, and none of the genetic syndromes associated with ASDs have been shown to selectively cause ASD. Mack’s research findings and the world-wide, rapidly escalating prevalence of Autism Spectrum Disorder has not, to date, been undertaken.
Numerous candidate genes have been located, with only small effects attributable to any particular gene. The large number of autistic individuals with unaffected family members may result from copy number variations—spontaneous deletions or duplications in genetic material during meiosis. At the very least we can declare that we know enough about them to state with certainty that they exist. Mack’s patients report having experienced these alleged ETA events during their reproductive years, and many report that gametes, either sperm or eggs, were harvested from them, against their will, at the time of their alleged ETA. Given that they exist and that they are, without question, extraordinary, we can righteously postulate that their existence may be impacting our civilization in ways that, due to our imposed ignorance surrounding these phenomena, we have not yet been truly free to seriously consider and investigate.
Additionally, alleged ETA events have ceased as the “experiencers” have moved past their reproductive years and into middle age.



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