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Does diabetes cause chronic kidney disease,jan andries van der veen,jan video - Videos Download

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Dear, after practicing for 12 years, we have developed HKIII Diabetes Diet Treatment which can successfully lower blood sugar level in 7 days. According to the statistic of World Health Organization, a total of 380 millions people around the globe have diabetes. Ingested K+ is absorbed rapidly and enters the portal circulation, where it stimulates insulin secretion. An increase in extracellular potassium concentration also stimulates aldosterone secretion (via angiotensin II), and aldosterone increases K+ excretion. Potassium homeostasis is maintained by the balance of potassium excretion and potassium cellular shifts. This applies to certain high-risk patient populations with cardiac disease, such as ischemic or scarred myocardium, left ventricular hypertrophy, congestive heart failure, or myocardial infarction).
Hypokalemia is found in about 20% of hospitalized patients, but it occurs in less than 1% of otherwise healthy adults. Hypokalemia can result from transcellular shifts (from extracellular into intracellular spaces), or when potassium losses are increased; these losses can be from renal or nonrenal causes (Box 1). When hypokalemia is reported, the initial step is to ascertain whether it is associated with clinical symptoms or arrhythmias that would require prompt intervention.
In a person with normal renal function and average potassium intake, FEK is approximately 10%.
The usefulness of the FEK and TTKG is limited by their variability with diet and chronic kidney disease. Cause of hypokalemia should be addressed (remove drug, change diet, stop gastric drainage).
All other situations except for renal tubular acidosis: replace with oral potassium bicarbonate, citrate, acetate, or gluconate.
More than 80% of hyperkalemic episodes are caused by impaired potassium excretion from renal insufficiency.1 Usually, another event that prevents or overcomes the renal adaptation precipitates the hyperkalemia. Patients with hyperkalemia are usually asymptomatic, although some patients may present with generalized muscle weakness, and cardiac conduction may be impaired. The initial step with reported hyperkalemia is to ascertain whether it is associated with clinical symptoms or arrhythmias that would require prompt intervention. The urine potassium concentration, FEK, and TTKG can also be used to help distinguish between renal and nonrenal causes of hyperkalemia. Ascertain whether it is associated with clinical manifestations, such as arrythmias, which require immediate treatment.
A careful history and a physical examination should be done, especially looking for evidence of catecholamine surge, hypertension, vomiting or diarrhea, and medication use. Ascertain whether it is associated with clinical symptoms, such as changes on the ECG, which require immediate attention.
Careful history and physical examination should be done, looking specifically for chronic kidney disease, medications, dietary, or clinical situations associated with transcellular shifts, such as hyperglycemia. Batlle DC, Arruda JA, Kurtzman NA: Hyperkalemic distal renal tubular acidosis associated with obstructive uropathy. Elisaf M, Siamopoulos KC: Fractional excretion of potassium in normal subjects and in patients with hypokalaemia.
Ethier JH, Kamel KS, Magner PO, et al: The transtubular potassium concentration in patients with hypokalemia and hyperkalemia.
West ML, Marsden PA, Richardson RM, Zettle RM, Halperin ML: New clinical approach to evaluate disorders of potassium excretion. West ML, Marsden PA, Richardson RM, et al: New clinical approach to evaluate disorders of potassium excretion. The final and severest clinical manifestation is hepatic encephalopathy- the worsening of brain function due to liver failure. Liver failure is diagnosed based on clinical signs, medical history (alcohol, drugs, narcotics, hepatitis). Symptomatic treatment: controlling the bleeding- vitamin K and other drugs which increase blood coagulation. However, your blood sugar level is still unstable?You are following medication and insulin injection and you have spent a lot of money on it.
The blood sugar level drops while taking medicine, however, it rises again when stop medication? The four major properties of HKIII Bamboo Salt has helped to improved a lot of modern health problems.
About 90% of total body potassium is intracellular and 10% is in extracellular fluid, of which less than 1% is composed of plasma. Insulin increases Na+,K+-ATPase activity and facilitates potassium entry into cells, thereby averting hyperkalemia. In the steady state, K+ excretion matches intake, and approximately 90% is excreted by the kidneys and 10% in the stool. Transcellular shifts can occur in pathologic conditions associated with a catecholamine surge, such as chest pain syndromes, or mediated by acid-base disturbances.
Manifestations of hypokalemia include generalized muscle weakness, ileus, and cardiac arrhythmias. In the absence of compelling indications for immediate therapy, a careful history and physical examination should be performed.


Hypokalemic patients with a lower FEK would suggest extrarenal loss of K+, whereas hypokalemia from renal losses would be associated with an elevated FEK. The numerator is an estimate of the luminal potassium concentration, and the osmolality ratio is used to correct for the increase in UK caused by water extraction. Mortality data caused by hyperkalemia are unavailable for the general population but accounted for 1.9% of patients with end-stage renal disease in the United States in 1993.
The earliest electrocardiographic changes are tenting of T waves, followed by widening of the QRS complex, atrioventricular conduction block, ventricular fibrillation, and then asystole. If no compelling indication for immediate therapy exists, a careful history and physical examination should be performed, with particular emphasis on medication, diet, and chronic kidney disease. Intravenous calcium is required for rapid reversal of conduction abnormalities that are present. Other common causes are chronic and acute hepatitis, toxic materials (drugs and narcotics). During liver failure the blood stops clotting, that makes hemorrhages from varicose veins even more dangerous. Ultrasound examination might reveal an enlarged liver, liver cirrhosis, liver tumour or ascitis.
Usually it is impossible to remove the cause that is why liver failure treatment is usually symptomatic. However, the complication of diabetes is coming to you, suffering from top to toe, affecting your personal and family members' lives?You are on a very strict diet, you pick your foods carefully in order to control your blood sugar level? These two properties stimulate normal insulin secretion and improve the sensitivity of insulin receptor. They also keep the body’s chemical balance, help control blood pressure, and make hormones. Renal K+ excretion is mediated by aldosterone and sodium (Na+) delivery (glomerular filtration rate [GFR]) in principal cells of the collecting ducts.3 K+ is freely filtered by the glomerulus, and almost all the filtered K+ is reabsorbed in the proximal tubule and loop of Henle (Fig. In patients with ischemic or scarred myocardium, left ventricular hypertrophy, congestive heart failure, or myocardial infarction, hypokalemia is associated with an increased incidence of ventricular ectopy, ventricular tachycardia, and ventricular fibrillation. Important clinical clues such as medication, vomiting, and hypertension should be specifically sought. Moreover, in patients with chronic kidney disease (lower GFR), adaptive responses increase K+ excretion, with resultant increases in FEK and TTKG. Pseudohyperkalemia can occur with thrombocytosis, hemolysis, and extremely high white cell counts. Impaired K+ excretion or impaired potassium entry into cells accounts for all other causes of hyperkalemia. The serum potassium concentrations are poorly correlated to the electrocardiographic and conduction abnormalities. An exception to this is hyperkalemia caused by digoxin toxicity because acute hypercalcemia can potentiate the toxic effects of digoxin. The liver stops filtering blood and toxic materials enter the systemic circulation, thus intoxicating the whole organism, especially the brain.
Furthermore, the patients often experience hemorrhages from their nose, gums and even uterus (women).
The number of "Little Diabetes Patient" is increasing, diabetes is no longer the adult's problem, young people may have, too. People with kidney failure undergo either dialysis, an artificial blood-cleaning process, or transplantation to receive a healthy kidney from a donor.
Yet you can often prevent diabetic neuropathy or slow its progress with tight blood sugar control and a healthy lifestyle. 70-80 types of minerals and trace elementsLarge amount of medical experiments have shown that chromium is an important factor for insulin sensitivity. Factitious or spurious hypokalemia, which can occur in patients with leukemia or elevated white cell counts because K+ is taken up by these metabolically active cells in the test tube, should be ruled out. Therefore, the normal values will vary, making the interpretation of significance difficult. In these cases, lysis of the cells in the test tube releases potassium into the serum and increases potassium concentrations.
Acute therapy is also directed at rapidly moving potassium into cells with intravenous dextrose and insulin. The blood stasis and increased pressure in the hepatic vein makes blood plasma diffuse through the walls of the vein and accumulate in the abdomen.
Later, neurological symptoms are present- dysartria (impaired speech), impaired coordination and seizures. This process produces a buildup of toxic acids in the bloodstream called ketones, eventually leading to diabetic ketoacidosis if untreated. One regulates K+ excretion, or external balance through the kidneys and intestines, and the second regulates K+ shifts, or internal balance between intracellular and extracellular fluid compartments. This absorption in the proximal part of the nephron passively follows that of Na+ and water, whereas reabsorption in the thick ascending limb of the loop of Henle is mediated by the Na+,K+,2Cl- carrier (NKCC2) in the luminal membrane. If true hypokalemia is present, then determine whether it was caused by a transcellular shift or a decrease in total body potassium.
Repeated fist clenching with a tourniquet can also release K+ from muscle cells and increase potassium concentrations factitiously. A plasma K+ determination, instead of serum determination, is sometimes necessary in addition, spurious hyperkalemia will not be associated with abnormalities on the electrocardiogram (ECG).


It is not only improving your diabetes, but also your overall health.For 12 years, the customer feedback is the power which support us for so long.
Hypokalemia from transcellular shift is managed by treating the underlying condition or removing the offending agent. In hyperkalemic patients, a value greater than 10 suggests normal aldosterone action and an extrarenal cause of hyperkalemia. If hyperkalemia is indeed present, then determine whether it occurred with a transcellular shift or reduction in potassium excretion, or in the setting of preserved renal function. Longer term therapy for hyperkalemia without conduction abnormalities should be directed toward minimizing intake and increasing excretion of potassium. The definite results can only be expected when various type of minerals and trace elements are administrated.2. 1) in the cortical and outer medullary collecting tubule, and the papillary (or inner medullary) collecting duct via luminal potassium channels (ROMK). Magnesium depletion reduces the intracellular potassium concentration and causes renal potassium wasting; it appears to be caused by an impairment of cell membrane Na+, K+-ATPase (Box 2). Secretion in these segments varies according to physiologic requirements, and is responsible for most of the urinary potassium excretion.
Urine potassium, chloride, creatinine, and serum aldosterone levels are determined to distinguish the causes of extrarenal and renal losses of K+ so that the primary condition can be treated, in addition to replacement therapy (see Fig. Eradicate - The combined effects of far infra red, high alkalinity, multi-minerals and anti-oxidation improve energy metabolism. Secretion in the distal segments is also balanced by K+ reabsorption through the intercalated cells (see Fig. Strong Anti-oxidation Reduction Potential (ORP)Free radicals are everywhere, it can be generated when you are breathing.
It helps to improve hypertension, high cholesterol, viscous blood, shortness of breath, numbness of limbs, fatigue, blurred vision, itching, ulcers and other diabetic complications. You can take steps to keep your kidneys healthier longer: Choose foods with less salt (sodium). This process is mediated by an active H+,K+-ATPase pump in the luminal membrane and results in both proton secretion and K+ reabsorption. To clear up the free radicals, the HKIII Bamboo salt can help  due to its very high ORP level -426mV.
What the Kidneys Do CKD and My Health How does my health care provider know I have CKD?Chances are, you feel normal and were surprised to hear that you have CKD. If the blood circulation if poor, nutrients cannot be transported and utilized, at the same time, the waste and toxins cannot be excreted out of the body. A diagram illustrating a healthy kidney with albumin only found in blood, and a damaged kidney that has albumin in both blood and urine.
People with CKD often take medicines to lower blood pressure, control blood glucose, and lower blood cholesterol.
Two types of blood pressure medicines—ACE inhibitors and ARBs—may slow CKD and delay kidney failure, even in people who don’t have high blood pressure. Tell your provider about all the medicines you take, including over-the-counter medicines (those you get without a prescription), vitamins, and supplements. They can also develop anemia (low number of red blood cells), bone disease, malnutrition, and heart and blood vessel diseases.
You may delay dialysis if you follow your provider’s advice on medicine, diet, and lifestyle changes.
The donated kidney can come from someone you don’t know who has recently died, or from a living person—a relative, spouse, or friend. You may have a condition that makes the transplant surgery dangerous or not likely to succeed.
CKD and My Lifestyle People with CKD can and should continue to live their lives in a normal way: working, enjoying friends and family, and staying active.
Some points to keep in mind: A chart that lists diet tips to help slow down CKD Your provider may refer you to a dietitian. Soda and other drinks — If you are told to limit phosphorus, choose light-colored soda (or pop), like lemon-lime, and homemade iced tea and lemonade.
Dark-colored sodas, fruit punch, and some bottled and canned iced teas can have a lot of phosphorus. Juice — If you are told to limit potassium, drink apple, grape, or cranberry juice instead of orange juice. Drinking too much can damage the liver, heart, and brain and cause serious health problems. Cigarette smoking can make kidney damage worse.Take steps to quit smoking as soon as you can. Required fields are marked *CommentName * Email * Website Protected by WP Anti Spam Notify me of follow-up comments by email.



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