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First of all, there is no true answer to the question of which type of diabetes is genetic. Some examples to support this evidence is, that families who have type 2 diabetes usually share the same lifestyle such as eating habits, exercise habits, as well as being overweight, which has been known to be one of the leading causes of type 2 diabetes.
So to answer the question of which type of diabetes is genetic, the short answer is there is a high probability that type 2 diabetes will have a stronger predisposition to get it, compared to type 1 diabetes. In other words, there is a predisposition of getting both type 1 and type 2 diabetes, but the type 2 diabetes has a bigger chance to run in the same family.
While we are not sure which type of diabetes is genetic for sure, or what triggers type 1 or type 2 diabetes to occur if you have a family member who suffers from it, the most important thing is to know that prevention is key especially in type 2 diabetes since studies have shown that diet and regular exercise can prevent it from happening, or in some cases reverse the symptoms. Recent Commentspatrice thompson on Free Diabetic Supplies – How to Get Them?munnaamalai on Type 1 vs Type 2 Diabetes ChartJessica I. ANN ARBOR—New research from a large international team of scientists offers a more complete picture of the genes responsible for type 2 diabetes, demonstrating that previously identified common alleles shared by many in the world are the biggest culprits—not the less common variants some scientists had hypothesized might play a large role in who gets the disease. The researchers also identified a novel variant specific to East Asians through their study that analyzed the genes of individuals from five ethnic groups, making this the largest multi-ethnic genetic sequencing study published to date. Led by researchers at the University of Michigan School of Public Health, the Wellcome Trust Centre for Human Genetics at the University of Oxford, the Broad Institute of MIT and Harvard, and the Massachusetts General Hospital, more than 300 scientists from 22 countries used DNA from 120,000 individuals to pinpoint genes and their variants, which influence the disease that impacts 10 percent of the world's population. The team identified a novel association between type 2 diabetes and a variant in the gene PAX4, present only in individuals from East Asia, including Korea, China and Singapore. The researchers completed whole genome sequencing of more than 2,600 people and exome sequencing of 13,000, complemented with genome- or exome-wide array genotyping of 111,000 people. Those studied included individuals with ancestral origins in Europe, South and East Asia, the Americas and Africa.
This in-depth look at the genetics of type 2 diabetes addresses the century-old debate of whether genetic differences that influence predisposition to common diseases, like diabetes, are ones that are widely shared within populations, or whether they are more likely to be rare or unique events, specific to an individual and his or her family. Type 2 diabetes is a major threat to global public health as deaths from the disease continue to rise rapidly, along with its complications including blindness, kidney failure, heart attack, stroke and amputation.
Data and discoveries generated through this project are available through the type 2 diabetes genetics portal developed as part of the Accelerating Medicines Partnership.
This research was supported by more than 60 funding organizations with primary contributions from the National Institutes of Health (National Institute of Diabetes, Digestion and Kidney Disease, National Human Research Genome Institute and Office of the Director) and the Wellcome Trust. Marquez M, Huyvaert M, Perry JR, Pearson RD, Falchi M, Morris AP, Vivequin S, Lobbens S, Yengo L, Gaget S, Pattou F, Poulain-Godefroy O, Charpentier G, Carlsson LM, Jacobson P, Sjostrom L, Lantieri O, Heude B, Walley A, Balkau B, Marre M, Froguel P, Cauchi S; DIAGRAM Consortium. Cauchi S, Rung J, Albrechtsen A, Shen L, Rocheleau G, Cavalcanti-Proenca C, Bacot F, Balkau B, Belisle A, Borch-Johnsen K, Charpentier G, Dina C, Durand E, Elliott P, Hadjadj S, Jarvelin MR, Laitinen J, Lauritzen T, Marre M, Mazur A, Meyre D, Montpetit A, Pisinger C, Posner B, Poulsen P, Pouta A, Prentki M, Ribel-Madsen R, Ruokonen A, Sandbaek A, Serre D, Tichet J, Vaxillaire M, Wojtaszewski JF, Vaag A, Hansen T, Polychronakos C, Pedersen O, Froguel P, Sladek R. The next EGID International Symposium will be held on 29 and 30 November in Lille Grand Palais.

Researchers recently discovered genetic mutations that could reduce the risk of Type 2 diabetes. Reducing your risk of diabetes soon could be possible, if drug companies can build on groundbreaking genetic research and design the appropriate therapy.
Sometimes a loss-of-function mutation can cause a disease, such as cystic fibrosis, but others can protect against disease. Approximately 300 million people in the world have Type 2 diabetes, and those numbers are rapidly escalating. This is one of the most common questions asked by people who are diagnosed with diabetes, or with parents who have children with diabetes. Researchers have found that although there is a predisposition for someone to have a diabetes, there are other factors other than genetics that may trigger the diabetes to occur. If one twin get a diabetes, the other one will likely to have it as well, although the possibility differs depending on which type of diabetes it is.
This means that if you have a family member with type 2 diabetes, you have a bigger chance of getting it too if you do not take extra measures in preventing it from happening. What researchers have not found out is the triggers that might shed a clue to why some identical twins both get it, and some others do not, even though they have the exact same genes. The majority of these were common variants, found in all human populations, and most had previously been detected by other genome-wide association studies. They also demonstrated that variants in the gene TM6SF2, previously linked to hepatic steatosis (commonly known as "fatty liver"), influences risk of type 2 diabetes. Type 2 is the most common form of the diabetes; it impacts the body's ability to regulate glucose (or sugar).
Precision medicine relies on the deep understanding of the genetic architecture of a disease.
While this large range of genetic risk may challenge our efforts at precision medicine, our consortium also offers a publicly accessible dataset, unprecedented in scope, for researchers around the world to advance our molecular understanding of type 2 diabetes," said co-lead author Jason Flannick, senior group leader at the Broad Institute of Harvard and MIT and research associate at the Massachusetts General Hospital. The pathogenesis of T2D is characterized by two major features: peripheral insulin resistance and impaired insulin secretion from pancreatic beta-cells. Even if it is still necessary to identify the etiologic variants, additional genetic and epigenetic factors may help understandig the missing heritability.
A drug that mirrors the behavior of these mutations could prevent diabetes even in people at greatest risk for the disease. They specifically chose to include subjects who did not have Type 2 diabetes but had significant risk factors such as being older and overweight.
Glucose is made of the sugars and starches the body digests from food to give cells fuel, and "insulin takes the sugar from the blood to the cell," according to the ADA's website.

David Altshuler, deputy director and chief academic officer at the Broad Institute and a Harvard Medical School professor, said in a statement.
While these results could prove hugely significant for those at risk of developing the disease, locating a drug target is only one step in the process. As you probably already know, there are two type of diabetes, type 1 diabetes and type 2 diabetes.
The reason is because even though type 1 diabetes and type 2 diabetes has different causes, but there is a bigger chance for both types of diabetes to happen if some family members also have it. In addition to its genetic components, environment and behavior play major roles in who gets the disease, with obesity, excessive stress and an inactive lifestyle among the contributors.
Our work provides this key information for type 2 diabetes," said lead author Christian Fuchsberger, who worked on the research as a postdoctoral fellow at U-M and now is at the Center for Biomedicine at the European Academy in Bolzano, Italy. Westernization may largely explain the progressive increase in the prevalence of type 2 diabetes in the early part of the 20th century, particularly in developing countries. The researchers also included low-risk subjects like "lean" non-smokers, according to The New York Times. On the other hand, type 2 diabetes twin will have a bigger chance of 75%, if the other twin has it. However, if your family member suffers from type 1 diabetes, your probability of getting it is slightly lower compared to type 2 diabetes.
Combined with other risk factors such as smoking, high cholesterol and high blood pressure, people with type 2 diabetes have a much higher risk of cardiovascular disease. They focused their analysis on a set of genes that previous studies suggested might be critical in the development or prevention of diabetes, although earlier research conflicted over whether the genes were harmful or helpful. Reduced beta-cell compensation to the insulin resistance associated with sedentary lifestyle and central obesity can gradually lead to T2D. However, this is not a true evidence that this is purely genetic, because it is very common in families to share the same lifestyle which is probably causing someone to have type 2 diabetes. However, the risk to develop the disease is very different between individuals of the same ethnic group living in the same environment. Since 2007, genome-wide association studies have identified ~50 loci for the most part associated with beta-cell defect.

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