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28.03.2016
Excessive alcohol (ethanol) intake or alcohol abuse can result in many health problems and is implicated as a cause or aggravating factor for several skin conditions. Alcohol abuse has been defined as recurrent alcohol use where it impacts on work, school or home, or to the point it is physically dangerous, gets you into trouble with the law, or continues despite the problems it has created. Immune system: direct toxic effect on bone marrow, reduced number and function of T-cells, reduced survival of immunoglobulins. One of the earliest signs of alcohol abuse is a persistently red face due to enlarged blood vessels (telangiectasia). Transient flushing is also a common side effect of alcohol, particularly in heavy drinkers. Up to 40% of northeastern Asians experience flushing and elevated heart rate after drinking even minimal amounts of alcohol, due to accumulation of acetaldehyde. Large numbers of spider angiomas are associated with liver cirrhosis (scarring of the liver) due to elevated oestrogen levels. High pressure within the venous system in the liver leads to high pressure in the venous system elsewhere in the body including the veins around the umbilicus (belly button).
The skin and sclera of the eyes often turn yellow in patients with alcoholic liver disease. Skin darkening (hyperpigmentation) around the eyes, mouth and on the legs may be associated with chronic liver disease.
Generalised skin itching (pruritus) may occur due to the build up of poorly metabolised substances that stimulate nerve endings in the skin. Clubbing: the nail bulges out instead of dipping in slightly before it meets the skin at the root of the nail, resembling a club.
Muehrcke nails: white bands running parallel to the lunula (moon of the nail) with normal pink nail between the bands.
Porphyria cutanea tarda (PCT) results in photosensitivity, skin fragility, blistering, erosions, crusts, milia, scleroderma and increased hair growth (hypertrichosis) on sun-exposed sites such as face and hands. Alcohol is the most common cause of acquired or type 1 PCT in susceptible individuals and is associated with chronic liver disease. Other factors that may trigger type 1 PCT include oestrogen, iron and viral infections (especially hepatitis C). Along with increasing the risk of liver, pancreatic and breast cancer, alcohol increases the risk of skin cancer including squamous cell carcinoma, basal cell carcinoma, and melanoma. Alcohol suppresses the immune system and impairs adequate nutrition, reducing the body's natural defense against skin cancer. The effects of ultraviolet radiation may be enhanced by photosensitising byproducts of alcohol.


Nutritional deficiency can develop when alcohol replaces normal food in the diet and the digestive tract and liver do not digest and process food the way they should resulting in malabsorption. Vitamin A deficiency results in xerosis (dry skin) and follicular hyperkeratosis (rough follicles). Vitamin B2 (riboflavin) deficiency presents with angular cheilitis (cracked corners of the mouth), atrophic glossitis (inflamed tongue) and a rash on the face that resembles seborrhoeic dermatitis.
Vitamin C deficiency (scurvy) results in swollen gums, follicular hyperkeratosis and corkscrew hairs. Zinc deficiency causes a condition known as acquired acrodermatitis enteropathica with dermatitis around the mouth, hands, feet and anus. Heavy drinking reduces options for treatment of psoriasis, as some medicines are contraindicated if the drinking has led to liver disease (methotrexate) or to high levels of triglyceride (acitretin). Nummular or discoid dermatitis occurs more frequently in alcohol abusers, particularly in those with abnormal liver function tests.
Skin infections occur more frequently in patients who drink alcohol excessively due to impairment of the immune system, nutritional deficiency and increased trauma. Urticaria (hives) may occur within minutes to hours of drinking alcohol and is sometimes due to allergic reaction.
Some apparent allergic reactions to alcohol are due to inherited defects in alcohol metabolising enzymes or allergy to other contents of the drink such as colouring agents, preservatives or flavouring. Pain in the mouth, sometimes severe but most intermittent, on account of deep cracks or grooves on the surface of the tongue is the main symptom of Tongue Fissures.  It can be accompanied by tiny blisters on the tongue which may or may not have symptoms of geographic tongue as well. Sage is a well-known and popular remedy for mouth-related problems, be it gingivitis or inflammation and it also works for fissures.  The essential oil of this herb is useful in the treatment of fissures which have become inflamed with food remnants on which bacteria have acted. A mouth rinse made of Echinecea works as an effective remedy to soothe the tongue which is fissured.  It has good antifungal and antibacterial properties.
Goldenseal is another good herbal remedy for curing the tissues of the mouth.It acts in two ways – one by cooling the tissues in the mouth,  as well as by eliminating bacteria from the fissures on the tongue.
Scraping the tongue with a special oxygenated toothpaste is another natural method of clearing fissures in the tongue.
Taking supplements of riboflavin and folic acid is known to have a remedial action on burning sensation in the mouth, including soreness of the tongue. Other B vitamins are also helpful in alleviating this condition.  Peppermint tea,  brown rice and quinoa are other sources of B vitamins which help cure tongue fissures. This entry was posted in General Health and tagged Get Rid of Tongue Fissures or Cracks, Get Rid of Tongue Fissures or Cracks at Home, Get Rid of Tongue Fissures or Cracks naturally on October 25, 2011 by James Roberts. This appears because regulation of vascular control in the brain fails with sustained alcohol intake.


This is because of a mutation in acetaldehyde dehydrogenase (ALDH2), the enzyme that converts acetaldehyde to acetate. Blood vessels (the spider legs) radiate out in all directions from a central blood vessel (its body).
A study of 82 patients with liver cirrhosis showed significantly higher numbers of spider angiomas in alcoholic cirrhotic patients than non-alcoholic cirrhotic patients, indicating there may be an additional effect such as vasodilation to account for this difference. They are more common in women, especially during pregnancy, as they are influenced by the female hormone, oestrogen. This is also thought to be due to oestrogen, as it sometimes observed during normal pregnancy. The colour, known as jaundice, is due to bilirubin, a product broken down from haem derived from red blood cells. The angle between the nail plate and proximal nail fold is called the Lovibond angle and is normally less than 180° (indicating a dip and rise where the nail and skin meet). Porphyrins build up because of deficiency in uroporphyrinogen decarboxylase (UROD), an enzyme important in the sythesis of the blood protein haem.
In a case-control study of 175 people with rosacea and 145 people with normal skin, there was no significant difference in alcohol consumption between the two groups. The distribution of psoriasis has been observed to be particularly prominent on the fingers and hands of heavy drinkers.
Patients with psoriasis and high alcohol intake are also more likely to suffer from depression. Along with urticaria (see above), patients may develop low blood pressure, diarrhoea, shortness of breath and low heart rate (anaphylaxis).
Spider Angiomas in Patients with Liver Cirrhosis: Role of Alcoholism and Impaired Liver Function. People who have psoriasis and drink more than 80g of alcohol per week have been found to have more severe treatment-resistant psoriasis, including erythrodermic psoriasis. Even tiny amounts of alcohol may induce urticaria in people who have had a severe reaction previously, although allergy testing is often negative.



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