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Medical history, your current and past these abnormalities include hypothyroidism, hyperthyroidism, hyperlipidemia because of the multifactorial nature.

01.04.2015

Tinnitus one ear brain tumor, help for sleepless nights - How to DIY

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Explains about different types of brain tumor and its symptoms, treatments and surgical procedures. The Brain & Spine Centre Uttar Pradesh gives all the latest way to treat the brain tumor even if it is cancerous or non cancerous tumors.
Tinnitus is a symptom, not a disease, and it has a variety of causes that may arise anywhere in the hearing mechanism. A brain tumor can be either benign (non cancerous) or malignant (cancerous), primary, or secondary. Brain aneurysm (cerebral aneurysm) is caused by microscopic damage to artery walls, infections of the artery walls, tumors, trauma, drug abuse.
The Eustachian tube is a membrane lined tube that connects the middle ear space to the back of the nose. Ear was is a natural substance secreted by special glands in the skin on the outer part of the ear canal.
Cauliflower ear, or "Boxer's Ear" is caused by an injury to the ear, usually by blunt trauma from sports such as boxing, wrestling, or martial arts.
Objects or insects in the ear can be placed in the ear by patients themselves, or an insect crawling in the ear.
Adult brain tumors are diseases in which cancer (malignant) cells begin to grow in the tissues of the brain. The most common vessel to be involved is the carotid artery in the neck that carries blood from the heart to the face and brain. The internal carotid artery is inappropriately contacting the malleus of the middle ear, which is in close contact with the inner ear.
Tinnitus described as a fast, rhythmic clicking may be caused by involuntary twitching of muscles in the middle ear.
Exposure to loud noises, use of certain medications, and an underlying hearing loss are just some of the causes of ringing in the ears. Tinnitus associated with a sensation of ear fullness, dizziness, and hearing loss should raise suspicion for Meniere's Disease.
This form of tinnitus heightens a suspicion of a vascular etiology, meaning that the blood vessels around the inner ear may be causing the problem. Plaques within the carotid artery that build up over time can cause turbulent flow, and the pulsations can be transmitted to the inner ear. The tensor tympani and stapedius muscles are attached to the ossicles (bones) in the middle ear.
After exposure to loud noise over a period of time, it is not uncommon to have a temporary hearing loss associated with tinnitus. Most cases of tinnitus are subjective, but occasionally the tinnitus can be heard by an examiner.


A person viewing it online may make one printout of the material and may use that printout only for his or her personal, non-commercial reference. This causes the Eustachian tube, which helps equalize pressure in the ears, to repeatedly open and close. Pulsatile tinnitus may be associated with headaches, hearing loss, imbalance, and blurred and double vision. Otologic problems, especially hearing loss, are the most common causes of subjective tinnitus. Certain medications can also cause tinnitus, such as aspirin and quinine-containing medicines. Common causes of conductive hearing loss include external ear infection, cerumen impaction, and middle ear effusion. If the symptoms become troubling, a minor procedure done through the ear canal with division of the tendinous attachments to the ossicles is highly effective. Frequently, a hearing aid can help mask out the tinnitus so it becomes less noticeable and can also provide better hearing. Objective tinnitus usually is caused by vascular abnormalities of the carotid artery or jugular venous systems. Initial evaluation of tinnitus should include a thorough history, head and neck examination, and audiometric testing to identify an underlying etiology. Unilateral or pulsatile tinnitus may be caused by more serious pathology and typically merits specialized audiometric testing and radiologic studies. In patients who are discomforted by tinnitus and have no remediable cause, auditory masking may provide some relief. Epidemiologic data reveal that approximately one fourth of persons with tinnitus are discomforted by it, whereas the remaining three fourths experience the condition without significant symptoms.3Tinnitus takes different forms and has different classification proposals. One classification system stresses distinctions between vibratory and nonvibratory types, while another system groups the different forms of tinnitus into subjective or objective classes.Vibratory tinnitus is caused by transmission to the cochlea of vibrations from adjacent tissues or organs.
Nonvibratory tinnitus is produced by biochemical changes in the nerve mechanism of hearing.Subjective tinnitus, which is more common, is heard only by the patient.
Objective tinnitus can be heard through a stethoscope placed over head and neck structures near the patient's ear.The mechanism that produces tinnitus remains poorly understood. Tinnitus may originate at any location along the auditory pathway from the cochlear nucleus to the auditory cortex. Some leading theories include injured cochlear hair cells that discharge repetitively and stimulate auditory nerve fibers in a continuous cycle, spontaneous activity in individual auditory nerve fibers, hyperactivity of the auditory nuclei in the brain stem, or a reduction in the usual suppressive activity of the central auditory cortex on peripheral auditory nerve activity.4This article discusses the causes of subjective and objective tinnitus, and techniques used for evaluating tinnitus. Conductive hearing loss is caused by the inhibition of sound transmission to the inner ear.
It is continuous and less disturbing than the tinnitus of Meniere's disease.14Ototoxic medications or substances are another common cause of bilateral tinnitus.


Currently, almost every major group of medication includes one or more compounds with ototoxic properties (Table 2).2,10,15 Ototoxicity may affect hair cells, the eighth cranial nerve, or their central nervous connections. Temporomandibular joint disorder has been associated with vertigo and tinnitus, although the exact mechanism is unclear.Various metabolic abnormalities may be associated with tinnitus. These abnormalities include hypothyroidism, hyperthyroidism, hyperlipidemia, anemia, and vitamin B12 or zinc deficiency.Many patients with tinnitus exhibit signs of psychologic disorders. Although tinnitus may be a contributing factor to the development of depression, the common association of tinnitus and depression may be the result when depressed patients, particularly those with sleep disturbances, focus and dwell on their tinnitus more than patients who are without an underlying psychologic disorder.OBJECTIVE TINNITUSObjective tinnitus is rare. Patients with objective tinnitus typically have a vascular abnormality, neurologic disease, or eustachian tube dysfunction.4Patients with vascular abnormalities complain of pulsatile tinnitus. Arterial bruits may be transmitted to the ear from arterial vessels near the temporal bone. This type of tinnitus is a soft, low-pitched venous hum, which can be altered by head position, activity, or pressure over the jugular vein.4Congenital arteriovenous shunts are usually asymptomatic, while the acquired type often are associated with pulsatile tinnitus. Glomus tumor is a vascular neoplasm arising from the paraganglia around the carotid bifurcation, the jugular bulb, or the tympanic arteries. The symptoms may disappear with Valsalva's maneuver or when the patient lies down with the head in a dependent position.Evaluation of TinnitusHISTORYThe evaluation of a patient with tinnitus begins by taking a thorough history.
Precipitous onset can be linked to excessive or loud noise exposure or head trauma.LocationUnilateral tinnitus can be caused by cerumen impaction, otitis externa, and otitis media.
Tinnitus associated with unilateral sensorineural hearing loss is the hallmark of acoustic neuroma.PatternContinuous tinnitus accompanies hearing loss. The cranial nerves should be examined for evidence of brain-stem damage or hearing loss.9 Auscultation over the neck, periauricular area, orbits, and mastoid should be performed. Tinnitus of venous origin can be suppressed by compression of the ipsilateral jugular vein.Specific testing for sensorineural or conductive hearing loss is the next part of the examination. The sound lateralizes to the opposite ear in patients with a sensorineural hearing loss, but to the same side in those with a conductive hearing loss.
Patients with normal hearing or equal deafness in both ears hear the sound at the same level in both ears.In the Rinne test, the tuning fork is placed against the mastoid process to measure the conduction of sound by bone. Patients with unilateral or pulsatile tinnitus are more likely to have serious underlying disease and typically merit referral to an otolaryngologist.2,5 A full clinical evaluation should precede radiologic studies. Because pulsatile tinnitus suggests a vascular abnormality, the preferred imaging study is contrast-enhanced computed tomography (CT) or magnetic resonance imaging (MRI) of the brain21 (Figure 2).



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