Treatment of painful bladder syndrome and interstitial cystitis

Interstitial Cystitis or Painful Bladder Syndrome (PBS) affects millions of women around the world. Mainstream medicine’s approach mainly deals with medications to alleviate symptoms and of course come with unwanted side-effects. This natural remedy for painful bladder syndrome has been around for hundreds of years, but only until recently has medical research refined the application to achieve better results.
I had been suffering from Interstitial cystitis for around 5 years and it had progressively worsened, recently I’d had a particular bad bout and was in a lot of pain.
So now I use the QF28-3 Q magnet as needed, if it’s practically bad the QF28-6 is stronger and seemed to work better.
We have a Q magnets Treatment Protocol for PBS and a Promo Code for a special offer for the PBS Q magnets Pack. Enter your name and email address below to receive our physiotherapy approved treatment plan for Painful Bladder Syndrome. The minimum sized device we recommend for treating PBS is the QF28-3, which sells for $69.00 (Australian Dollars). Now, if you listen to the skeptics, you might think there is no evidence for this type of treatment. The women that wore the active magnet had on average a 53% reduction in pain, while those wearing the placebo only a 15% reduction.
Dr Eccles is a medical doctor and saw the positive results with patients in his own practice. What’s more, Q magnets when cared for will last for decades so they can be used routinely for all sorts of current and future injuries and pain such as bruising,  shin splints, hamstring tears and low back pain.
Before using Q magnets for interstitial cystitis, please ensure you have seen a medical doctor or even a specialists such as a urologist and have a professional diagnosis. Do not wear Q magnets near sensitive medical equipment or implants such as pacemakers, dorsal column stimulators, infusion pumps, or any other magnetically programmable medical devices.
If you are uncertain if these contraindications apply to you, consult your health care professional prior to use. 12 May , 2012Positioning Neuromagnetics in the “window of effectiveness” for magnetic therapy. Correspondence Address:Lakshmi VasAshirvad Institute of Pain Management and Research, Shubh Ashirwad Building, 1st Floor, Plot No. Crohn's disease Crohn's disease causes Crohn's disease symptoms Crohn's disease Tests and DiagnosisDr.
Arslan Malik is noted health blogger, public health activist, aspiring dreamer and avid reader. You may have no complains apart from an incidental finding of blood in the urine at your routine medical or insurance check up.
Q magnets are a break through as a static magnet medical device to provide targeted pain relief to the treated area. Over the past few years I’d been using Q magnets for pain relief on my back and sore muscles and it occurred to me to try it on my bladder.

I’ll sometimes tape it over the bladder area with sports tape and leave it on for a few days. To our knowledge, there is no published research investigating the treatment of bladder pain with magnetic field therapy. A Randomized, Double-Blinded, Placebo-Controlled Pilot Study to Investigate the Effectiveness of a Static Magnet to Relieve Dysmenorrhea. While purchasing the larger devices (such as QF28-3) might be a little more expensive up front, it provides greater flexibility for future use. Q magnets are not a cure for PBS, but can be a very usful non-drug and non-invasive method for providing natural pain relief.
These factors include heredity factors, environmental factors, infectious agents and immunological factor. Arslan MalikTags Crohn's disease Crohn's disease causes Crohn's disease symptoms Crohn's disease Tests and DiagnosisAbout Dr. With doctor in pharmacy, he has worked closely with various health organizations, multinational pharmaceuticals and community health programs.
Alternatively if I’m going to need to wear it longer, I use the Flux Plate Keeper to attach it onto my underwear, which is a good alternative if you have sensitive skin. Enter your details so we can send you the details of the special offer and you will also receive the PDF physiotherapy approved treatment protocol. The closest related study is by a UK doctor, Dr Eccles in 2005 which looked at menstrual pain. We surmised her pain, and irritative voiding to be secondary to constant straining against a dysfunctional pelvic floor.
Beside his professional practice, he has an avid interest in writing and teaching Physiology and Medicines. He covers variety of topics from Nutrition and Natural Care to Diseases, Treatments, Drug Interactions, Preventive Care and Clinical Research. This was a randomized, double-blind placebo controlled postal questionnaire trial that followed 35 women with menstrual pain.
Botulinum toxin type A injection into pelvic floor muscles appeared to address their dysfuction. A lady of 52 years was referred to us by a urologist with a diagnosis of ICS for pain management.
In men and women weighing more than 55 kg, a single capsule of 75 mg pregabalin is prescribed. In the absence of side-effects like dizziness and excessive somnolence, the dose may be escalated to a twice-daily regimen after 5 days. After five more days, if the pain persists at a level greater than five on Numerical Rating Scale NRS, we increase the dose to three-times daily.
The starting dose of amitryptyline used is 5 mg at night, which may be increased to 10 mg if the patient is still in pain after 5 days.Pregabalin 37 mg was advised initially and amitriptyline 10 mg was added 1 week later according to our protocol.
After 1 month of neuromodulators, she showed a favorable response [Table 1], but opted for CCEI and botulinum toxin A injection because her social life continued to revolve round the severe restraints posed by her frequent distressing attempts to void.

Increase in voided urine volume to 500 mL was decided as an end point for terminating the CCEI.The interventions were carried out in an operation theater with appropriate monitoring (oximeter, ECG, non-invasive blood pressure) and antibiotic cover with intravenous cefuroxime 750 mg.
The patient was advised oral antibiotics and was instructed to follow-up on an alternate day basis and take care of the caudal epidural infusion and its discontinuation in case of urinary retention.
The CCEI catheter was prematurely removed even though the catheter insertion and tunnel exit sites appeared normal, because it was placed in the central neuraxis.
At this time, all parameters like pain, frequency (from 15-30 min to 2-4 hr) and urine volume (from 150 to 400 mL) had improved.One week after CCEI termination, Botulinum toxin A injection was given intramuscularly into the perineal muscles (external urethral sphincter -70 units, 5 units each into the ischiocavernosus, bulbospongiosus and transverses perinei muscles on both sides under topical analgesia with lignocaine jelly). The patient continued to maintain the improved parameters after catheter removal and 15 days after botulinum toxin A, she reported effortless voiding. The clinical details and findings of the urodynamics studies before and after treatment are shown in [Table 1]. Physical therapy, oral medications (amitryptiline, cimetidine or hydroxyzine, pentosanpolysulfate), bladder instillations (DMSO, heparin or lidocaine) are considered if the first-line treatment fails. Treatment of Hunner's ulcers and hydrodistention (low pressure, short duration) are applied for refractory cases. The next line of management would involve neuromodulation (sacral or pudendal nerve block) and Cyclosposine A or Botulinum toxin injection (BTX-A) into the bladder. Ultimately, if all other forms of treatment fail, surgical intervention may be the last resort in the form of urinary diversion or augmentation cystoplasty.However, there is an emerging consensus as to the central role of epithelial dysfunction, bladder sensory nerve up-regulation and mast cell activation in the genesis of IC.
The importance of the central nervous system and PFD in the pathogenesis and clinical manifestations of IC is now recognized. We assumed that peripheral sensitization of bladder C fibers was triggered by the ongoing pain from neuroinflammation and straining against an unrelenting obstruction due to the pelvic floor neuromuscular dysfunction. Peripheral hyperexcitability of pain fibers led to persistent activation of the spinal cord neurons, which evolved to be centrally maintained. Progression to central sensitivity led to the development of a generalized irritable bladder that could not hold a normal volume of urine (reduced capacity) and developed irritative voiding symptoms.The response of our patient to the neuromodulator pregabalin emphasized the contribution of neuropathic mechanisms in her symptoms.
This was confirmed by resolution of pain, frequency, urgency and nocturia after CCEI, which attenuated the peripheral and central sensitization.
However, the persistence of straining indicated that obstructive mechanisms from the dysfunctional pelvic floor muscles were still operational.
We addressed this with pelvic floor botulinum toxin A injection rather than intravesical botulinum toxin A. This combination therapy addressed the two issues separately, with the belief that pelvic floor muscle spasm was the primary issue leading to peripheral and central sensitivity.
This combination has been developed from our prior experience in the past decade in several patients.The concept of peripheral and central sensitization in visceral pain has been reported.

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