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The container provides insulation from the immune system, which would normally treat the cells as intruders and work feverishly to eradicate them. The therapy would eliminate the need for recurrent insulin injections and blood sugar testing. ViaCyte, who is conducting the human trials, was able to obtain private, exclusive rights to 145 patents and 565 pending patent applications through diabetes research firm Janssen BetaLogics, a Johnson & Johnson company. Type 1 diabetes patients cannot produce insulin, a hormone that converts sugar into energy, as all the beta cells in the pancreas have been destroyed by the immune system.
In a related Inquisitr report, researchers with MIT and Harvard working on a cure for diabetes found that implanting insulin-producing beta cells into mice restored insulin levels back to normal.
Roughly 29.1 million Americans have diabetes, according to the American Diabetes Association.
A new device being tested by ViaCyte may someday eliminate the need for daily insulin shots by Type 1 diabetes patients. Within 12 weeks of the implant, the capsule attached to nearby blood vessels as intended and new insulin-producing cells have been multiplying. Another round of testing is planned with a new set of patients who will receive the same cell dose. Even if the Johnson & Johnson diabetes cure works in the lab, the device will still need to be approved by regulators. In past research projects, scientists have tried introducing new substances in the body to treat diabetes, but the sufferers' immune systems have always rejected them. About 27 million people have Type 2 diabetes and 1 million have Type 1 diabetes, according to the American Diabetes Association. Non -alcoholic fatty liver disease (NAFLD), as the name implies, refers to the infiltration of fat in the liver that is NOT due to excessive alcohol use. This fatty accumulation is known as steatosis, and NAFLD is sometimes also called non-alcoholic steatosis. If inflammation develops in the fatty liver, the condition can progress to non-alcoholic steatohepatitis (NASH).  NASH is the most extreme inflammatory form of NAFLD, and is a major cause of cirrhosis of the liver. There is also a growing body of work in animal models of fatty liver disease that suggests a so-called two-hit hypothesis.  In this theory, the first hit is the fatty liver (steatosis).

Regardless of the cause, the bottom line is that the single most effective treatment for obese people with NAFLD or NASH is to simply lose weight through diet and exercise.
Should something go wrong, the protective capsule holding the stem cells, which is mostly flat and smaller than a business card, can be quickly removed.
Once the clinical trials are complete and shown to be working, Johnson & Johnson hopes to have the treatment available sometime in the near future. Normally, after a person eats a meal, the cells make insulin to balance out rising sugar levels in the blood. If diabetes is left untreated, health complications like blindness, kidney failure, heart disease, amputations, and even death can occur. About a dozen people with Type 1 diabetes were implanted with a capsule, named VC-01, which released a small dose of insulin-producing cells.
Sometime late this year, a third round of testing will involve Type 1 patients getting a larger dose of the cells through the device.
Their current research project involving embryonic stem cells is ambitious, but one scientists believe could lead to a cure. Tom Donner, director of the diabetes center at Johns Hopkins University School of Medicine, according to ABC Channel 9 News. In this new study, scientists place embryonic stem cells in a Petri dish and provoke them to turn into insulin-producing cells.
Koch Institute for Integrative Cancer Research, in collaboration with scientists at Harvard Stem Cell Institute (HSCI), created the implantable device which can last up to six months.
Another 86 million have prediabetes, which means that their glucose levels are not normal, but not high enough to be diabetes yet. The basic cause of NAFLD is the insulin resistance that decreases the effect of insulin, and is typically found with prediabetes-metabolic syndrome and diabetes.
The presumptive diagnosis of NAFLD or NASH is made in people who are insulin resistant, have mildly elevated liver enzymes (transaminases) in the blood, and have signs of fatty liver on an ultrasound study. The exact reasons and mechanisms by which the disease progresses from one stage to the next are not known.
One theory is that the simple process of the accumulation of fat in the liver eventually leads to the development of NASH. Granted this is not an easy undertaking , however weight loss is achievable if you set your mind to it.

Reportedly, the capsule emulates the pancreas by manufacturing insulin for diabetic patients who cannot make it on their own. Too much insulin can lead to very low blood sugar and also kill a person, especially children. While people with Type 1 cannot make insulin, Type 2 patients can make insulin; but, the body does not use it efficiently.
Patients must adhere to a strict diet, exercise frequently, inject insulin multiple times daily, and test blood sugar levels through finger pricks several times a day. These patients will have no other known cause for these liver enzyme elevations or for fatty liver, along with no significant alcohol use. According to this theory, the large amount of fat in the liver is thought to be a source of peroxidation that results in the generation of free radicals. The number of Type 2 cases is exploding worldwide due to higher obesity rates and inactive lifestyles. Many people know that insulin is important for blood sugar regulation, but insulin also plays an important role in blood fat (lipid) management. If weight loss results in a decrease or normalization of elevated liver enzymes, the diagnosis of NAFLD is likely. Ask your doctor what you can do regarding lifestyle changes as part of managing this problem.
Only a liver biopsy, however, can confirm the diagnosis of NAFLD and NASH and determine the severity of the disease. The result of these changes is an increased uptake (infiltration and accumulation) of triglyceride fat into liver cells. Whether or not it is important to do a liver biopsy in people with suspected NAFLD or NASH is still debated among liver specialists since no specific treatments are available. The triglycerides are absorbed from the diet as well as channeled from abdominal fat and peripheral muscles to the liver. Simple fatty liver is not overly harmful and disappears with weight loss, however a minority of those affected go on to develop non-alcoholic steatohepatitis (NASH), which is the more harmful inflammatory stage of NAFLD.

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