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August 11, 2013 By Marina Gutner The diabetes epidemic is comparable and has a lot in common with thyroid disease: millions have it and even more people remain undiagnosed. Both hypothyroidism and diabetes are growing at an alarming rate and with the current state of medical care that only manages the symptoms and doesn’t offer a cure, the situation only gets worse. The truth is that there are many doctors and medical professionals who successfully reverse diabetes using alternative nutritional treatments that work for up to 90% of diabetics.
There are some risk factors such as age, heredity and ethnicity that can make you more prone to diabetes. What many people don’t know is that hypothyroidism affects how we process carbohydrates and predisposes us for diabetes and other blood sugar related health conditions such as pre-diabetes, metabolic syndrome X and insulin resistance.
Many thyroid patients are surprised and even angry when they hear that lack of thyroid hormones contribute only to 10% of body weight and most of it due to salt and water retention. Undiagnosed, untreated, undertreated and subclinical hypothyroidism causes insulin resistance and carbohydrate metabolism disorders which are responsible for most of the weight gain in thyroid patients. Thyroid hormones directly control insulin secretion and both hyper- and hypothyroidism impair carbohydrate metabolism.
If you are hypothyroid and don’t get an optimal thyroid treatment you are setting yourself up for blood sugar related health problems in the future such as diabetes, weight gain and heart disease.
Your first step to improve your carbohydrate metabolism and prevent more weight gain is to get a proper thyroid diagnosis and optimize your thyroid labs for TSH around 1.0 and free T4 and T3 at high normal. Type 1 diabetes is an associated disease with Hashimoto’s disease which is the main cause of hypothyroidism in industrialized countries.
Type 1 diabetes is a deficiency of the pancreas when the beta cells of the pancreas get damaged as the result of an autoimmune attack and can no longer produce insulin.
In Type 2 diabetes as opposed to type 1 diabetes, the pancreas works in overdrive producing the insulin to compensate high glucose levels in the blood.
Weight gain often is the only indicator of metabolic imbalance because the blood sugar begins to rise only after the pancreas cannot keep up and override the cellular resistance to insulin. A waist circumference is greater than 102 cm (40 in) in men and 88 cm (35 inches) in women. The good news is that there is a new diabetes treatment where the same principles apply to the reversing of insulin resistance, metabolic syndrome X, pre-diabetes and diabetes type 1 and 2. As of today conventional medicine doesn’t offer a cure for diabetes and sets the main goal of the treatment to keep blood sugar levels in the normal range to prevent any possible complications. 3 meals, 2-3 snacks in between of the meals and never skip the meal to prevent hypoglycemia or low blood sugar levels.
Meglitinides helps achieve lower blood sugar by forcing the pancreas to produce more insulin immediately after the meal. Alternative diabetes treatments are often based on the fact that the intake of carbohydrates and sugars causes higher insulin response and blood glucose levels than eating proteins and fats. Diets low in carbohydrates such as Atkin’s and low-carb version of Paleo diet can provide some great benefits and improvements of carbohydrate metabolism in people who have insulin resistance because this way of eating addresses both glycemic load and satiety.
The basic principle is to treat the weakness by limiting how much glucose enters into the body so that the pancreas doesn’t need to perform at their optimum and the blood sugar doesn’t rise so much. While this approach works and it can help with weight loss, insulin and glucose management and overall health, it doesn’t fix the main problem and doesn’t provide the ultimate solution or cure for diabetes.
Humans have a highly adaptable glucogenesis and with an absence of carbs in the diet your body starts to convert more protein and fats into sugar to support the brain’s demand in glucose for healthy functioning. Any dieting and low-carb diet in particular are stressful for your body and adrenal glands that lead to higher cortisol levels. Raising blood sugar levels and insulin is a normal physiological response to food that we eat. There is a new scientifically proven diabetes treatment that can help significantly reduce the need for insulin for type 1 diabetics and reverse type 2 diabetes, pre-diabetes and insulin resistance. It works if you were recently diagnosed or have been suffering from diabetes for a long time. Legal:All publications and any information provided on this blog is for informational and educational purposes only, and should not be used as an alternative or a substitute for professional medical advice, diagnosis, or treatment. Diabetes symptoms mayo clinic ] ,diabetes symptoms mayo clinic if include diabetes type 2 then generic januvia can help you you to choose from.. Eliminate diabetes type 2 diabetes symptoms mayo clinic treatment of diabetes diabetic association: there is a cure for diabetes type 2 diabetes symptoms mayo clinic. High blood sugar noticeable symptoms december 7, 2012 dear mayo clinic: high blood sugar noticeable symptoms.
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Honey and Type 1 Diabetes MellitusMamdouh Abdulrhman1, Mohamed El Hefnawy2, Rasha Ali1 and Ahmad Abou El-Goud1[1] Pediatric Department, Faculty of Medicine, Ain Shams University, Abbasia - Cairo, [2] National Institute of Diabetes, Cairo, Egypt1. Jenkins (1987) defined the glycemic index as the ratio between the blood glucose areas produced after ingestion of a studied sugar compared to the blood glucose area produced after glucose ingestion itself. Insulin pumps also known as continuous subcutaneous insulin infusion (CSII)  provides an alternative to insulin injections.


Insulin pump is smaller than a mobile phone and delivers a steady flow of short acting insulin like humalog, apidra or novorapid round the clock. The pump delivers a varied dose of insulin continually which can be adjusted based on your activity and food intake. You will still need to check blood glucose more frequently but with newer pumps even this could be avoided.
584 x 822 jpeg 143kB, 404 (Page Not Found) Error - Ever feel like you're in the wrong place? Copyright © 2015 Caroldoey, All trademarks are the property of the respective trademark owners. However, regardless of the risk factors, complications of diabetes can be very dangerous and include heart and kidney disease, blindness, nerve damage, gastroparesis, infections, ulcers, teeth and gum disease.
All these conditions result in blood glucose levels that are higher than normal and contribute to the stubborn weight gain and inability to lose weight in hypothyroid patients. According to a study conducted by the Endokrinology and Diabetology Depatment of Collegium Medicum University, Poland on patients with Hashimoto’s disease, advanced carbohydrate metabolism disorders such as type 1 and type 2 diabetes were confirmed in half of Hashimoto’s patients.
It often occurs as a part of Polygrandular syndrome type II or Schmidt’s syndrome which is a combination of adrenal insufficiency, Hashimoto’s disease and type 1 diabetes.
It develops slowly and starts on the cellular level with insulin resistance that can continue for many years. Since people with type 1 diabetes don’t produce enough insulin, the use of insulin shots can help lower the blood sugar by moving the sugar from the blood into the cells. All carbohydrates convert into sugars eventually promoting weight gain, high blood sugar and heart disease.
It can work especially well for those who have low insulin output due to the burnout of the beta cells in their pancreas (predisposition to type 1 diabetes).
There is other more healthy and sustainable way to not just improve your carbohydrate tolerance by avoiding carbs but repair and restore your ability to process carbohydrates. It is based on studies that confirmed that this treatment works twice better than metformin as well as doesn’t have any of the side effects or problems.
Hashimoto’s thyroiditis and carbohydrate metabolism disorders in patients hospitalised in the Department of Endocrinology and Diabetology of Ludwik Rydygier Collegium Medicum in Bydgoszcz between 2001 and 2010. Metabolic syndrome prevalence in subclinic and overt hypothyroid patients and the relation among metabolic syndrome parameters. Effects of treatment with L-thyroxin on glucose regulation in patients with subclinical hypothyroidism.
Insulin sensitivity and counter-regulatory hormones in hypothyroidism and during thyroid hormone replacement therapy. C-peptide following equivalent amount of glucose, sucrose or honey in non-diabetics.Table 1.
Introduction Type 1 diabetes mellitus is by far the most common metabolic and endocrinal disease in children (Peters & Schriger, 1997). ResultsNo significant difference was found between patients (diabetics) and controls (non-diabetics) as regards the age and anthropometric measures (table 1). He stated that the glycemic response to food affects the insulin response which in turn is also potentiated by other non-glucose dependent factors in this food (Ostman et al., 2001). Insulin pumps are suitable for people with Type 1 diabetes however recent studies show that insulin pumps may also benefit people with Type 2 diabetes.
This insulin is delivered through a very fine tube that runs from the pump to a cannula, which is inserted under the skin. Our diabetes specialist nurse, dietitian and consultant diabetologist will support you to achieve better blood glucose control and also improve your quality of life.
During this time both your weight and fasting glucose levels gradually increase as the condition progresses to metabolic syndrome X, pre-diabetes and diabetes.
A suggested solution to diabetes is a low-carbohydrate diet when the person is recommended to avoid sugars and carbs all together.
I am a researcher, medical writer, Author of forthcoming series on thyroid, hypothyroidism and autoimmune disease. The major dietary component responsible for fluctuations in blood glucose levels is carbohydrate.
Samnata et al (1985) defined the peak incremental index of a certain sugar as the ratio between the maximal increments of the glucose level after ingestion of the sugar compared to the maximal increment produced after ingestion of glucose.
The chronic hyperglycemia of diabetes is associated with long-term damage, dysfunction and failure of various organs especially the eyes, kidneys, nerves, heart and blood vessels (American Diabetes Association, 2001). He also mentioned that both the glycemic and the peak incremental indices are closely related, highly dependent and positively correlated to the plasma glucose produced after ingestion of any given sugar. The glycemic effect of any foodstuff is defined as its effect on blood glucose level postprandially. Therefore, any change in the plasma glucose level after ingestion of a certain sugar will markedly affect both the glycemic index and the peak incremental index. Both the glycemic index (GI) and the peak incremental index (PII) are used to assess the glycemic effect of different food stuffs (Jenkins et al., 1981). Hence, the glycemic and the peak incremental indices measure how fast and how much a food raises blood glucose levels.


Jennie et al (2003) who studied the use of low glycemic index diets in the management of diabetes found that diets with low glycemic indices (GI), compared with conventional or high-GI diets, improved overall glycemic control in individuals with diabetes, as assessed by glycemic index, peak incremental index, reduced HbA1c and fructosamine. Foods with higher index values raise blood sugar more rapidly than foods with lower index values do in case of the glycemic index and much more in case of peak incremental index. They concluded that using low-GI foods in place of conventional or high-GI foods has a clinically useful effect on postprandial hyperglycemia similar to that offered by pharmacological agents that target postprandial hyperglycemia.
In our study, no statistically significant differences were found between diabetic patients and non-diabetic controls regarding the glycemic and the peak incremental indices of the studied sugars.
Similarly, the American Diabetes Association (2002) stated that the use of low-GI foods may reduce postprandial hyperglycemia.Honey is the substance made when the nectar and sweet deposits from plants are gathered, modified and stored in the honeycomb by honey bees. On the other hand, both the GI and PII of honey were significantly lower when compared with sucrose in patients and controls. Similarly, Samnata et al (1985), who studied the glycemic effect of glucose, sucrose and honey in 12 normal volunteers, eight patients with insulin-dependent diabetes mellitus (IDDM) and six patients with non-insulin-dependent diabetes mellitus (NIDDM), found no significant differences between the normal volunteers and diabetic patients regarding the glycemic and peak incremental indices of both sugars. It is composed primarily of the sugars glucose and fructose; its third greatest component is water.
Since the glycemic index (GI) is the ratio between the area under curve (AUC) of the studied sugar and the AUC of glucose, and the peak incremental index (PII) is the ratio between the maximal blood glucose increment of the studied sugar and that of glucose; it may be expected that both GI and PII will be the same in both diabetics and non-diabetics.
Glucose and fructose, the major constituents of honey, account for about 85% of the honey solids. Also, Shambaugh et al (1990) found that sucrose caused higher blood sugar readings than honey in normal volunteers.
In the study of Samnata et al (1985), honey ingestion in both diabetics (IDDM) and non-diabetics also resulted in a significantly lower PII compared to the glucose and sucrose.
The principal oligosaccharides in blossom honeys are disaccharides: sucrose, maltose, turanose, erlose. In the study done by Al-Waili (2004), honey compared with dextrose and sucrose caused a lower elevation ofplasma glucose levels (PGL) in both diabetics (IDDM) and normal subjects.
Also, Watford (2002) demonstrated that very small amounts of fructose, which is the main component of honey, could increase hepatic glucose uptake and glycogen storage, as well as reduce peripheral glycemia which could be beneficial in diabetic patients.
In the study performed by Agrawal et al (2007), honey was found to produce an attenuated postprandial glycemic response especially in subjects with glucose intolerance. They referred these results to the possibility that the glucose component of honey might be poorly absorbed from the gut epithelium. Also, Tirgoviste et al (1983) studied blood glucose and plasma insulin responses to various carbohydrates in type 2 diabetes, and they found that the increase in plasma glucose was significantly higher after administration of more refined carbohydrates such as glucose than after the complex ones such as honey. Meanwhile, Oizumi et al (2007) and Arai et al (2004) found that consumption of a palatinose (a disaccharide found in honey)-based balanced formula suppressed postprandial hyperglycemia, glycemic and peak incremental indices and produced beneficial effects on the metabolic syndrome–related parameters (namely, the lipid profile and visceral fat accumulation) in diabetic patients.
Measurement of C-peptide, however, provides a fully validated means of quantifying endogenous insulin secretion.
They stated the reason of this observation to be due to the fact that although palatinose is completely absorbed, yet it has the specific characteristics of delayed digestion and absorption as reported by Dahlquist et al (1963) and Lina et al (2002).
C-peptide is co-secreted with insulin by the pancreatic cells as a by-product of the enzymatic cleavage of proinsulin to insulin. Our results showed that honey, compared to glucose and sucrose, caused a significant elevation in the C-peptide levels in non-diabetic subjects. Meanwhile, in diabetic patients, the plasma C-peptide levels did not differ significantly between the three types of sugars.
Aim of the studyThe aim of this work was to compare the effects of honey, sucrose and glucose on plasma glucose and C-peptide levels in children and adolescents with type 1 diabetes mellitus.3.
To our knowledge, no similar work was done to study the effects of honey on C-peptide levels in type 1 diabetes mellitus. However, several studies were performed in healthy and in type 2 diabetic patients to evaluate the effects of honey on the insulin and C-peptide levels, and the results were controversial.
In the study of Al Waili (2003), inhalation of honey solution, when compared with hyperosmolar dextrose and hypoosmolar distilled water, resulted in a significant elevation of plasma insulin and C-peptide in both normal individuals and in patients with type 2 diabetes mellitus.
All subjects were within 68–118% and 77–125% of their ideal body weight and height, respectively. The patients were recruited from the regular attendants of the children clinic of the National Institute of Diabetes in Cairo, Egypt. The study was approved by the local ethical committee, and an informed written consent was obtained from at least one parent of each subject before the study. The honey dose for each patient was calculated based on the fact that each 100 gm of the honey used in this study contained 77.3 gm sugars. Venous blood was sampled just before ingestion and then every 30 min postprandial for 2 h thereafter.



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Comments

  1. Dusty

    Options out there and so it tends to be a favored celiac disease, gluten intolerance, or a wheat allergy.

    29.04.2016

  2. Aviator

    Any butters and oils made high protein, low carb weight from different sources (i.e.

    29.04.2016

  3. RuStam_AhmedLi

    Researchers are working with folks whom have a history that factors most likely.

    29.04.2016

  4. shahrukhkhan

    People need to restrict their carbohydrate however steadily.

    29.04.2016