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Causes and treatment of pitting edema unilateral,healthy food north edmonton,zinc overdose erectile dysfunction - New On 2016

Hand edema, or “puffy hand syndrome,” is seen frequently in primary care practice, and given its broad differential diagnosis (Table A Table B), presents physicians a significant diagnostic challenge. Figure 1 – A former heroin addict with hepatitis C virus infection has bilateral “puffy hands.” The hand edema begins as pitting edema, followed by thickening and fibrosis of the subcutaneous tissue. Immobility due to surgery or any other causes can force fluid pressure on the immobile part of the body. Varicose veins can cause edema that develops due to increased fluid pressure from the veins. Liver cirrhosis or kidney failure or gastrointestinal problems like ulcerative colitis, or Crohna€™s disease can cause pitting edema. Compression of large veins or lymph nodes or undergoing hip replacement surgery or local infection on the hand or feet can cause generalized edema. Blood vessels of the body may sometimes leak out fluid into the tissues nearby this leads to swelling of organs causing edema. With suitable treatment methods and controlled diet and persistent follow ups one can overcome the edema or swelling of tissues. When gentle pressure is applied on the affected part, it creates identification on that area.
Suitable medications and treatments are given if edema occurs due to liver or kidney problems.
Edema occurs due to leakage of fluid into the tissues thereby accumulating more fluids in the same area. In addition, the history can be instrumental in ruling out the more common rheumatologic causes, such as rheumatoid arthritis (RA), gout, and pseudogout.
The latter resulted in repeated exposure to HCV infection (and subsequent development of chronic liver disease). Hepatitis C virus prevalence and outcomes among injecting drug users on opioid replacement therapy. Upper-extremity deep vein thrombosis after central vein catheterization via the axillary vein. The diagnosis and treatment of peripheral lymphedema: consensus document of the International Society of Lymphology. Our mission is to save precious time by offering the best and latest on rheumatology in an easy-to-read format, always hyperlinked for more in-depth reading later. What's inside?


Further doctor may order for blood test, urine test, X-ray and ECG to find out the root cause of swelling. Diuretics are prescribed in suitable doses to help kidney to expel excess of water from the body.
Degree of prognosis depends on the severity of the problem (kidney or liver failure) and also on the age and health condition of the patient. Now this is an essential diagnosis to consider in such patients who have unilateral or bilateral puffy hands. News summaries based on studies published in leading medical journals and specialty medical journals, conference coverage, case-based quizzes and more. When you press the swollen area of the skin by giving gentle pressure with your fingers, it creates identification on the site even after you have released the finger from the site. If required, liver function and kidney function test is also done to rule out problems in these vital organs.
In this article, we discuss the history taking and physical examination for patients who present with puffy hands.
We also publish case studies and practice-related articles by rheumatologists and other specialists. This can be illustrated by simple examples like having indication on the body after removing tight clothing, tight socks etc. Identification on the affected area will not be present in case of non-pitting edema like lymphedema or lipoedema.
The doctor will have to find out the exact cause of pitting edema to prevent leakage from tiny blood vessels.
Pitting edema is caused by various organ diseases or sometimes by local conditions that affect the hands and legs.
Significant IV drug use resulting in chronically puffy hands strongly supports a diagnosis of HCV infection. In addition to RA, gout, and pseudogout, the history may help rule out anasarca resulting from cardiac, renal, or liver disease.
A history of infection or trauma would suggest an infectious cause or fracture; a history of autoimmune disease (eg, scleroderma or Sjgren syndrome) often is associated with hand edema. PHYSICAL EXAMINATION The physical examination should start with evaluation of the patient’s general appearance; the presence of scleral icterus, jaundice, and anasarca should be noted.


On abdominal examination, tender hepatomegaly, splenomegaly, or lymphadenopathy may be elicited.
Patients may have purpura resulting from coagulopathy or encephalopathy resulting from chronic hepatic disease. Patients also should be examined for cardiac murmurs or scars suggesting previous cardiac surgery. Renal disease may be suggested as the cause of edema by oliguria, hypertension, foamy urine, or hematuria.
Patients’ hands may suggest an association with RA with the appearance of symmetrical inflammation of the metacarpophalangeal and proximal interphalangeal joints and finger deformities, such as ulnar deviation.
Evaluating whether upper extremity edema is unilateral or bilateral and whether the edema involves the whole arm or is isolated to the hand helps delineate the cause of the puffy hands.
Many of the possible diagnoses are the ones that are most often involved (eg, congestive heart failure, nephrotic syndrome, and liver disease).
Portal hypertension ensues with liver failure, often resulting in encephalopathy, hepatosplenomegaly, and formation of varices because of collateral blood flow.5 Another common clinical feature of HVC infection is bilateral puffy hands. When first described by Abeles6 in 1965, this variation of hand edema was attributed to poor venous return because of thrombosis of antecubital veins after years of injected drug use. Recurrent infections at the injection site also were thought to contribute.6 Schneiderman1 suggested that the cause is lymphedema associated with the sclerosing properties of the drugs. Neviaser and colleagues7 and Foldi8 suggested that lymphatic obstruction persists after injected drug use stops, along with scarring resulting from chronic inflammation. In addition, heroin often is diluted with quinine, which is notable for destroying lymphatics.7 The hand edema begins as pitting edema, followed by thickening and fibrosis of the subcutaneous skin (see Figure 1). Primary thrombosis, known as Paget-Schroetter syndrome, occurs in healthy, young adults with strenuous exercise. Fibrocytes are then activated and, with time, connective tissue overgrowth occurs as fibrosis ensues (Figure 3).15 Figure 3 – Right-arm lymphedema occurred in this patient after axillary lymph node dissection. Primary conditions are differentiated by age and are hereditary or spontaneous disorders of lymphatic architecture.15-18 Secondary causes are responsible for most lymphedema.



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