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30.06.2015
April 10, 2010In this article we will discuss a systematic approach to the differential diagnosis of bone tumors and tumor-like lesions. The differential diagnosis mostly depends on the review of the conventional radiographs and the age of the patient. It is important to realize that the plain radiograph is the most useful examination for differentiating these lesions. The most reliable indicator in determining whether these lesions are benign or malignant is the zone of transition between the lesion and the adjacent normal bone (1).
Once we have decided whether a bone lesion is sclerotic or osteolytic and whether it has a well-defined or ill-defined margins, the next question should be: how old is the patient? Finally other clues need to be considered, such as a lesion’s localization within the skeleton and within the bone, any periosteal reaction, cortical destruction, matrix calcifications, etc.
In the table on the left the morphology of a bone lesion is combined with the age of the patient. In order to classify osteolytic lesions as well-defined or ill-defined, we need to look at the zone of transition between the lesion and the adjacent normal bone.
The zone of transition is the most reliable indicator in determining whether an osteolytic lesion is benign or malignant (1). The zone of transition only applies to osteolytic lesions since sclerotic lesions usually have a narrow transition zone. A small zone of transition results in a sharp, well-defined border and is a sign of slow growth.
Metastases under the age of 40 are extremely rare, unless a patient is known to have a primary malignancy. Metastases could be included in the differential diagnosis if a younger patient is known to have a malignancy, such as neuroblastoma, rhabdomyosarcoma or retinoblastoma. There are two tumor-like lesions which may mimic a malignancy and have to be included in the differential diagnosis.
Infections and eosinophilic granuloma are exceptional because they are benign lesions which may seem malignant due to their aggressive biologic behavior.
These lesions may have ill-defined margins, but cortical destruction and an aggressive type of periosteal reaction may also be seen. Infections have to be included in the differential diagnosis of any bone lesion at any age.
In patients > 30 years we must always include metastases and myeloma in the differential diagnosis.
A periosteal reaction is a non-specific reaction and will occur whenever the periosteum is irritated by a malignant tumor, benign tumor, infection or trauma. An aggressive type is seen in malignant tumors, but also in benign lesions with aggressive behavior, such as infections and eosinophilic granuloma. Detecting a benign periosteal reaction may be very helpful, since malignant lesions never cause a benign periosteal reaction.
A benign type of periosteal reaction is a thick, wavy and uniform callus formation resulting from chronic irritation. In the case of benign, slowly growing lesions, the periosteum has time to lay down thick new bone and remodel it into a more normal-appearing cortex. This type of periostitis is multilayered, lamellated or demonstrates bone formation perpendicular to the cortical bone.
A Codman's triangle refers to an elevation of the periosteum away from the cortex, forming an angle where the elevated periosteum and bone come together.
Cortical destruction is a common finding, and not very useful in distinguishing between malignant and benign lesions. Complete destruction may be seen in high-grade malignant lesions, but also in locally aggressive benign lesions like EG and osteomyelitis. More uniform cortical bone destruction can be found in benign and low-grade malignant lesions. Endosteal scalloping of the cortical bone can be seen in benign lesions like FD and low-grade chondrosarcoma. The images on the left show irregular cortical destruction in an osteosarcoma (left) and cortical destruction with aggressive periosteal reaction in Ewing's sarcoma. In ballooning the destruction of endosteal cortical bone and the addition of new bone on the outside occur at the same rate, resulting in expansion. This 'neocortex' can be smooth and uninterrupted, but may also be focally interrupted in more aggressive lesions like GCT. A benign, well-defined, expansile lesion with regular destruction of cortical bone and a peripheral layer of new bone. A locally aggressive lesion with cortical destruction, expansion and a thin, interrupted peripheral layer of new bone.
Notice the wide zone of transition towards the marrow cavity, which is a sign of aggressive behavior.


In the group of malignant small round cell tumors which include Ewing's sarcoma, bone lymphoma and small cell osteosarcoma, the cortex may appear almost normal radiographically, while there is permeative growth throughout the Haversian channels.
These tumors may be accompanied by a large soft tissue mass while there is almost no visible bone destruction.
The image on the left shows an Ewing's sarcoma with permeative growth through the Haversian channels accompanied by a large soft tissue mass. The location of a bone lesion within the skeleton can be a clue in the differential diagnosis. In some locations, such as in the humerus or around the knee, almost all bone tumors may be found. Many lesions can be located in both or move from the metaphysis to the diaphysis during growth.
SBC, eosinophilic granuloma, fibrous dysplasia, ABC and enchondroma are lesions that are located centrally within long bones.
Osteosarcoma, NOF, chondroblastoma, chondromyxoid fibroma, GCT and osteoblastoma are located eccentrically in long bones. Osteoid osteoma is located within the cortex and needs to be differentiated from osteomyelitis. Calcifications or mineralization within a bone lesion may be an important clue in the differential diagnosis. There are two kinds of mineralization: a chondroid matrix in cartilaginous tumors like enchondromas and chondrosarcomsa and an osteoid matrix in osseus tumors like osteoid osteomas and osteosarcomas.
Calcifications in chondroid tumors have many descriptions: rings-and-arcs, popcorn, focal stippled or flocculent.
Mineralization in osteoid tumors can be described as a trabecular ossification pattern in benign bone-forming lesions and as a cloud-like or ill-defined amorphous pattern in osteosarcomas. NOF, fibrous dysplasia, multifocal osteomyelitis, enchondromas, osteochondoma, leukemia and metastatic Ewing' s sarcoma. This 'Mini Brain' appearance of plasmacytoma in the spine is sufficiently pathognomonic to obviate biopsy (9).
Tom Brokaw was diagnosed with multiple myeloma, a treatable but incurable blood cancer, in August 2013.
The following essay is by Tom Brokaw, as told to NBC News senior national producer Tim Uehlinger. So he did some blood tests and called me into a meeting with the head of internal medicine, who is also a hematologist, a blood specialist. Odd thing is, I guess I didn't know enough about it at that time, because my heart didn't accelerate.
Tom interviews Mary Shannon McGinnis and Katie Hambright from Memorial Sloan Kettering Cancer Center in New York. One of the things that I learned, and I had really good doctors, as individuals sometimes it wasn't as collective as I wanted it to be.
So one of the things that you have to learn, even if you don't have the kind of advantage that I do — with the high profile and the access — you have to learn to manage your case. For more information on multiple myeloma, including how you can help, visit The Multiple Myeloma Research Foundation.
This may include loosening tight joints in the neck, releasing tight soft tissues through massage or dry needling and prescribing appropriate exercises to address the cause of the problem. The International Business Machines Corporation - IBM, made known a lab-on-a-chip device technology that can separate biological particles at the nanoscale and could help physicians to detect diseases such as cancer before symptoms appear.
The results of the IBM team show size-based separation of bioparticles down to 20 nanometers in diameter, a scale that gives a means of approach to important particles such as Deoxyribonucleic acid (DNA), viruses and exosomes. Once these particles are separated, physicians can analyze it and can potentially reveal signs of disease, even before patients experience any physical symptoms and when the end result of treatment is most positive, according to PR News Wire.
IBM, which is an American multinational technology corporation, is collaborating with a team from the Icahn School of Medicine at Mount Sinai. However, the computer hardware company stated that it's pretty early in the research, where they need more time to figure out if the lab-on-a-chip device could feasibly and reliably be utilized to analyze fluids. If the result of the research will be positive, it could lead to more affordable, compact methods of identifying illnesses and they can possibly provide an in-home devices for self-monitoring, Engadget reported.
As IBM has been able to categorize particles down to 20 nanometers, IBM researcher Joshua Smith stated that they are talking about separating biological entities. Lab-on-a-chip technologies have become an awesome tool for physicians as the technology is significantly faster, portable and easy to use.
Aug 14, 2016 AM EDTDriven by his tough childhood, James Casap desires to make education easier for the next generation through technology. Aug 08, 2016 PM EDTDo you know that there is more to Rio Olympics 2016 awards than just a medal? Sorry Niantic, The Banned a€?Pokemon GOa€™ Cheaters can Still Cheat to Unban; Player Reveals How It Works!


Aug 15, 2016 AM EDTNiantic should have worked harder to remove all the unauthorized apps and bots to cheat "Pokemon GO". Aug 11, 2016 AM EDTNiantic Lab has been working relentlessly to solve servers issue while slashing all the third-party apps used for cheating "Pokemon GO". Aug 16, 2016 AM EDTAs the day is getting closer to badminton final matches, here's the update for Rio Olympics schedules.
Aug 12, 2016 PM EDTDay 6 is the starting day for all badminton athletes competing in Rio Olympics. Aug 16, 2016 AM EDTGoogle Nexus 2016 codenamed Sailfish may have been all over the web but the recent exclusive image leak might confirm all the rants. Department of Medical Oncology, Institut Paoli-Calmettes, 232 Boulevard de Sainte Marguerite, 13009 Marseille, France. There is periosteal bone formation perpendicular to the cortical bone and extensive bony matrix formation by the tumor itself.
Through months of specialized treatment, he is in remission and he is opening up about his battle with the disease.
Frank Reynolds, the ABC anchorman, who I had talked to toward the end of his life, not knowing what he had, died from it.
My family is not only attractive — I can say that because I'm paterfamilias — but they're really smart, and they're very, very compassionate.
When I got the Presidential Medal of Freedom, I wrote to everyone saying, "This is yours as much as it is mine. Their collaboration will continue to develop this lab-on-a-chip technology and they plan to test it for prostate cancer - which is the most common cancer in men in the United States. And to their knowledge, it's the largest particle that's been separated, according to Fast Company.
And the technology requires less sample volume to help detect diseases, as the goal is to decrease to a single silicon chip all of the processes necessary to examine methodically a disease. Use of this Web site constitutes acceptance of our Terms and Conditions of Use and Privacy Policy. If the proposed first-line treatment is contraindicated owing to a patient's clinical status, the treatment approach recommended for the subsequent disease stage should be considered. He obtained the Board certification in Gastroenterology and Hepatology in the Liver and Gastroenterology Unit, Hospital Clinic, Barcelona (2001–2005) and since 2005, has worked at the Barcelona Clinic Liver Cancer (BCLC) group directed by Prof.
And so I couldn't imagine that I was going to go through the ordeal that it turned out to be. But everybody has to be constantly conscious of the fact, it's your body, it's your health, it's the cancer that's affecting you. His clinical activity is based on the care of cirrhotic patients with hepatocellular carcinoma (HCC) and his investigational fields are the early diagnosis (doctoral thesis) and the treatment of advanced-stage HCC. In addition, he is Associate Editor of Liver International, and has acted as reviewer for the most-cited peer-reviewed journals in hepatology (Hepatology, Gut, Journal of Hepatology and Liver Transplantation) and oncology (Lancet Oncology).
She graduated in Oncology in 2011 from Mediterranee University, Aix-Marseille, France, following medical studies at Aix-Marseille University, France. She is a member of the Clinical Research Team, Centre de Recherche en Cancerologie de Marseille, INSERM UMR 1068, Paoli-Calmettes Institute, Aix-Marseille University, France, where she completed her PhD in 2014. He has been Principal Investigator of studies and clinical trials that have changed practice in the field of HCC, including the development of diagnostic criteria and prognostic models, and establishing chemoembolization and sorafenib as standard therapies. He has developed and sequentially updated the BCLC staging and treatment strategy that has been endorsed by several international scientific associations to guide the management of patients with HCC. Dr Bruix is a member of the European Association for the Study of the Liver (EASL) and the American Association for the Study of Liver Diseases (AASLD). He founded the International Liver Cancer Association (ILCA) and was nominated President from 2006 to 2009.
He has been Associate Editor of Journal of Hepatology, Liver Transplantation, and Hepatology, and is currently Associate Editor of Seminars in Liver Disease. He is currently Head of the Gastrointestinal Oncology Team at the Comprehensive Cancer Centre, Paoli-Calmettes Institute. He has held the position of Professor of Medical Oncology at Rennes University since 2000, and in 2005 he was appointed Visiting Professor at the University of Perth-Fremantle, Western Australia.
Jean-Luc Raoul has clinical and research interests in gastrointestinal cancers mainly oesophageal and pancreatic cancers and HCC, in particular the application of targeted internal radiation therapy and targeted therapies in HCC. He is a member of several societies including the American Society of Clinical Oncology (ASCO), the ILCA, the EASL, the ESMO (European Society of Medical Oncology.



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