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A weblog on the sciences and practices of living healthily very long – perhaps hundreds of years.
There is strong and widespread current interest in gut microbiota and I have found thousands of relevant research publications related to gut microbiota.  My purpose here it to introduce the topic in this blog, to establish its relevance, and to identify some recent key research findings. Aging of a human happens in a meta-organism consisting not only of that person’s body and cells but also of the 100 trillion organisms that exist in that persons gut. The 2009 publication Acquisition, evolution and maintenance of the normal gut microbiota reports: “The gut is sterile at birth, but is rapidly colonised by faecal and vaginal bacteria of maternal origin. From Genomic Insights into Bifidobacteria: “The human large intestine is a very complex ecosystem that is still not fully understood, and while its microbial composition consists primarily of just four bacterial phyla, Bacteroidetes, Firmicutes, Actinobacteria, and Proteobacteria (as well as some Archaea, Eukarya, and viruses), it is highly variable at the genus level between individuals, likely due to factors such as age, health, diet, etc. The 2007 publication Intestinal bacteria and ageing reported: “Advancements in science and medicine, as well as improved living standards, have led to a steady increase in life expectancy, and subsequently a rise in the elderly population.
The 2010 publication Role of the gut microbiota in defining human health reported: “The human superorganism is a conglomerate of mammalian and microbial cells, with the latter estimated to outnumber the former by ten to one and the microbial genetic repertoire (microbiome) to be approximately 100-times greater than that of the human host.
While the study of gut bacteria has been going on for more than 100 years, many new insights are now flowing from massive genomic sequencing techniques. Bifidobacteria appear to be an important set of species of gut bacteria from a health viewpoint. Gut microflora may play an even more important role in maintaining human health than previously thought.  Specifically it appears they are involved in the control of energy and metabolic homeostasis.
This theme has been repeated in a substantial number of publications for over five years now. The August 2011 publication Programming of host metabolism by the gut microbiota reports: “The human gut harbors a vast ensemble of bacteria that has co-evolved with the human host and performs several important functions that affect our physiology and metabolism. The mixture of kinds of bacteria in the gut may contribute to metabolic disorders such as obesity, diabetes, and cardiovascular diseases or can contribute strongly to maintenance of health homeostasis.
The November 2010 publication Ecology and Physiology of the Intestinal Tract reports: “The number of microorganisms inhabiting the human digestive tract exceeds the number of body cells by a factor of ten. The January 2012 publication Is the gut microbiota a new factor contributing to obesity and its metabolic disorders? A discussion of the role of gut flora in autism and inflammatory bowel disease can be found on this webpage of science-autism.org which is about published scientific data concerning autism. Image from Gut Flora in Autism:  “A well considered pathway for the effect of bacterial flora on the inflammatory response of the gut wall in IBD. With advanced aging, changes typically happen in the ecology of the gut microorganism, system resulting in increased susceptibility to infectious diseases and related infirmities.
On the other hand, the 2010 e-publication Human intestinal microbiota and healthy ageing review publication reports: “Earlier studies have indicated a decrease in anaerobes and bifidobacteria and a concomitant increase in enterobacteria in the intestinal microbiota with ageing. TLR and NLR signaling in intestinal epithelial cells triggered by gut microbiota contribute to a range of homeostatic mechanisms including proliferation, wound healing, epithelial integrity, and regulation of mucosal immune functions. The 2010 publication The role of innate signaling in the homeostasis of tolerance and immunity in the intestine reports: “In the intestine innate recognition of microbes is achieved through pattern recognition receptor (PRR) families expressed in immune cells and different cell lineages of the intestinal epithelium.
Our old friend NF-kappaB can be activated by TLR and NOD signaling by gut microbiota, resulting in possible positive or detrimental effects. The 2010 publication Role of NF-kappaB activation in intestinal immune homeostasis reports: “Inflammatory bowel diseases (IBD) are characterised by a disturbance of intestinal immune homeostasis, either caused by or followed by inappropriate responses to the resident commensal bacteria.
The May 2011 publication Influence of gastrointestinal commensal bacteria on the immune responses that mediate allergy and asthma reports: “The human intestine contains more than 100 trillion microorganisms that maintain a symbiotic relationship with the host. Raising bifidobacteria levels by the use of probiotics and prebiotics appears to be the most-established strategy for modifying microbial balance in human colons to promote health.  However, much remains to be learned. The December 2009 publication Food-based strategies to modulate the composition of the intestinal microbiota and their associated health effects reports: “The most well known food-based strategies to modulate the composition of the intestinal microbiota are the dietary use of prebiotics, probiotics and their combination, synbiotics.
Prebiotics may be useful for the prevention or treatment of insulin resistance, diabetes and obesity.
The 2011 publication Gut microbiota and the pathogenesis of insulin resistance reports: “Several reviews recently explored how the gut microbiota was able to control host energy metabolism, and thereby the development of adiposity. The LKM512 strain of yogurt bacteria has attracted particular attention as a probiotic for increasing polyamines levels and inhibiting inflammation. The 2009 publication Polyamine-rich food decreases age-associated pathology and mortality in aged micerelates polyamine consumption to senescence markers in mice: “The purpose of this study was to test whether oral intake of foods rich in polyamines (spermine and spermidine) suppresses age-associated pathology in aged mice.
The 2009 publication Dynamics of fecal microbiota in hospitalized elderly fed probiotic LKM512 yogurt reports: “The comprehensive dynamics of intestinal microbiota including uncultured bacteria in response to probiotic consumption have not been well studied. Some researchers think targeting the gut microbiota may be an avenue for human life extension. While this discussion is theoretical, it does appear that life extension in mice is possible by feeding them probiotics.
I was motivated to research and generate this blog entry by coming across the August 2011 publication Longevity in mice is promoted by probiotic-induced suppression of colonic senescence dependent on upregulation of gut bacterial polyamine production. In your digestive system there is a community of flora, or naturally-occurring bacteria, that it's important to keep in balance.
An intestinal mucus layer lines the digestive tract and acts as a barrier and protection to keep bad bacteria out of your body. Probiotics help stimulate the production of the protective mucus layer and can also line the mucus layer to add an extra level of defense. Immune cells in your intestinal immune system are constantly on guard, testing all the things that enter your body to decide whether they are good (let them in) or bad (keep them out). The ubiquitous use of prescribed antibiotics, over the counter drugs and our feedlot animals impair beneficial bacteria necessary for a healthy digestive system. Beneficial bacteria, known as friendly bacteria or probiotics (for life), lives in the gut in massive amounts. The Lactobacillus family contains many types of friendly bacteria that help the body’s resiliency to illness and disease.
Bifidobacterium is found mainly in the large intestine and also in the lower part of the small intestine. Due to our lifestyles, the body constantly fights the invasion of bad bacteria and toxins that lead to chronic illness and disease. A study was conducted in Sweden in a company examining the effect of probiotics on their ability to improve work-place health by reducing short term sick-leave caused by respiratory or gastrointestinal infections.
Everyone is exposed to toxins and bacteria on a daily basis so it is not only prudent, but necessary to take probiotics for preventive measures.
Contrary to popular belief, consuming frozen yogurt, fruity dairy yogurts, yogurt covered raisins or nuts, does not provide any protection.
You may wish to consult an integrative health professional familiar with the benefits of friendly bacteria.
As always, a wholistic lifestyle that includes a good multi-vitamin, healthy eating, exercise, stress management, rest and moderation of behavior (temperance) are important. Morgellons Disease Awareness -  Artist and Morgellons sufferer, Ayla, journals her experience with the disease   I'd like to introduce myself, I'm Ayla, a 59year old artist and graphic designer who contracted Morgellons in Jan.
In your body good and bad bacteria compete for the same nutrients throughout the intestine, we want to have more of the good and less of the bad.
In addition to intestinal well-being, probiotics also help to promote healthy immune function.* The “gut” plays an important and complex part in our overall immune health. Not all bacteria are bad, but with the introduction of “antibacterial products,” we have been on a mission to rid our environment from disease and all bacteria. Long recognized as a leading manufacturer of the highest quality probioitc supplements, American Health continues to offer a full line probiotic products in a variety of flavors, delivery forms and potencies. This assumption imposes a more holistic view of the ageing process where dynamics of the interaction between environment, intestinal microbiota and host must be taken into consideration. Over the succeeding weeks, months and years, a complex microbiota develops that plays a major role in host physiology. The intestinal microbiota is important for maintenance of host health, providing energy, nutrients and protection against invading organisms.
Given the ability of the immune response to rapidly counter infectious agents, it is striking that such a large density of microbes can exist in a state of synergy within the human host. Identification of Bifidobacteria goes back to 1900.  Also from the September 2010 publication Genomic Insights into Bifidobacteria “Tissier (315) suggested that the large number of bifidobacteria in the feces of healthy breast-fed infants was likely the reason for their lower incidence of infantile diarrhea. The 2007 reviewpublication Gut microflora as a target for energy and metabolic homeostasis reported “Purpose of review: Gut microbiota plays an important role in health and disease, but this ecosystem remains incompletely characterized and shows a wide diversity. The human gut is sterile at birth and is subsequently colonized with bacteria from the mother and the environment. Relates: “The gut microbiota refers to the trillions of microorganisms residing in the intestine and is integral in multiple physiological processes of the host.
We observed that the microbial composition and diversity of the gut ecosystem of young adults and seventy-years old people is highly similar but differs significantly from that of the centenarians. However, new data obtained with molecular techniques suggests decreased stability and increased diversity of the gut microbiota with advancing age. Toll-like receptor (TLR) and nucleotide-binding and oligomerization domain-like receptor (NLR) families are emerging as key mediators of immunity through their role as maturation factors of immune cells and triggers for the production of cytokines and chemokines and antimicrobial factors. Under normal conditions, these bacteria are not pathogenic and in fact confer health benefits to the host. By far the most important predominant populations are in the colon where a true symbiosis with the host exists that is a key for well-being and health. Currently established prebiotic compounds are mainly targeting the bifidobacteria population of the colon microbiota.
In this review, we focused on the state of the art that supports a link between the gut microbiota composition and activity, and the management of glycemia associated with overweight and diabetes.
Synthetic polyamines were mixed into experimental chows, and 24-week-old Jc1:ICR male mice were fed one of three chows containing differing polyamine concentrations. The aims of this study were twofold: firstly to analyze the impact on intestinal microbiota of yogurt fermented by Bifidobacterium animalis subsp. During evolution, an integrative circuitry fundamental for survival has been established between commensal gut microbiota and host.
There are three different ways that the digestive system and probiotics help build your immunity. They also help out-compete the bad bacteria that may cause harm, and they secrete chemicals that kill off some bad bacteria. Probiotics help stimulate and exercise the immune cells by going through a sampling and screening process that acts just like a fire drill. The gut is responsible for digesting foods aided by several organs, specific enzymes, good bacteria and nutrients. Lactobacillus acidophilus is present mainly in the small intestine and works at lowering cholesterol levels, LDL levels and killing candida yeasts. These friendly bacteria prevent toxicity that comes from nitrites in food, helps detoxify bile and assists in the management of some liver conditions and illnesses.
Because friendly bacteria have such powerful ability to rid the body of toxicity and unfriendly organisms, it is necessary to consume them on a regular basis. Recent research suggests a significant percentage of the body’s immune system is actually located in the digestive tract. In combination with food safety regulations, increased additives in our foods and heat processes used to extend shelf life, neither the bad nor good bacteria are surviving the manufacturing process. While the digestive tract is colonised to varying degrees by micro-organisms throughout its length, due to acid pH and the short retention time of gastric contents, bacterial numbers in the stomach are usually low.
Although the colonic microbiota is relatively stable throughout adult life, age-related changes in the gastrointestinal (GI) tract, as well as changes in diet and host immune system reactivity, inevitably affect population composition. This is particularly true of the distal gastrointestinal (GI) tract, which houses up to 1000 distinct bacterial species and an estimated excess of 1 x 10(14) microorganisms. In his pediatric work, he used bifidobacteria for the treatment of this intestinal diarrhea, and this likely represents the first example of the oral administration of a live microorganism for the treatment of a disease (316).
The complexity of the gut microbiota is increased during childhood, and adult humans contain 150-fold more bacterial genes than human genes. The metabolic potential of the gut microbiota is immense affording the extraction of energy from otherwise indigestible carbohydrates (dietary fiber) and the conversion of host-derived substances, non-nutritive dietary components and drugs. Recent research has shown that gut bacteria play a role in metabolic disorders such as obesity, diabetes, and cardiovascular diseases.
After 100 years of symbiotic association with the human host, the microbiota is characterized by a rearrangement in the Firmicutes population and an enrichment in facultative anaerobes, notably pathobionts.
At the mucosal surface chronic activation of the immune system is avoided through the epithelial production of a glycocalyx, steady-state production of antimicrobial factors as well as the selective expression and localization of PRRs. The enormous amount of commensal bacteria in the intestine might play a role in the distinct functions of NF-kappaB in the intestine, as they can initiate signalling to NF-kappaB through both Toll-like receptors and NOD-like receptors in intestinal epithelial cells as well as mucosal immune cells.
The microbiota interacts with the innate and adaptive arms of the host’s intestinal mucosal immune system and through these mechanisms drives regulatory cell differentiation in the gut that is critically involved in maintaining immune tolerance. Several microbial-derived compounds are related to disturbances of glucose homeostasis including the gram-negative-derived lipopolysaccharides. On the basis of recent knowledge in worms, flies, and humans, an important role of the gut microbiota in aging and longevity is emerging. In mammals, levels of polyamines (PAs) decrease during the ageing process; PAs are known to decrease systemic inflammation by inhibiting inflammatory cytokine synthesis in macrophages. So, probiotics help prepare your immune cells to fight off any bad bacteria that come their way! The digestive process feeds the cells and provides energy for life, however the average American diet assaults the digestive processes with toxins that come from our foods, water and air. Ten out of 94 workers using the probiotic took sick time during the study compared to 23 in the placebo group. I've been moved to create awareness tools that help educate healthcare practitioners and the public about Morgellons Disease (see Awareness Poster) as well as share what I've learned from this experience.Many of the skin photos in the photo galleries are mine. This part of the immune system is able to recognize and reject microorganisms that may challenge the body’s defense system.
Add to that an over reliance on antibiotics and it’s no wonder our good bacteria are being depleted. Here we review the current knowledge of the changes occurring in the gut microbiota of old people, especially in the light of the most recent applications of the modern molecular characterisation techniques. The rapid passage of digestive materials through the upper gut does not provide time for significant bacterial growth to occur, but cell numbers increase considerably in the distal ileum.
Recent studies indicate shifts in the composition of the intestinal microbiota, which may lead to detrimental effects for the elderly host. An ever-increasing body of evidence implicates the GI microbiota in defining states of health and disease. The abundance of bifidobacteria in the feces of breast-fed infants was thought to be due to the Bifidobacterium-stimulating properties of human breast milk (38, 46, 60, 96, 196, 321).
Recent advances in next-generation sequencing technology and mechanistic testing in gnotobiotic mice have identified the gut microbiota as an environmental factor that contributes to obesity.
Recognized functions of the gut microbiota include provision of colonization resistance against pathogens and priming of both the innate and the acquired immune systems.
The presence of such a compromised microbiota in the centenarians is associated with an increased inflammatory status, also known as inflammageing, as determined by a range of peripheral blood inflammatory markers. Except for the reduced immune function, ageing itself may have relatively little effect on overall gastrointestinal function.
Additionally, the polarization of epithelial TLR signaling and suppression of NF-kappaB activation by luminal commensals appears to contribute to the homeostasis of tolerance and immunity. However, the exact individual contributions of different NF-kappaB-activating stimuli as well as the target cells that mediate the detrimental or beneficial functions of NF-kappaB in the intestine are still elusive. Specifically, the microbiota can activate distinct tolerogenic dendritic cells in the gut and through this interaction can drive regulatory T-cell differentiation. The present document has been written by a group of both academic and industry experts (in the ILSI Europe Prebiotic Expert Group and Prebiotic Task Force, respectively).
Some nutrients with prebiotic properties, which escape the digestion in the upper part of the gut, modify the composition of the gut microbiota in favor of bacteria that could play a beneficial role on glucose homeostasis, namely by modulating the endocrine function of the gut, and by reinforcing the gut barrier. An increase in concentration of polyamine in the blood was found only in mice fed the high polyamine chow at 50 weeks of age.


The complex bacterial community that populates the gut and that represents an evolutionary adapted ecosystem correlated with nutrition appears to limit the accumulation of pathobionts and infections in all taxa, being able of affecting the efficiency of the host immune system and exerting systemic metabolic effects.
Reductions in intestinal luminal PAs levels have been associated with intestinal barrier dysfunction. Furthermore, none of the 26 workers using the probiotics lost any time due to illness compared to nine out of 27 in the placebo group. The hypothetical involvement of the age-related gut microbiota unbalances in the inflamm-aging, and immunosenescence processes will also be discussed. The rate of movement of intestinal contents slows in the colon, which facilitates the development of complex bacterial communities. Increased numbers of facultative anaerobes, in conjunction with a decrease in beneficial organisms such as the anaerobic lactobacilli and bifidobacteria, amongst other anaerobes, have been reported.
Recent data suggest that the modulation of gut microbiota affects host metabolism and has an impact on energy storage.
Germ-free mice are protected against developing diet-induced obesity and the underlying mechanisms whereby the gut microbiota contributes to host metabolism are beginning to be clarified.
However, the intestinal microbiota may also contribute to the development of diseases such as ulcerative colitis and colorectal cancer. Gut microbiota has evolved with humans as a mutualistic partner, but dysbiosis in a form of altered gut metagenome and collected microbial activities, in combination with classic genetic and environmental factors, may promote the development of metabolic disorders.
Concomitant changes in nutrition, increased incidence of disease and corresponding use of medication with advancing age modify the composition of the microbial community of the gastrointestinal tract. Several studies have demonstrated that TLR signaling in epithelial cells contributes to a range of homeostatic mechanisms including proliferation, wound healing, epithelial integrity, and regulation of mucosal immune functions. In addition, the microbiota is important in driving T(H)1 cell differentiation, which corrects the T(H)2 immune skewing that is thought to occur at birth.
Interestingly, the changes in the gut microbes can be reversed by dieting and related weight loss. While the body weights of mice in all three groups were similar, the survival rate of mice fed high polyamine chow was significantly higher than those in the other two groups (p=0.011). There is an urgent need to disentangle the underpinning molecular mechanisms, which could shed light on the basic mechanisms of aging in an ecological perspective. The large intestine is an intricate ecosystem that contains a complex microbiota composed of several hundred different types of bacteria. These changes, along with a general reduction in species diversity in most bacterial groups, and changes to diet and digestive physiology such as intestinal transit time, may result in increased putrefaction in the colon and a greater susceptibility to disease. The obese phenotype is associated with increased microbial fermentation and energy extraction; however, other microbially modulated mechanisms contribute to disease progression as well. Culture-dependent studies provided basic knowledge on the gut microbiota, but only the advent of culture-independent molecular methods led to a better understanding of host-microbe interactions.
If appropriate immune tolerance is not established in early life and maintained throughout life, this represents a risk factor for the development of inflammatory, autoimmune, and allergic diseases. A large number of human intervention studies have been performed that have demonstrated that dietary consumption of certain food products can result in statistically significant changes in the composition of the gut microbiota in line with the prebiotic concept. The intestinal microbiota can be considered as a metabolically adaptable and rapidly renewable organ of the body. The qualitative and quantitative changes in the intake of specific food components (fatty acids, carbohydrates, micronutrients, prebiotics, probiotics), have not only consequences on the gut microbiota composition, but may modulate the expression of genes in host tissues such as the liver, adipose tissue, intestine, muscle.
Mice fed the high polyamine chow analyzed at 88 weeks of age, corresponding to the end of the study, demonstrated lower incidence of glomerulosclerosis and increased expression of senescence marker protein-30 in both kidney and liver compared to those fed the low polyamine chow.
Like most, I panicked bought a lot of unnecessary products—some toxic, then managed to pull myself back from the brink of actually hurting myself. The growth and metabolism of microbial communities in the large intestine are determined by many factors, such as diet, environment and host physiological processes, as well as the anatomic structure of the digestive tract, disease, immunity, host genetics, drugs and ageing. An understanding of their full interactions with intestinal microbes and the host is needed to scientifically validate any health benefits they may afford. The literature provides new evidence that the increased prevalence of obesity and type 2 diabetes cannot be attributed solely to changes in the human genome, nutritional habits, or reduction of physical activity in our daily lives. The gut microbiota has profound effects on host gene expression in the enterohepatic system, including genes involved in immunity and metabolism. The application of metagenomics to the gut microbial ecosystem revealed truly remarkable correlations between certain diseases and the gut microbiome.
Early-life events are instrumental in establishing the microbiota, the composition of which throughout life is influenced by various environmental and lifestyle pressures. Among the numerous purported health benefits attributed to probiotic bacteria, the (transient) modulation of the intestinal microflora of the host and the capacity to interact with the immune system directly or mediated by the autochthonous microflora, are basic mechanisms. However, unbalances in its microbial community and activities are found to be implicated in disease initiation and progression, such as chronic inflammatory bowel diseases and colonic cancers. This in turn may drive or lessen the development of fat mass and metabolic disturbances associated with the gut barrier function and the systemic immunity. Modifications in diet and host immune system activity, as well as physiological changes in the digestive tract affect microbiota composition in older people. These include dietary supplements containing prebiotics, probiotics and a combination of both of these, synbiotics. Recently, the genome sequences of nine strains representing four species of Bifidobacterium became available. For example, the gut microbiota affects expression of secreted proteins in the gut, which modulate lipid metabolism in peripheral organs. They are supported by an increasing number of in vitro and in vivo experiments using conventional and molecular biologic methods.
The more data are accumulating, the more it will be recognised that such changes in the microbiota’s composition, especially increase in bifidobacteria, can be regarded as a marker of intestinal health. Restoration of this balance by increasing bifidobacteria levels has demonstrated to reduce disease severity of patients and to improve well-being in healtly volunteers. The relevance of the prebiotic or probiotic approaches in the management of obesity in humans is supported by few intervention studies in humans up to now, but the experimental data obtained with those compounds help to elucidate novel potential molecular targets relating diet with gut microbes.
The T-RFLP patterns in five of the six volunteers were changed in a similar fashion by LKM512 yogurt consumption, although these patterns were individually changed following consumption of placebo.
Always wary of doctors, I tried to stay out of that loop but finally caved when my beloved ones pressured me to seek help.
The elderly have fewer bifidobacteria and higher numbers of enterobacteria and clostridia than young adults.
A comparative genome analysis of these genomes reveals a likely efficient capacity to adapt to their habitats, with B. Scientists may take into consideration a key question: could we manipulate the microbiotic environment to treat or prevent obesity and type 2 diabetes? In addition, the gut microbiota is also a source of proinflammatory molecules that augment adipose inflammation and macrophage recruitment by signaling through the innate immune system. In addition to these, a limited number of randomized, well-controlled human intervention trials have been reported.
The review is divided in chapters that cover the major areas of nutrition research where a prebiotic effect has tentatively been investigated for potential health benefits. New emerging evidence on the difference in the composition of the colonic microbiota between obese and lean volunteers has opened new areas for pre-, pro- and synbiotic research. A dermatologist gave me Ivermectin which caused a Herxheimer (die-off) reaction and many of the black particles I'd been seeing with a scope surfaced. TLRs (Toll-like receptors) are integral parts of the innate immune system and are expressed by both macrophages and epithelial cells.
The prebiotic effect has been shown to associate with modulation of biomarkers and activity(ies) of the immune system. Nutrients with prebiotic properties allows, by changing the gut microbiota, to promote the endocrine function of the gut (increase in GLP-1, and GLP-2 producing cells), and to modulate the activation of the endocannabinoid system in the intestine and in the adipose tissue.
We then analyzed inflammatory and intestinal conditions by measuring several markers using HPLC, ELISA, reverse transcription-quantitative PCR, and histological slices. This gave me great relief from the discomfort of what felt like fiberglass embedded in my skin.
Activation of TLRs in macrophages dramatically impairs glucose homeostasis, whereas TLRs in the gut may alter the gut microbial composition that may have profound effects on host metabolism.
All those effects contribute to lessen gut permeability (improved distribution of the tight junction proteins ZO-1 and Occludin), thereby decreasing endotoxemia, and systemic inflammation. LKM512 mice showed altered 16S rRNA gene expression of several predominant intestinal bacterial groups. This concomitantly improves stool quality (pH, SCFA, frequency and consistency), reduces the risk of gastroenteritis and infections, improves general well-being and reduces the incidence of allergic symptoms such as atopic eczema. Changes in GLP-1 contribute to decrease food intake, fat mass, glycemia and insulin resistance. There were NO fibers visible in my skin in the previous months before taking the medication. Changes in the gut microbiota composition are classically considered as one of the many factors involved in the pathogenesis of either inflammatory bowel disease or irritable bowel syndrome. Colonic mucosal function was also better in LKM512 mice, with increased mucus secretion and better maintenance of tight junctions. The similar changes in the intestinal microbiota of the elderly caused by consumption of the LKM512 yogurt were found to be influenced by the LKM512 strain itself, and not by the lactic acid bacteria in the yogurt.
An intriguing observation is the loss of much of this genome potential when strains are adapted to pure culture environments, as highlighted by the genomes of B.
Prevention and alleviation of unspecific and irregular complaints of the gastrointestinal tracts in healthy people. Promising beneficial effects have been demonstrated in some preliminary studies, including changes in gut microbiota composition (especially increase in bifidobacteria concentration). Within two days of taking the Ivermectin, I watched in horror as fibers started to rise to the surface of my skin.
This indicates that ingestion of specific probiotics may be an easy approach for improving intestinal health and increasing lifespan. Beneficial effects on microbial aberrancies, inflammation and other complaints in connection with: inflammatory diseases of the gastrointestinal tract, Helicobacter pylori infection or bacterial overgrowth. That plus coming off the tail-end of the pharmaceutical seemed to stop all the symptoms of itching, biting and crawling for approximately the next 4 months.
Some of these studies (including one human trial) have also reported that, in such conditions, gut microbiota composition was modified (especially due to increased concentration of bifidobacteria). The only thing that hadn't cleared up was the hyper-sensitivity to my skin which I thought I was stuck with for the rest of my life, an unfortunate consequence of having Morgellons. Had I not been actively using a microscope to look at my skin during this period, I might have presumed I was well. Normalization of passing stool and stool consistency in subjects suffering from obstipation or an irritable colon. Dietary intake of particular food products with a prebiotic effect has been shown, especially in adolescents, but also tentatively in postmenopausal women, to increase Ca absorption as well as bone Ca accretion and bone mineral density.
The skin however, doesn't lie and told a whole other story for under it's surface, fibers and particles were clearly still present—multitudes of them. Early on, I'd heard about the testing that Pam Crane of Morgellons Focus On Health (site no longer active) was championing and felt that it was probably the way to go. Recent data, both from experimental models and from human studies, support the beneficial effects of particular food products with prebiotic properties on energy homaeostasis, satiety regulation and body weight gain. Pathways that were downregulated by ageing were upregulated by LKM512 administration and vice versa. My only problem with moving forward was a lack of practitioner, as I felt having the best testing in the world wouldn't mean anything unless someone was up to the task of decoding it's contents into a meaningful protocol, sort of like buying a Cadillac that arrives without the key. I wanted to have a conversation with whatever doctor I was going to see and to hear them speak about their philosophy of healthcare. Together, with data in obese animals and patients, these studies support the hypothesis that gut microbiota composition (especially the number of bifidobacteria) may contribute to modulate metabolic processes associated with syndrome X, especially obesity and diabetes type 2.
In other words, LKM512 administration suppressed ageing-associated change in gene pathways.
It is plausible, even though not exclusive, that these effects are linked to the microbiota-induced changes and it is feasible to conclude that their mechanisms fit into the prebiotic effect.
Just try to get a physician on the phone before actually making an appointment and you will find it is an impossible task (especially the high-end specialists). Prevention of respiratory tract infections (common cold, influenza) and other infectious diseases as well as treatment of urogenital infections. However, the role of such changes in these health benefits remains to be definitively proven.
Insufficient or at most preliminary evidence exists with respect to cancer prevention, a so-called hypocholesterolemic effect, improvement of the mouth flora and caries prevention or prevention or therapy of ischemic heart diseases or amelioration of autoimmune diseases (e.g. She talked about The Great Plains Labs and why the testing used in Functional Medicine is so important for our community. She sounded like she knew how to do her job well and was more than capable, indeed she had impressed me as understanding how to approach the disease in a holistic manner that aligned with my own healthcare philosophies. In other words, LKM512 administration suppressed ageing-associated change in gene pathways.” – “The anti-inflammatory effects of LKM512 administration were also revealed by a DNA microarray. I believe my first words to her were, "Where do I sign the dotted line?" (While focusonhealth has been taken down, here is another radio show with Nancy speaking with Pam. Today, only bifidogenic, non-digestible oligosaccharides (particularly inulin, its hydrolysis product oligofructose, and (trans)galactooligosaccharides), fulfill all the criteria for prebiotic classification. Expression levels of genes in the TNF-NF?B, IL-1, IL-2, and IL-6 pathways were higher in the control group than in LKM512 and younger mice; additionally, gene expression levels in LKM512 mice were similar to those in younger mice (Fig. They are dietary fibers with a well-established positive impact on the intestinal microflora.
Other health effects of prebiotics (prevention of diarrhoea or obstipation, modulation of the metabolism of the intestinal flora, cancer prevention, positive effects on lipid metabolism, stimulation of mineral adsorption and immunomodulatory properties) are indirect, i.e. Let me remind you that I felt fine when I first contacted Nancy, but the emerging fibers showed me I was not. Nancy forewarned me that treatment would kick up all the initial symptoms I had when first ill with Morgellons, such as itching and stinging and that I might experience more fatigue with die-off, possibly even get lesions although I hadn't had many lesions previously. Within a week and a half of beginning this new protocol all the discomforts she mentioned came back.
Black particles began coming out from the skin, sand-like crystals were suddenly pushing from the scalp, I was itchy and uncomfortable but this time around it was at a much lower volume then at onset.
I have experienced some lesions, in the form of 'papercuts' and blood spots appearing on the skin. They were tiny and healed quickly. It took close to 3 months for the symptoms of this detox to disappear, lessening with each week. We added in another few products to my protocol fine-tuning the process. Once all my symptoms had abated I was ready to move on to the next step and do a colon cleanse. I used supplements for one month that gently cleansed the colon (Colon Cleanse by DaVinci Labs??). After that, I began with chelation therapy (Chelex by Xymogen) to remove heavy metals. Nancy has taken me through this journey with constant support through email. The fact that she is able to tell me what I can expect and interpret what I am going through is invaluable.
Having gone through the early treatment phases and emerging symptom-free, makes me feel that I did indeed work through something—hopefully clearing biofilm. So where am I right now and what are the changes that happened over these few months of treatment?My skin cleared of many of black particles, although I still have them (my great lament was that I didn't have a camera back when I first got sick, because my skin was so saturated it was alarming!). Actually, I feel spry and 10 years younger than my 55 years of age (at the time this was written), an unexpected and very welcome gift.
Still, they are here and I am monitoring them carefully. I am convinced of the value of the Great Plains and Metametrix testing. It just makes sense that one would want to know the particular weaknesses of their body before embarking on a journey of healing. Getting the labwork done and seeing Nancy are the two smartest things I did throughout my ordeal. Morgellons for me is like that impossible teacher I had in my youth.
But it has also left me savoring hard-won battles and set me straight on how to live more in balance.
Therefore, I try not to rail against the unfairness of the experience, but to look at how much I learned and the ways in which it has stretched me as a human being.
Yes it has been a miserable teacher, but by it's hand I have been become an attentive student.I wish you all full recovery.
AylaWhile I am not comfortable sharing my protocol as it is specific to my lab results, I will outline what I feel are the bare-bones minimum I'd look to take if I were building a protocol.


This would be a good probiotic (I use 3 different kinds throughout the day as was indicated by the results of my testing). FOS, according to Nancy can promote yeast growth so I'd stay away from a probiotic that includes it.
I'd include a biofilm buster which breaks down the cellular matrix in the gut that inhibits absorption of necessary nutrients. I used Interfase Plus and Wobenzym at different times (there are different kinds of Wobenzyme. I've used Carlson's liquid D3 which is lipid based, but recently have switched because of assimilation issues with fats to a dry encapsulated D3 (Prothera brand 5,000 I.Us a day).
Certain of the B's can become depleted when the body is fighting infection, so a good B-complex seems necessary. Also, Mr. Common Sense and now Clifford Carnicom have written about NAC and it's benefits along with vitamin C in helping quell the symptoms of our condition. I'd look into this as a close friend from the community began taking both high amounts of Vitamin C and NAC and her symptoms disappeared. She was taking 3,000-5,000 mg of C spread throughout the day and 600 mg 2x's a day of NAC both in the morning and at night. She eventually reduced it to 2,000 mg of vitamin C and 600 mg of NAC because the NAC was giving her headaches. The lesser dosage has been fine for keeping her symptoms of crawling, biting and stinging away.
When she went off the protocol symptoms came back and when she again resumed, they went away.
I might mention, that my protocol has always called for 1000 mg of vitamin C, three times a day.
While I was not on NAC as a separate supplement, it is in another product I take daily ?(Liver Protect by Xymogen). Nancy has stated that the clients she sees that do this first make themselves much worse and take longer to get better. If I am going to do this right, I want to know what my baseline toxic load is so I can measure my progress over time.
The results arrived right after I finished my third week of chelation. My total toxic representation was OFF the chart with high levels of antimony, silver, cadmium, tin and magnesium. I have a host of other metals as well, they just were at lower levels than those mentioned above. Once Nancy reviewed my test results and knew how I was responding to the treatment she modified my protocol to include a liver support, and the addition of 3-4 new products that would also help balance me out. I keep an ongoing visual journal using photos which I share with her so she can see any new developments. During chelation I was more tired, needing to take naps and sleep longer on most days.
I also had some tiny red spots appear on the skin without breaking through.Red spots appear on the skin. Based on what was going, on Nancy pulled back on the chelation so that the process is even slower and gentler until my body can adapt.
Using shea butter and coconut oil mixture as a moisturizer seems to draw fibers and black particles from my skin if left on for 3-4 hours.
Most are not readily apparent unless you really look for them. Hair The Morgellons has moved into my hair.
An acupuncturist, pointed out after looking at my hair photos, that there were multiple "hairs" emerging from one follicle. This is probably why my hair feels thicker even though it has been brittle, weak and falling out. Helicobacter pylori, the bacteria I had with high levels the first time I was tested, is completely gone. There was a new bacteria that showed up on this round of testing that wasn't on the previous.
Nancy feels that this is something that could have morphed, or, could have been present all along, but like peeling an onion back, was hidden under layers. She is adding seven new supplements to my protocol to address adrenal support as sleep has become fitful and I feel somehow too 'plugged in.' Fight or flight mode. I'll be taking a prebiotic to address the new bacteria as well as some new products from Xymogen to open detox pathways. I'm excited to be moving in a new direction. Every time we do a new test, I see the value of this approach. Randomly throwing supplements at the body without being tested, with the hope that something might work, seems a dangerous practice.
Not terrible full-blown itchy, just enough to let me know that I was reinfected or maybe this is an allergic flare-up.
This episode might have lasted longer than necessary had I immediately washed all my clothes but I got lazy and didn't wash them for a few days. Was not constantly itchy, but found I'd get itchy at odd intervals, sometimes once a day, sometimes not for days at a time and sometimes for a mere few seconds or so. I am putting a garden in upstate and often have my hands in the soil (without gloves) and have found that I am fine. The only itching I've got is from the occasional mosquito bite (and yes, these are real mosquitoes). Now, instead of direct application of GSE I brush my teeth twice, once with toothpaste and once with a clean brush that has two drops of GSE on it. Note that I have been letting my supplements run out in the last few months because I needed a break from taking approx. I wonder if the return to itchiness had something to do with my letting down my guard and not watching my pH.
After the first day of swimming, I noticed small circular red blotches appear under the skin of my calves and thought, uh-oh, here comes trouble.
I FINALLY have a microscope again (it was replaced after more than 7 months at the manufacturer!).
I had a few breakthrough 'papercut lesions' on the hands which I don't know what to make of.
I stopped the Ormus because I wanted to document F-6, which has gotten quite a buzz in the Morgellons community. I still have 1.5 bottles left and intend to take it in a few months feeling I didn't give it a proper shot. For now, I want to concentrate on doing one therapy at a time, so I can really be sure what each does. October 17, 2011: Began using F-6 and have started a new link documenting that experience including photos showing what is emerging from the skin. I'm going to take it slow with this product as I don't want to have any major herxheimer episodes.
Caroline Carter, A healthcare worker that has Morgellons and works with ozone therapy (and who made her Morgellons worse with ozone saunas) said she cannot even be around it anymore.
I have since talked with another person that had strong outbreaks from doing an ozone sauna.
Ozone therapy is typically great for cancer, AIDS and herpes, but for this disease might make us worse.
There seems to be improvement to the hair which feels stronger and seems to be falling out less. A lot of "debris" is coming up through the skin, fibers, black particles, granules and many more fibers then ever seen before.
A week and a half later I ate some quinoa with my lunch and two hours later had 3 oatmeal cookies (oatmeal, walnuts, carob chips, baking soda, vanilla). This forage into complex carbs was unusual for me.While eating the cookies I started to itch. By the third cookie, I was having full-blown symptoms of itching, stinging and biting sensations (no crawling, thank God!).
The fact that it came on with such force and was related to food, makes me think that Morgellons counts among it's causes a hyper-allergic reaction to food.
In this case, I don't really know what specific ingredient triggered the reaction.By eliminating carbs again, symptoms quieted down. I am noticing flare-ups of the arthritis in my foot which had so improved at the beginning of my treatment (but I kill my feet dancing one night a week, so that could play into this). The ones I can visually see without a scope are bigger then I've had before but there is no increase in the amount I generally see with my scope in the skin.
Will keep you posted of new developments.I've noticed that when it's very hot (93-97 degrees) and my skin is exposed to the air, I have more sensations on my arms.
It's not unusual to feel a single pin prick sensation on my left upper arm, often I'll feel the same type of prick mirrored in the same spot on right arm sometime later, as if they were somehow linked.The other day on my bike I felt as if I hot poker were being pressed momentarily to my arm. When I looked down, there were three distinct tract marks in that spot, about a half, to an inch long that hadn't been there before. Since having Morgellons, it feels like the skin is no longer a reliable friend but is capable of erratic changes suddenly.
They took about 5 days to disappear.?September 27, 2012 • Live blood microscopyJust posted a new link with photos of my recent session with Rick Panson, a microbiologist and healthy body coach who did a live blood and dried blood microscopy and gave me additional guidance for my health.
It's a bird's eye view into the system, giving a fuller, deeper understanding of what's going on  then just looking at lab results—although lab tests have their place!
In the next two weeks I get more testing done so will post those results when in. There were many problems with the blood. We are in a seasonal shift and my fingers have had a few tiny papercut lesions since the change began.Started the protocol Oct. Beginning with one pill a day of capyrilic acid with one pill olive leaf extract and 1 tsp. The L-glutamine is supposed to strengthen the gut lining and prevent leaky gut syndrome caused by the yeast die-off. Will stay on this for a few days and slowly double the dosage, then determine where to go from there. 16, 2012 Went back to Rick Panson for another live blood analysis and posted the photos under my last blood microscopy.
This is encouraging for all of you who wonder how long it will take to knock back candida and even if our protocols are working. Rick recommended Candex and Nancy told me she loved the product except now they are adding corn to Candex (and it's unknown if it's GMO corn), so she is no longer recommending it.
Adrenal system had a number of markers indicating a lot of stress to the system, very low levels of vitamins C and B's, four different pathogenic bacteria showed up—Helicobacter Pylori was high, Clostridia, E.
Kreb cycle needs support. Heavy metals hair test—all toxic metals were down except arsenic which went up with cadmium. Lead which was extremely high on my first test showed a considerable decrease as well as silver but lots more chelation ahead. It's been 2 years since I started using supplements to pull the metals out and I think it will take a couple more years of work. The process is slow but steady which is a good thing because I think if the body released too much metal into the system at once there would be negative impact on the health. Nancy pointed out that lead and cadmium are antagonistic to calcium absorption and bone health, so seeing this rise in cadmium and knowing I still have too high levels of lead makes me think that I might have to give up the painting at some point or switch mediums rather than compromise bone health. The heavy metals test also measures "essential and other elements," (minerals) It seems as a consequence of pulling heavy metals out, the minerals have been compromised. Nancy suggested this organism is a pleomorph moving from bacteria to fungus according to favorable conditions in the body. This is the third time it's changed. Feb 3, 2013 Just an acknowledgment that I just passed my 3rd anniversary since getting sick with Morgellons.
The only time I really have symptoms is when I really mess up on diet (itchiness will start) or when ?very overheated and sweating from physical exertion like dancing, where it sometimes feels like needle pinpricks are sticking my skin from the INSIDE out, accompanied by itchiness.
Thieves is a great oil for preventing flu or when one is coming down with illness, but I am unclear if it can damage beneficial bacterias. Stopped taking thieves after a few weeks but do still take lemon and added in lemongrass and grapefruit to my daily capsule. Itchiness did not return. September 22, 2013 Decided to go on Stephen Buhners herbal protocol for Lyme disease (crafted from his excellent book, Healing Lyme). Although I tested negative for Lyme and feel fine, I don't trust the traditional testing one gets with their regular MD, and even though I don't think I'm dealing with Lyme, I can't discount the fact that so many Morgellons sufferers also have Borrelia and other co-infections that might be related to our disease which seem to improve with Lyme treatment. Also, his protocol addresses other issues I feel are pertinent to my health (even before Morgellons). Certainly, I see over time that my immune system is weak, and my gut reaction now to coasting is that it could be an eventual time bomb.
I'm taking Cats Claw, Sarsaparilla root (Jamaican—which I grind in my coffee grinder), Eleuthero (Siberian Ginseng), Astragalus, Japanese Knotweed and Andrographis. To his formula I've added in Ashwagandha and Sweet Cinnamon (from Sri Lanka) for the brain, as well as nettles (excellent herb for trace minerals and general support of system) and Pau D'arco (fungal).
I take a quarter level teaspoon of each herb (which is roughly the equivalent of one 00 capsule) added to some water and drink it down. Surprisingly, I did not feel exhausted, although the protocol was pretty intense and caused a lot of flushing through the digestive system. This is not a new symptom and usually happens in the first few weeks of starting any new protocol.
As these breakouts have always been so minor and short-lived? (an occasional papercut lesion or what looks like a pin prick), I paid them little mind.
In this case, the skin continued to break open for the duration of treatment and I had some long "scratch" marks come up as well.
It seemed like the last papercut I got was deeper and for the first time I started to worry that perhaps I was giving myself a lesion problem that I might not be able to turn off. This little spot has bothered me on occasion on and off throughout the years of my Morgellons experience, but during the Buhner protocol, it itched almost constantly. On the plus side, I could tell there was a lot of detoxification going on and all of those bitter herbs were really helpful for quelling sugar cravings. I believe I will revisit this protocol modified in the future, but for now, am taking a rest from it.
Note that when I ceased treatment, the papercut and pinprick lesions stopped, although I recently had a three-inch long "scratch" appear on my leg weeks after stopping his treatment some weeks back. This was a surprise, since problems usually only show up on my hands and 3 inches is rather a statement!For the first time, the scratch resembled images I've seen for Bartonella. There is much about it that I think is helpful, and I encourage people to study these herbs and supplements carefully, and decide for themselves if they want to incorporate them. This is a very difficult protocol and for any who have detoxification pathway issues, this could impact their health negatively. The release of toxins in a system that cannot remove them properly can be deadly, so it is important to not rush carelessly forward with something this strong. I did feel like the protocol helped me on many levels, but I could not tolerate the heart issues with pressure on the chest, burning, and breathing difficulties. The creator of the protocol feels that Bartonella is at the core of the Morgellons infection, and Bartonella likes to set up shop in the endothelial tissue of the heart.
Thinking this was just a severe Herxheimer reaction, I went back on the protocol but was unable to advance more than one drop of each tincture before heart and breathing issues returned with a vengeance. This time, the heart issues did not abate immediately, and it's been some weeks now in which they often come back on a low level. His original protocol (which he posted on Jenna's blog and which is now removed) did not use MSM, or Silver Sol or various other supplements. It also did not include baking soda, which is not inherently a problem, but at 2 teaspoons a day, might become one.



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