Grand mal seizures or tonic-clonic seizures, according to MayoClinic website, is characterized by a sudden loss of consciousness and followed by severe involuntary muscle contractions. Grand Mal Seizures occur due to a generalized, abnormal electrical activity, and unregulated impulses throughout the whole brain.
Hypoglycemia is the condition wherein there is a sudden drop in the blood glucose levels, which can be caused by several factors, such as too high insulin dose, fasting, and many more.
During hypoglycemia, sudden drop of blood glucose levels can have bad effects to the brain. Flax for Diabetics -a nutritional powerhouse whole grain food has so many benefits for fighting diabetes. Flax for diabetes is a low glycemic food and has many benefitsl due to the Omega 3, fiber, protein + Lignans found in flaxseed.
Flaxseed benefits diabetes and adding flax to your daily diet may be the answer youA’ve been looking for. Protein in flax helps the body get nutrition that takes longer to digest and process helping provide more nutrition for longer. Research has shown that flax for diabetes as part of a daily diet can help lower and stabilize blood sugar levels. Flaxseed is an easy thing to add as part of a balanced diet and can make dramatic differences in your blood sugar as well as overall health.
Many of our own customer's have told us stories of how they have tried flax for diabetes and of the successes they have had in lowering and stabilizing blood sugars.
When I was introduced to Golden Flax, I was drinking fiber every day and still had to live on Senna. We don't want too high or too low blood sugar levels -as these extremes can cause damage to our bodies. The more vegetables, whole grains and low glycemic foods we eat, the easier it is for our body to keep blood sugars stable and within the normal levels. The more processed foods, sugar, white flours, white rice, etc; the harder it will be to control blood sugar levels and the more likely it is that we will suffer from diabetes and it's consequences. Here is a closer, detailed look into each of the 10 Ways Flax can help Fight + Prevent Diabetes. Flax for diabetics -a low glycemic food: you may have heard of low glycemic foods -they are foods that help stabilize blood sugars. With flax, blood sugar levels will slowly and gently rise to a lower plateau and then the blood sugar levels will stay there for a longer period of time.
After a few hours, your blood sugar level will slowly go down, this is the key to how flaxseed benefits diabetes. Whole grains are foods that take a longer time to break down and so our body gets more nutrition for longer, which are known as low glycemic foods.
Flaxseed benefits diabetes because it is a whole grains that is a very low glycemic foods -there are different values of low glycemic food.
Our blood sugars don't have to constantly spike up and down because the fiber plays a part in keeping them stable for longer. Soluble fiber slows down the absorption of glucose -this means that the body doesn't get slammed with the glucose all at once, but slower, this helps the body manage glucose levels and insulin production. Flax for diabetics is an economical and powerful step to take to help stabilize blood sugars toward the goals of fighting and preventing diabetes!
Endocrine glands are ductless glands that produce and release hormones to the blood through diffusion. Endocrine glands may be strictly endocrine, such as the pituitary, thyroid, parathyroid, adrenal, pineal and thymus; or they may be organs that have hormone production as one of many functions, such as the pancreas, gonads, hypothalamus, and others. Hormones are long-distance chemical signals that are secreted by the cells to the extracellular fluid and regulate the metabolic functions of other cells. Most hormones are amino acid based, but gonadal and adrenocortical hormones are steroids, derived from cholesterol.
Water-soluble hormones (all amino acid-based hormones except thyroid hormone) exert their effects through an intracellular second messenger that is activated when a hormone binds to a membrane receptor. Lipid-soluble hormones (steroids and thyroid hormone) diffuse into the cell, where they bind to intracellular receptors, migrate to the nucleus, and activate specific target sequences of DNA.
Target cell response depends on three factors: blood levels of the hormone, relative numbers of target cell receptors, and affinity of the receptor for the hormone. The concentration of a hormone reflects its rate of release, and the rate of inactivation and removal from the body. The half-life of a hormone is the duration of time a hormone remains in the blood, and is shortest for water-soluble hormones.
Permissiveness occurs when one hormone cannot exert its full effect without another hormone being present (reproductive hormones need thyroxine to properly stimulate development of reproductive organs). Synergism occurs when more than one hormone produces the same effects in a target cell, and their combined effects are amplified (glucagon + epinephrine together stimulate more glucose release from the liver than when each acts alone).


Antagonism occurs when one hormone opposes the action of another hormone (glucagon antagonizes insulin).
Nervous system modulation allows hormone secretion to be modified by the nervous stimulation in response to changing body needs. The pituitary gland is connected to the hypothalamus via a stalk, the infundibulum, and consists of two lobes: the anterior pituitary, or adenohypophysis, and the posterior pituitary, or neurohypophysis.
Two neurohormones are synthesized by the hypothalamus and secreted by the posterior pituitary.
Growth hormone (GH) indirectly (through insulin-like growth factors, IGFs) stimulates body cells to increase in size and divide.
Direct effects are insulin-sparing: mobilization of fatty acids for fuel, inhibition of insulin activity, release of glucose from liver to blood, and stimulation of amino acid uptake by cells. The thyroid gland consists of hollow follicles with follicle cells that produce thyroglobulin, and parafollicular cells that produce calcitonin. Thyroid hormone consists of two amine hormones: thyroxine (T4) and triiodothyronine (T3), that act on all body cells to increase basal metabolic rate and body heat production.
The parathyroid glands contain chief cells that secrete parathyroid hormone, or parathormone. The adrenal glands, or suprarenal glands, consist of two regions: an inner adrenal medulla and an outer adrenal cortex. The adrenal cortex produces corticosteroids from three distinct regions: the zona glomerulosa, the zona fasciculata, and the zona reticularis. The adrenal medulla contains chromaffin cells that synthesize epinephrine and norepinephrine (stimulus is acetylcholine released by preganglionic sympathetic fibers).
Insulin is an anabolic hormone and will stimulate not only glucose uptake but also storage in the form of glycogen (glycogenesis), fat (lipogenesis) and amino acid incorporation into proteins (inhibits amino acid breakdown by liver to form new glucose molecules - gluconeogenesis). Stimuli for insulin release are primarily high blood glucose levels but insulin release is also potentiated by rising blood levels of amino acids and fatty acids and release of acetylcholine by parasympathetic neurons (all of these things happen after a meal). Glucagon is released by the pancreas in response to low blood glucose levels (primarily) and raises blood glucose levels back to within normal range by stimulating glycogenolysis, gluconeogenesis, and release of glucose to the blood by the liver.
Indirectly receives input from the visual pathways in order to determine the timing of day and night.
Adipose tissue produces leptin, which acts on the CNS to produce a feeling of satiety; secretion is proportional to fat stores.
Adipocytes also produce adiponectin, which enhances insulin activity, and resistin, an insulin antagonist. Osteoblasts in bone produce osteocalcin, which stimulates pancreatic beta cells to divide and secrete more insulin.
Adiponectin levels are low in type II diabetes, suggesting higher levels may help reverse the insulin resistance characteristic of type II diabetes. Endocrine glands derived from mesoderm produce steroid hormones; those derived from ectoderm or endoderm produce amines, peptides, or protein hormones.
Environmental pollutants have been demonstrated to have effects on sex hormones, thyroid hormone, and glucocorticoids.
With the bleak outlook that 75% of Americans will be overweight or obese in 10 years, it is essential to find efficient help very soon. Given its vital importance, procurement of sufficient energy and essential nutrients is defended by a complex system consisting of redundant pathways to make it fail-safe. To make decisions, the brain keeps track of internal nutrient availability, a process often referred to as nutrient sensing. Both the absence and presence of nutrients in the gastrointestinal tract generates powerful signals informing the brain and other peripheral organs (FIGURE 2?). All other known gut signals essentially produce satiation of hunger, with little evidence that their absence reciprocally stimulates hunger. Not only the brain, but also the pancreas, is interested in information regarding about-to-be-absorbed nutrients, since it secretes hormones important for efficient partitioning and stabilization of plasma levels of nutrients. Tonic clonic seizures are usually associated with epilepsy, however, there are instances that people who are not epileptic can experience from a single attack of  generalized seizures during their lifetime.
As blood glucose levels rapidly drop, brain cells and different organs in the body are deprived with ATP, the energy source of all cellular functions in the body. When blood sugar is stable without a lot of peaks and valleys, it is easier to lose weight and IA’m game for that.
Flaxseed benefits diabetes because it is an excellent low glycemic food and flax will help stabilize blood sugars.
Flaxseed benefits diabetes and stabilizes blood sugars, so eating flax for diabetics is very helpful for stabilizing blood sugars.
Be first to find out about great discounts, delicious flax recipes, valuable health tips, flax seed articles and info! The stimulus for GHRH release is low blood levels of GH as well as hypoglycemia, low blood levels of fatty acids, and high blood levels of amino acids.


Hypersecretion of GH in childhood results in gigantism; in adulthood hypersecretion of GH causes acromegaly (increase in size of flat bones after epiphyseal plates of long bones have sealed). Thryroid releasing hormone (TRH) from the hypothalamus stimulates TSH release; Thyroid hormone (Thyroxine) exerts negative feedback control of both TRH and TSH. Excretion of ketoacids (with their negative charge) by the kidney is accompanied by loss of cations, particularly K+ and Na+. Secretion of resistin is proportional to fat stores; secretion of adiponectin is inversely proportional to fat stores.
Knowledge of the powerful and complex neural systems conferring the basic drive to eat is a prerequisite for designing efficient therapies. It causes secondary health risks such as Type 2 diabetes, cardiovascular disease, sleep apnea, and depression that significantly reduce the quality of life and burden the public health system. The major components of this system are accurate sensors of the internal milieu and the external world, flexible and adaptive integrators that make sense out of all this diverse input, and powerful effectors that act on both the input and the output arm of energy balance (FIGURE 1?). Although nutrients circulating in the blood satisfy immediate needs and are sensed continuously, information from stored nutrients and nutrients ready to be absorbed from the gut are in many ways more important, because they safeguard supply over longer time periods and decrease vulnerability to external factors.
Ghrelin secreted from the mucosa of the empty stomach is the only known hormone to stimulate ingestive behavior in a feed-forward fashion (22, 23).
In the stomach, such signals include mechanical distension of vagal stretch and tension receptors and locally produced leptin acting on vagal afferents (4, 65, 86, 91). This role of gut hormones on the endocrine pancreas was coined the incretin effect (FIGURE 2?). For people who are suffering from frequent seizures, daily doses of anti-seizure medications may be prescribed to help in controlling severity and the occurrence of seizures. During hypoglycemia, the brain’s supply of glucose is decreased, which can cause dizziness, fatigue, sudden weakness, headaches, shorter attention span, confusion, changes in the mental function, and worst, it can cause seizures and coma. These flax fibers fill up the body and take longer to digest -this means that we are getting the nutrition our body needs for longer and so we feel full for longer! Recent studies suggest that the cross talk between brain areas involved in cognitive, emotional, and metabolic-regulatory functions may explain why energy homeostasis breaks down for many predisposed individuals in our modern environment. With little or no effective treatments available, it is important to come to a better understanding of the controls of appetite and body weight and the major factors contributing to their breakdown in obesity.
Research efforts in the postleptin discovery era have mainly focused on the “metabolic brain,” identifying some of the crucial neural circuits in hypothalamus and hindbrain. Its plasma levels peak just before meals are requested and decline rapidly after nutrients enter the duodenum. The bowel generates a number of nutrient-related signals with some degree of macronutrient-specific encoding as an emerging principle (FIGURE 2?).
Together with circulating metabolites, incretins determine pancreatic secretion of a number of hormones that, in addition to their many peripheral actions, powerfully affect the brain. Generalized convulsive seizures may be life-threatening when it occurs in certain situations, since it can cause sudden loss of consciousness. Brain is one of the first organs that can be affected with low levels of glucose, since the brain has only a limited glucose reserve. Genetic predisposition and physiology are important determinants of proper functioning of these control systems, but clearly the changing environment and lifestyle are the primary engines of the obesity epidemic.
However, to understand how metabolic need is translated into strong behavioral actions that successfully compete with other motivated behavior, the role of the “cognitive and emotional brain” cannot be neglected. Ghrelin may be the explanation for “hunger pangs” thought to originate from the empty stomach mucosa rubbing against each other.
Although fat and protein appear to signal primarily through the release of CCK and perhaps PYY(3–36) (34, 43, 68, 76), glucose signaling may involve the sweet-specific G-protein-coupled taste receptor-1 heterodimers T1R2+3, the sodium-glucose transporter, and ultimately the release of 5-HT and GLP-1 from enteroendocrine cells (31, 38, 49, 50).
Given the exquisite glucose-sensitivity of the endocrine pancreatic hormone insulin and the co-secreted amylin, these two hormones provide important information regarding availability of the preferred brain fuel, glucose. However, most people suffering from it can feel an aura that they can recognize for an impending seizure attack. Diabetics who are taking daily insulin shots or other diabetic medications should be cautious, since they are at higher risk for grand mal seizures.
It will be interesting to see whether “taste receptors” are more generally used by the gut to drive nutrient-specific patterns of hormone release, thus encoding the macronutrient content of the gut.
It will be equally important to fully characterize other factors modulating hormone release from enteroendocrine cells, such as neural inputs from the enteric and autonomic nervous systems, paracrine inputs from neighboring enterocytes, and humoral inputs from the circulation. Perhaps, together with gastric volume sensors, intestinal nutrient sensors are able to quantitatively meter the gut content of each macronutrient available for absorption during the few hours after consumption.



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Blood glucose monitoring system ppt


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