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Diabetes is a disease characterized by high levels of blood sugar (glucose) in the bloodstream, resulting from defects in insulin production or insulin resistance. Let’s turn to a well-respected source of medical information, Lehninger’s Principles of Biochemistry (the so-called “bible” of biochemistry). Pretty straight forward, right?  People get fat because they eat too much relative to how much they exercise.
This subtle difference in actual causality, and the resulting misunderstanding that has led to current health and nutrition policies, is the root cause of the health problems afflicting us today. I think you should read a lot more studies on the actual distribution of nutrients rather than fixating on all of the known physiological effects of insulin. I should add that there is a lot more in all of the biochemistry involved that I truly grasp ?? and so I may have misunderstood some of this stuff. Dr Karim El Harchaoui (Academic Medical Center, Amsterdam, the Netherlands) and colleagues report their findings online January 22, 2007 in the Journal of the American College of Cardiology. Coauthor Dr Matthijs Boekholdt (Academic Medical Center) told heartwire that, in patients with only moderately elevated LDL-C, clinicians would do better to pay attention to HDL levels and triglycerides as markers for future CAD risk rather than worrying about testing LDL-P.
Sam, it’s certainly a good place to start by looking at TG to HDL-C ratio, and that it probably more helpful than LDL-C, but at least 30-40% of people with low or normal LDL-C have elevated LDL-P. I think the biggest mistake that many make when studying the etiology of obesity, which is a multi-faceted and multi-dynamic phenomenon, is to simplify it. We can’t pluck one hormone as the culprit because really, insulin is just part of the end game. I still am confused because I believe in the concept of low carb for insulin resistance and fat and ketosis but research seems to show a clear link between high protein diets and inflammation in the body that leads to disease. So therefore adding the fat keeps the protein lower and keeps inflammation lower in the body? Now we don’t have a nice yearly hungry moon or two or three, and our plenty is doing us in.
Question: Is the insulin receptor on a fat cell any different than the ones on a muscle cell?


The first statement in this comment is only applicable if one can accurately measure, for each individual human being, how they actually convert various food substances to energy or to store it as fat, or muscle etc.
Although I am not diabetic I have many hypoglycemia crisis, reason why I decided to become fat adapted. I wonder if there is a direct relationship (causation) between the high intake of carbohydrates and the diseases in the diagram.
First, I really like the way you put the science back into this discussion and that is what actually prompted me to reach out to you. Unfortunately I do not have the proper education to really understand what they are talking about but it sounds like they have shown that B-OHB fuels cancer growth.
This has been said to keep blood glucose and insulin levels constant, which should, in turn, promote insulin sensitivity. Can this be true, even when fructose is ingested from a natural source and combined with a form of protein? I am very interested in this due to my current studies on PCOS and its relation to insulin. Insulin also inhibits the breakdown and utilization of fat.  Parenthetically, this means no one disputes this graph.
Will it be possible for the body to change its metabolism to ketosis if the switch is a sudden one, not a gradual one? Richard Johnson finds that Fructose, by raising uric acid and inhibiting nitric oxide, completely blocks the effect of glucose to stimulate the secretion of insulin to increase the uptake of glucose in cells, thus causing insulin resistance.
I have been very low carbing for a few years and had achieved normal weight and good health but since reading your blog I reduced the proteins and increased the fat. Is it that Ketosis gets rid of this link whereas low fat protein diets like Dukan don’t?
Second the assumption does not take into account, gut absorption efficiency of various foods. Third the assumption does not take into account how composition of food effects hormones which have feedback mechanisms to alter how fats, carbs or proteins are metabolized by individual cells and their relative efficiencies. First attempt was tough, because I would pass out just from fat, if I didn’t ate carbs. If I understand correctly, the logic is:you get fat because you eat too many carbohydrates.
I would be interested to know if you could achieve similar results by changing the fasting days to lipid only days.
I have previously thought that cancer cells are exclusively fueled by glucose and never ketones. His clinical interests are nutrition, lipidology, endocrinology, and a few other cool things. Wow I now feel fabulous it have given me so much more energy, have more endurance, less hunger, clearer mind.


Most doctors do not look at HDL-C or triglycerides because, unlike LDL-C, treatment for these parameters is not mainstream clinical practice. On my low carb diet I have expected to be safe from developing cancer, but this piece of information suggests that all the ketones in my blood could potentially be feeding cancer cells.
While it is true that insulin is an anabolic hormone, the most negative effect of a high carbohydrate diet is that it strongly promotes the oxidation of carbohydrates. It would be more useful to be aware of these results, particularly in those with the metabolic syndrome, abdominal obesity, or diabetes,” he said. Experiencing with nutritional ketosis and I don’t need carbs at every hour and I can snack on nuts and fat without feeling hypoglycemia symptoms after. You see, the body actually cares about and monitors carbohydrates, while being oblivious to fat intake and storage. So just wanted to be sure that I was giving the correct advice since they all only had the standard lipid panel that the doctor orders. On a diet high in carbohydrates, ingested fat will be sent directly to adipose tissue while almost all the carbohydrate will be oxidized.
Robert Lustig, says that Fructose keeps insulin levels elevated thus causing more fat storage.
At the end of the day eating100 grams of lean meat does not equal 100 grams of sucrose in weight control even though they both represent 400 calories. It may not be a large issue in practical terms (you will still be adding fat) it will not be due to insulin directing glucose into the fat cells to be converted to fat. This is totally giving my life back, the only problem is I’m experiencing a pain in the right side.
In fact, that would be the best possible outcome, since the process is inefficient enough that you would only gain around 65% of the weight you would gain by directly storing fat.
It has been calculated (see Hellerstein) that less than 10% of the fat in your body was synthesized by lipogenesis rather than ingested as fat.
For folks on a standard diet, a full 50% of their ingested fat will go directly into a fat cell rather than be oxidized. For folks on a high carbohydrate diet, they will have to consume more calories than they can burn or store as glycogen to even begin to show appreciable conversion of carbohydrate into fat.
Yes, but it is a minor metabolic pathway in humans, and most glucose will be disposed into muscle and liver.



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Comments

  1. 25.05.2016 at 15:43:44


    And eating a diet rich in vegetables, fruit and must be emphasized so that progression insulin release.

    Author: SAMURAYSA
  2. 25.05.2016 at 23:22:32


    And they're only $9.95 for 50 strips factors listed above?should be tested for.

    Author: 027
  3. 25.05.2016 at 19:33:54


    Sugars to rise into the diabetic testing.

    Author: Immortals
  4. 25.05.2016 at 17:34:52


    Body, to try and alter the levels of insulin.

    Author: 202