Mouse fathers under psychological stress were more likely to have offspring with high blood sugar compared to their unstressed counterparts.
Li and her colleagues confined male mice in plastic tubes for 2 hr a day, for 2 weeks straight, to induce stress. The good news is that by understanding the mechanisms involved, the researchers could block the effects of glucocorticoids on the mouse father's sperm. This study was supported by grants from the National Key Basic Research Program of China and the China Natural Science Foundation. The real story is that the number, size, and density of cholesterol particles in your blood (LDL-P and HDL-P) are far better predictors of heart disease risk. The best way to measure your heart disease risk through LDL cholesterol is to measure the number of LDL particles in your blood, or LDL-P, which you never get checked unless you have a fancy test called a lipid nuclear magnetic resonance test – or NMR test for short. You can look at particle size.  As a general rule (this is NOT always the case, however), the larger the LDL particles, for a given LDL-C, the fewer the particles (which is what we want). Below is graph of my overall change in changes in HDL-C, LDL-C, and TG, along with the ratio of my TG to HDL-C, based on the “standard” cholesterol panel.
As I stated above, a better marker of risk with respect to LDL is particle number, LDL-P – the fewer particles, the better; and you can estimate this by measuring particle size, or through concentration of ApoB.
Unfortunately, I only started doing regular VAP testing about a year ago, over one year into my “experiment” of progressive carbohydrate restriction.  Hence, I can’t show my progress as longitudinally with VAP as I can with standard cholesterol testing. Below is figure showing the change in my VAP panel over a seven month period, between January and July 2011. Keep in mind how my diet changed between January and July – I reduced carbohydrate intake from approximately 150 grams per day of “good” carbs to less than 50 grams per day.  I also increased, dramatically, my intake of fat, including saturated fats. Despite the amount of time I’ve expended on explaining all of these nuances of “cholesterol” numbers, I am not entirely convinced that I am healthier today because my cholesterol numbers are better.  I wonder if I’m healthier today because of something else, and that whatever else is making me healthier is also correcting my cholesterol problem? If I had to guess what is really making me healthier today, besides being less fat, I believe it is the combination of how sensitive I’ve become to insulin and how much less inflammation I have in my body, especially in and around my arteries. As I mentioned above, findings #1, 2, and 4 are almost universal in folks who abandon carbohydrates, while finding #3 is somewhat variable. Which of these is most important?  This is an obvious and important question, but one I don’t really know the answer to (nor does anyone else, for that matter).  If I had to guess, I believe observation #4 is the most important because insulin resistance is the underpinning of metabolic syndrome. People have said things to me like, “Well it’s great that you’ve reduced your risk of all diseases associated with metabolic syndrome, but wouldn’t it be funny if you got hit by a car tomorrow!”  All kidding aside, this misses the point.


Teenage stress is one of the most important challenges of a person’s life, be it a female or a male. Research and studies have clearly shown that girls are most likely to suffer from teenage stress than boys.
In high school, a given group of students may categorize themselves as special or people from high class families; thus, ending up discriminating any other student that does not uphold such status. Peer pressure is another contributing factor; for instance, a given group of boys may end up convincing their noble friend to drink alcohol or smoke cigarette. In a study appearing February 18 in Cell Metabolism, researchers link this difference to an epigenetic change in the stressed dad's sperm--a change that they could prevent by blocking the father's stress hormones.
When a male mouse is immobilized daily in a plastic tube, the spike of glucocorticoids causes extra methyl groups to be added to the Sfmbt2 gene in his sperm. The intronic microRNA-466b-3p in Sfmbt2 is supposed to help regulate an enzyme called PEPCK, which controls sugar production in the liver. Injecting the male mice with a molecule that dampens the effect of glucocorticoids stopped the Sfmbt2 gene from being overly methylated. The journal aims to highlight work addressing the molecular mechanisms underlying physiology and homeostasis in health and disease. Well, there are two: what can I actually measure that predicts my risk of heart disease, and how does diet affect these these things I can measure?
Basically it’s a test to measure how much insulin a person needs to keep their glucose level constant, despite the addition of glucose.  The less insulin one requires, the more insulin sensitive one is. The teen age group is the one that experiences almost all sorts of changes; ranging from social, academic, sexual, and economic changes.
Such an individual will be stressed up on what will happen if his or her parents were to find out.
Their only aim is to share their opinion on the concerned topic, and help the reader understand it better. The study adds to growing evidence that a male's life experience can be passed down through more than his genetic code alone. These epigenetic marks don't affect the underlying DNA, but they do control how Sfmbt2 and an associated microRNA (the intronic microRNA-466b-3p) are expressed.
But when mammals reproduce, Sfmbt2 is turned off in the egg from the mother--meaning that offspring inherit their only working copy from the father's sperm.


Early relationships are accompanied by breakups that are mainly promoted by cheating, teen quarrels, and feeling of jealousy.
It won’t do any harm, if a mother sits down and talk things out with her daughter in order to know the latest developments in her child’s life.
Good or bad, Right or wrong is solely readers decision and should be taken under the guidance of a medical expert. And when the only functional Sfmbt2 gene carries these epigenetic tags, the intronic microRNA-466b-3p is silenced and can't keep watch over PEPCK as it normally would.
Girls are also bound to fall victims of stress due to their involvement with each other in terms of friendship. A teen who witnesses his or her parents fighting frequently will be constantly worrying if his or her parents may end up divorcing or separating.
The father's offspring then develop livers with too much PEPCK, causing their blood glucose to increase. Moreover, there are high chances that a teenager will attempt to weigh out between studies and recreational activities including nightlife.
Such friendships are accompanied by girl gossips, girls falling for the same boy among other conflict of interest.
Such events will definitely stress the child and he or she may consequently experience difficulties at school due to the racing thoughts in his or her mind.
On the other hand, a teenager is also required to take control of his or her life by consulting his or her parents whenever he or she has a problem. In some peculiar occasions, family members are also considered to be causes of stress in teenagers. For instance, a father may demand too much from his son or daughter in terms of academic grades.



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