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Human Growth Hormone Steroid Hygetropin Kigtropin Jintropin HGH CAS 96827-07-5 IGF Quick Detail: Improved cholesterol profile Jintropin Human Growth Hormone for injection. Background and Objectives: Recombinant human growth hormone (rhGH) is approved for use in children with Turner's syndrome (TS) in most industrialized countries and is recommended in the recently issued guidelines.
How to cite this article:Ismail NA, Metwaly NS, El-Moguy FA, Hafez MH, Abd El Dayem SM, Farid TM.
How to cite this URL:Ismail NA, Metwaly NS, El-Moguy FA, Hafez MH, Abd El Dayem SM, Farid TM. 7.van Pareren YK, de Muinck Keizer-Schrama SM, Stijnen T, Sas TC, Jansen M, Otten BJ, et al.
17.Rongen-Westerlaken C, Corel L, van den Broeck J, Massa G, Karlberg J, Albertsson-Wikland et al. Bone age is the best predictor of growth response to recombinant human growth hormone in Turner's syndrome. TS is caused by deletion of all (monosomy) or part (partial monosomy) of the second sex chromosome. The turner syndrome consensus study group care of girls and women with turner syndrome: A guideline of the turner syndrome study group. Final height in girls with Turner syndrome after long-term growth hormone treatment in three dosages and low dose estrogens. Spontaneous pubertal development in turner's syndrome: Italian Study Group for Turner's Syndrome. Fertility preservation in girls with turner syndrome: Prognostic signs of the presence of ovarian follicles. Reference values for height, height velocity and weight in Turner's syndrome: Swedish Study Group for GH Treatment.
Effect of growth hormone treatment on adult height in peripubertal children with idiopathic short stature: A randomized, double-blind, placebo-controlled trial. Growth hormone therapy of turner syndrome: The impact of age of estrogen replacement on final height. Adult height and pubertal growth in Turner syndrome after treatment with recombinant growth hormone. Impact of growth hormone supplementation on adult height in Turner syndrome: Results of the Canadian randomized controlled trial.
Prediction of long-term response to recombinant human growth hormone in Turner syndrome: Development and validation of mathematical models.


Treatment with two growth hormone regimens in girls with Turner syndrome: Final height results. Multiple body systems can be affected to varying degrees, presenting both diagnostic and management challenges for the pediatrician. Bone age delay is to be considered as a predictive factor which may negatively influence the effect of rhGH therapy on final height. Analyses based on comparison with historical controls have shown variable estimates of gains in mean height, ranging from minimal in a study by Canadian investigators [7] to up to 17 cm for high doses of GH. In utero, the ovaries have a decreased number of primordial follicles; these appear to undergo premature apoptosis [10] and are usually absent by adult life. Therapy may be started as early as 2 years of age, although there is presently only limited experience of treating children of this age. Early GH treatment can correct the growth failure and normalize the height in infants and toddlers with TS.
Their demographic, anthropometric and hormonal factors which may be related to their therapeutic response to GH were analyzed.
Also, the effect of therapy on GH mediators, such as IGF-I and IGF-I BP3, in the same group of patients was evaluated.PatientsFifty-six prepubertal girls with TS, documented by peripheral blood karyotype, were enrolled.
Inclusion criteria were euthyroid TS cases having short stature, more than 2SD below the mean, or growth velocity (monitored over 6-12 months) below the 10 th centile for age and sex. The exclusion criteria were phenotypic females with identifiable Y chromosome material, cases with chronic diseases or any relevant cardiac or renal abnormalities, and those who have had prior treatment with GH. Puberty was assessed by rating the breast development in girls, pubic and axillary hair development, according to Tanner's classification. Thyroid profile (FT3, FT4, TSH) was done to exclude primary or secondary hypothyroidism as a cause of short stature. Thyroid stimulating hormone (TSH) was estimated by immunoradiometric assay (IRMA), while FT3 and FT4 were estimated by radioimmunoassay kits from Diagnosis Product Corporation, (Los angeles, CA, USA.)2.
Routine general laboratory tests, if needed, which include complete blood picture, renal and liver function tests.3. Insulin like growth factor-1 (IGF-1) and IGF binding protein-3 (IGFBP-3) were determined at diagnosis, by solid phase IRMA, using kits from Diagnostic System Laboratories Inc. DSL-5600 IGF-1 (IRMA) was included in a sample extraction step in which IGF-1 was separated from its binding protein in serum.
Puberty was not induced by giving sex hormones during GH treatment, since the treatment was started relatively late in these patients.
The laboratory of DEMPU, Cairo University children's Hospital, provided the mean values for IGF-1 and IGFBP-3.Statistical analysisThe SPSS software computer program was used for data analysis, and Harvard graphic was used for figures.


Quantitative data were presented as mean ± SD, range and frequencies, and qualitative data were presented as percentage.
For comparison of the two groups, the Student's t-test for dependent and independent variables, was used.
For comparison of more than two groups, one-way analysis of variance (ANOVA) was used, followed by post hoc test, when significant. It is easy to misinterpret the absence of puberty and small size of these patients as due to constitutional delay. Being a type of skeletal dysplasia was previously suggested by Bridges and Brook; [21] however, X-rays lacked convincing evidences for skeletal dysplasia. They randomly assigned 154 children to treatment with GH or no treatment and both the groups were observed until the adult height was reached. The mean duration of treatment was 5.7 years, and puberty was induced pharmacologically at a chronological age of 13 years. For the second year, growth velocity of the first year was the strongest predictive factor (positive correlation), followed by target height SDS (negative correlation) and IGF-1 SDS (positive correlation). For the third year, growth velocity of the preceding years was the most important predictive factor (positive correlation). Predictive factors in the second and third years of GH therapy explained all of the variability in the response.
So, it is apparent that during the period of catch up growth in girls with TS, BA delay was the most important predictor; the more the delay, the better is the growth response, giving a potential chance for better growth. Age, weight SDS, additional treatment with oxandrolone, the difference between the height SDS and mid-parental height SDS and frequency of injections were other predictors. These variables explained 46% of the variability of the response with an error SD of 1.26 cm.
Height velocity in the preceding year was the most important predictor in the second to fourth year, with GH dose, age, weight SDS and addition of oxandrolone to treatment as the other predictors. Monitoring of IGF-1 and IGF-BP3 levels in girls with TS during the initial 3 years of GH therapy showed a significant rise of IGF-1 values in the first 2 years, whereas the IGF-BP3 values showed a significant rise in the first year followed by a significant drop in the second year. The significant rise of growth factors IGF-1 and its binding protein-3 during early period of GH treatment, which is followed by their decline, may be related to the initial catch up growth which is followed by a steady pattern on the continuation of GH treatment.In conclusion, this study seems to show that GH therapy provides a satisfactory auxological result.



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