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Published 06.03.2015 | Author : admin | Category : How To Make More Money

Children’s increased use of smartphones and tablets could hinder the development of parts of their brain that affect social skills, a psychology researcher said. The parts of the brain that determine attention span can be severely limited if boredom is immediately alleviated rather than endured, Cavell said.
More than 70 percent of children under the age of 8 use a smartphone weekly and 28 percent of parents use technology as a parenting supplement, according to a 2012 American Academy of Pediatrics study.
The mood of a child can make a short trip to the grocery store a multi-hour ordeal, Black said. Parents who give their children devices to prevent outbursts are practicing a don’t-think-twice attitude that ignores proven child-rearing techniques, said Gene Saunders, a UA family science professor. More confrontational methods of parenting increasingly are abandoned because the results are not immediately apparent, he said. Saunders cited an essay published by the Yale University Press on the importance of boredom. Boredom coping mechanisms that are developed internally can increase an overall attention span because they can be engaged depending on the importance of information, he said. Researchers at Concordia University in Irvine, Calif., concluded that children born since 1990 have almost 80 percent fewer instances of social interaction in elementary school than previous generations.
Many parents disagree with electronic babysitting, said Andrew Miller, a parent and UA education major.
Anthony, Miller’s 6-year-old son, is limited to an hour of supervised electronics usage every day. Children should be allowed to use technology only when the parent knows that their child is mature enough to use it responsibly, he said. The Concordia research also concluded that the average age of social maturation has steadily increased.
Digital communication originally was designed to break long-standing communication barriers, but in practice it seems to have done the opposite, Saunders said. Instances of social hindrance caused by digital devices can be seen as early as elementary school, said Karen O’Hara, who teaches fourth grade in Harrison, Ark.
Her students haven’t formed bonds, because of smartphone use during recess and other breaks O’Hara said. The rule change that allowed students to use cellphones during breaks was originally designed to give students a chance to call working parents to arrange transportation and events. Children who are allowed to engage in this type of isolated behavior are having their potential curbed, Cavell said.
September 28, 2015 By Matthew Jones The moon has a reddish tint during a lunar eclipse on Sunday, Sept.
Technically, baclofen is a muscle relaxer and antispastic agent used to treat muscle symptoms caused by multiple sclerosis, including spasms, pain, stiffness, etc. The role of GABA is to inhibit nerve transmission in the brain thereby calming nervous activity. While a balanced brain receives regular, smooth electrical impulses, a GABA deficient one receives them in spurts. Natural anti-anxiety nutrition includes GABA producing foods you can easily incorporate into your daily diet. This entry was posted in PTSD Recovery Tips, PTSD Treatments and tagged baclofen, GABA, neurotransmitter, ptsd treatment. I was given baclofen for another condition but noticed my complex PTSD was suddenly absent. Been a while since this post was written, however, can I get in contact with you in some way?
Lately I’ve been getting better and better with a Paleo diet, but a ketogenic one which seems to reduce my pain levels a lot. I went on the keto diet because people were always prescribing things like Lyrica, Gabapentin, Amytriptiline etc… to try and control my pain and I was sick of it. This site complies with the HONcode standard for trustworthy health information: verify here. Even traditional clamshell notebooks have adopted tablet-like capabilities, as both Acer's new Aspire R 13 and 14 series, as well as the new Aspire Switch 10 and 11 2-in-1s, can now be detached or folded back into a tablet mode. Acer launched the next generation of its mainstream Aspire R series at the IFA show in Germany on Wednesday, touting the literal flexibility of both families of R-series notebooks. With more buyers at least considering the option of a tablet to replace or supplement their traditional PC, Acer seemed to bend over backwards to emphasize its flexibility.
While both of the Aspire R-series notebooks use traditional Intel Core microprocessors with either standalone or integrated graphics, Acer also adopted Intel's latest Atom microprocessors as low-cost, moderate-performance computing options for its consumer offerings.
Our earlier review of the Switch 10 found two significant problems: the anemic display and the equally poky processor.
The larger Switch 11, meanwhile, ships with 11.6-inch displays made out of Gorilla Glass 3. The Acer Aspire R13 in "easel mode," where the display can be brought forward to make viewing easier on the user.
Acer spent considerable effort highlighting the flexibility of each display, a design aesthetic that was popularized by the Lenovo Yoga and similar models. Somewhat surprisingly, perhaps, the larger Aspire R 14 family only includes a 14-inch, 1,366x768 display option. Acer only revealed pricing for the Aspire R 14 series, with prices starting at $599 in October. As PCWorld's senior editor, Mark focuses on Microsoft news and chip technology, among other beats. PCWorld helps you navigate the PC ecosystem to find the products you want and the advice you need to get the job done. An infection in the eye can be characterized in the form of redness, irritation, achiness, excess drainage and eyelid swelling.
If your eyes are affected by a burning sensation, you can use the tea bags for reducing the ill effect which is common with a number of eye infections.
A warm compress is helpful in reducing the burning and itching symptoms associated with eye infections such as blepharitis and sty. Omega 3 fatty acids are found in fish like salmon, tuna, sardines and mackerel, and vegetarian foods, including walnuts, hemp seeds, anchovies and egg yolks among others. Boric acid treats a number of eye problems like dryness, redness and also reduces burning sensation. The views expressed in the contents above are those of our users and do not necessarily reflect the views of MailOnline. You must have JavaScript enabled in your browser to utilize the functionality of this website. More interesting still is that you’ll be able to finally forget about dual-, triple-, and quad-channel memory configurations.
GDDR4 was used only in a few graphics cards, so why not jump straight to DDR5, like what AMD and nVidia did(GDDR3 to GDDR5)?
The ideal that there will be hay to make with hollywood specs,say in DRM,codecs as well is severely overrated. Polarization into single sources of products,and supposedly shortage of quality products,and topics including good articles of technological comparison ?
From time to time every psychiatrist comes across patients whose problems are at least in part related to the neuropsychiatric consequences (behavioral, cognitive, and emotional) of traumatic brain injury (TBI).
Several researchers agree that at a young age, children are unable to comprehend their purpose.
Parents use the bright screen of a cellphone to save the family from lengthy tantrums and public embarrassment. Boredom: A Lively History, included a survey that concluded the average person is bored for 40 percent of the day. External distraction however, is more likely to cause the person to completely ignore the situation. However, several teachers want to change the system because they feel the rule is being abused, O’Hara said.
Younger children especially will begin to associate technology as a form of social escape, even if they can’t comprehend the actual intent. She sent links withthe results of a study examining the efficacy of baclofen in PTSD treatment. As a result, the brain experiences arrhythmia, or dysrhythmia which directly affects overall emotional well-being. In a recent study of fourteen PTSD survivors taking baclofen in the eleven who completed the 8-week study every single one experienced a reduced rating on the Clinician-Administered PTSD Scale.

Taking steps to offer your brain nutritional support that can help stimulate the production of GABA, your brain’s very own calm inducing element, is an easy, economical and healthy way to do something that really takes good care of you. Because the medicine was prescribed for a different condition, obviously I wasn’t having a placebo effect.
I’d really would like to know some more about which CPTSD-symptoms Baclofen helped you with. Acer also launched new Switch all-in-ones, with a 10.1-inch model that features a full HD display and the new 11-inch Switch 11 series. The Aspire Switch SW5-171 uses a fourth-generation Core i5 driving a 1080p display, but the Switch SW5-111 includes an Intel Atom Z3745, a "Bay Trail" Atom chip Intel is trying to establish as an alternative to Qualcomm embedded processors via "contra revenue," or payments to hardware makers. Acer has solved at least one of those issues, offering either a 1,920x1,200 or 1,280x800 display option on the newA Switch SW5-012 for an undisclosed price. The Switch 10 and 11 can detach the tablet portion via a magnetic latch that, at least on the earlier Switch 10, was almost absurdly strong. All of the models include either aA A 2,560x1,440 or 1,920 x 1,080 IPS display with what Acer calls "air gap" technology to improve legibility.
The common eye infections include conjunctivitis (red eyes with accompanying irritation), sty (formation of small lump within the eye), blepharitis (itching and burning sensation with the eyelids), orbital cellulitis and keratitis (injury to the eye because of foreign object). It is important to consult the doctor when you have any symptom associated with eye infection.
Dip a clean cotton cloth in comfortably warm water (so that it does not causes burns), wrung it to remove excess fluid, and place it gently on the affected eye. It also cleans the infectious agents away. Put cold water into a basin or sink and fill the sink amply so that you can dip your face in it.
Mix equal parts of honey and distilled water, and apply the solution to the eye by using a clean cotton ball. The antibody by the name Immunoglobulin E, which is found in the milk, kills harmful bacteria and limit damage to the mucous membrane of the eye.
Intel needs to change their On Die Memory controller for their Desktop CPU’s to use the new memory unlike AMD where they tend to make a change that affects the entire product line. Intel likes to diferentiate their CPU’s and this just gives them another marketing point in their differentiation efforts. Just what from our perception does this constrasted non-existed conflict proposed to result in ? Actually what changed from DDR to DDR2 to DDR3  and now to DDR4 is always the same, they up freqs, lower voltages and increase latencies quite a bit (measured in clocks).
The article also concluded that the main harm done when using smartphones or tablets as boredom alleviation is that it doesn’t allow children to develop their own methods of coping with boredom. The cited cause for the increase was an increase in protective parenting and the increase in personal electronics. It elevates the production of alpha waves associated with feeling relaxed (without drowsiness) and boosts mental alertness. It took some research before I found a 2008 small sample study with baclofen and PTSD that showed a 100% efficacy. The SW5-171 is the cheaper model, with a 1,366x768 display, the Intel Z345 chip found in the earlier Switch 10, 2GB of memory, and a 64GB MMC flash hard drive. Users can choose from either Core i5 or Core i7 microprocessors, but not the upcoming Intel "Broadwell" Core M chips, apparently. Home remedies can be used for lowering the irritation, burning, swelling and other associated symptoms and health conditions that relate to eye infections. As Bit-tech pointed out, this is a continuation of the simplification process that memory has undergone ever since the move to DDR. Think it should be gauged when the products are there to comment on an compare rather than monopolising what we can or cannot bring to our perception.
The end result is they consume less energy, have higher bandwidths and access time doesn’t change much.
Those who are vulnerable to mental illness (eg, persons with alcohol abuse or antisocial personality disorder) are particularly at risk.
GABA lowers beta waves, impulses that contribute to a state of nervousness, racing thoughts and hyperactivity.
Acer's Aspire R 13 series models can also be folded back on themselves, Yoga style, to serve as a display or "tent mode." The R 14 uses a 180-degree hinge that allows the display to face away from the user or support the weight of the notebook in a tent or "easel" mode, sliding forward. Use clean and hygienic eye dropper for putting 2 to 3 drops of the milk in the infected eye. Then with the cell phone,sure with the advent of an HDMI connection you will be able to utilize the big screen,but just take a look at the limitations and cost of the monopolies carriers (and government) have on that technology.
Reproduction in whole or in part in any form or medium without express written permission of Ziff Davis, LLC. Patients with TBI often have poor insight and may need hospitalization for their own safety. Fortunately, the Windows 8.1-powered 2-in-1 also ships with a year's subscription of Microsoft's Office 365 software installed.
The R 13 series is Skype-certified and includes the Microsoft Surface-like Acer Active Pen, which can be used to launch Microsoft OneNote or a screen-capture app. Strain the mixture by using a clean and hygienic cloth, and use the solution like an eyewash. Robert Pearce, the company’s senior technical marketing specialist, gave me some good information. This means that while there is higher peak bandwidth (which can provide performance improvements in a server environment where multiple accesses from completely independent threads can be queued up), the time needed to get data for a given request will not really change.”Sure, what will happen with DDR4 remains to be seen, but put me in the “optimistic” column. Just look at the monthly cost of haveing one of those cell phones (smart phones),- the carrier makes sure the meter is running every day.
I recall people saying a lot of similar things about DDR3, too, a few years ago, and that eventually earned wide-scale adoption. The illustration is a composite of the location of contusions found in 50 patients who died of TBI. Also, I could split a pill and take 5-10 mg extra when faced with a particularly stressful situation. Even if DDR4’s changes on most fronts are minor, I think the industry will come around. The sites of specific vulnerability to contusions are the medial orbital frontal lobe and the anterior temporal lobes (Figure 1). Areas where contusions rarely occur include the primary motor, somato-sensory, and visual cortex. I can do without my baclofen completely although I do keep some on hand in case I’m ever in another high stress situation. Diffuse axonal injury affects white matter anywhere throughout the cerebrum and brain stem. It’s the only thing that was able to retrain my brain to stop the excessive fight-or-flight reaction. It may be followed by generalized atrophy with ventricular enlargement (Figure 2); this may take a few weeks or months to develop.
Diffuse axonal injury in the brain stem is usually responsible for the slurred speech and severe ataxia that are seen in some severely disabled patients after TBI. I hate that so many people are suffering excessively when there is something so simple that can immediately be done to ease the symptoms.
Contusions and diffuse axonal injury may be complicated by anoxic brain injury that may occur soon after trauma because of poor cerebral perfusion secondary to raised intracranial pressure and focal strokes. In some patients, localized infarction occurs (Figure 3).Neuropsychiatric assessmentThe neuropsychiatric assessment starts by evaluating the severity of brain injury.
In this way, the likely outcomes attributable to direct effects of brain injury can be determined, and any mismatch between these and what is observed can be attributed to psychological reactions or independent events.
So, for example, in somebody with a severe psychotic illness that develops 3 months after an injury with no loss of consciousness, one can be fairly confident that the illness is not a direct consequence of the effects of brain injury on delusion formation.
It is possible that the psychological trauma of the injury has allowed an acute psychotic reaction, or even that the injury was irrelevant and that the person was on the path to becoming schizophrenic anyway. If PTA lasts longer than 1 month, significant disability is likely; a good proportion of those affected will not be able to return to work or to independent living. In general, younger individuals (those in their late teens or 20s) tend to do much better.An MRI scan is essential in cases where the extent of damage is unclear because it may show unexpected brain injury.
Gradient echo sequences are the most sensitive and should be undertaken, particularly in those with mild injury. A normal MRI scan does not rule out brain injury, but it does make significant disability as a direct effect of severe brain damage unlikely.
Electroencephalography is usually not helpful, even as a predictor of posttraumatic epilepsy.Neuropsychometric assessment can be useful in defining the severity of cognitive impairment and any areas of particular impairment.

Such tests as the North American Adult Reading test are available and provide an estimate of the patient’s preinjury IQ.
Such assessment is necessary for the accurate interpretation of a patient’s postinjury performance. Note, though, that normal neuropsychometric test results do not rule out brain injury as the cause of problems with executive functions in everyday life.Cognitive and behavioral sequelaeIrritability and aggression are probably the most common behavioral consequences of TBI. However, it can be difficult to know the extent to which brain injury is a factor in aggressive behavior. Many patients who have sustained a TBI were prone to antisocial behavior before the injury. It is sometimes argued that because a behavior is sensitive to psychological cues, it is not the result of a brain injury; however, this is not correct. For example, just because extreme aggression is seen only in the context of the family and never at work does not necessarily mean that the aggressive behavior is unrelated to a brain injury.Teasing out the role of brain injury can be difficult and usually relies on a good objective history of the behavior before and after the injury, along with an assessment of the likelihood of significant brain injury, which depends in part on the location of the injury. Cold, goal-directed aggression is seen in patients with psychopathic personality disorder, whereas the aggression of the brain-injured patient is usually impulsive and quite out of proportion to the trigger. But in practice, it is not easy to discern the origins of aggression (constitutional vs brain injury) from its phenomenology.In patients with severe brain injury, a typical clinical picture consists of significant cognitive impairment, particularly in the domains of attention and concentration, psychomotor speed, memory, and executive function, as well as fatigue and problems with motivation. Approximately one-half, one-quarter, and one-eighth of patients still have significant symptoms at 3, 6, and 12 months, respectively.4 Typical symptoms include headaches, fatigue, dizziness, depression, and difficulties with concentration and memory, which are often complicated by anxiety symptoms related to travel and posttraumatic stress disorder.
Alteration of cerebral blood flow during working memory tasks 1 month after injury and long-standing changes in fractional anisotropy—an MRI measure of white matter integrity—have been shown.5These postconcussional symptoms are nonspecific and are seen almost equally often in patients with musculoskeletal injuries but no head injury and in patients with chronic pain or chronic fatigue syndrome. In some patients with long-standing postconcussional symptoms, the extent and severity of the symptoms suggest that the illness is a form of somatization disorder.
Perhaps the best model is that symptoms soon after injury are secondary to the direct effects of trauma to the head and brain, but that over time psychological factors intervene to prevent a healthy recovery.6Management of patients with TBIAgitation in the early postinjury period (eg, days or weeks after a severe injury) is common and usually self-limited. There may be evidence of delirium and the patient is likely to show poor orientation and poor insight.The development of agitation is a warning that something may not be right physically.
For example, the patient may have thrown off some fat emboli from a fractured femur or be in urinary retention. Agitation may also be the first sign of infection entering through a cerebrospinal fluid leak.
Intoxication from medication may be to blame, and in some, agitation is a manifestation of craving because of substance abuse at the time of the injury.Management rests on principles similar to those used in patients with delirium. The main task is to ensure the safety of the patient and others, monitor the patient’s physical recovery and prescriptions, and wait for improvement.In the longer term (months and years after injury) aggression can be a major disability.
A good rehabilitation program can help by improving engagement in activities and self-confidence. Specific anger management techniques should be tried, but they do not always work.Drug treatment should not be started at the first sign of agitation and aggression. Wait before starting drug therapy and, if possible, get repeated baseline measures of severity to see whether the problem persists. Symptoms wax and wane, often for no identifiable reason, and improvements can be attributed to the medication when in fact they were merely the result of the passage of time.It is not possible to provide good evidence-based guidance on which drug to choose to manage agitation and aggression.
Which to choose may be determined by comorbid symptoms (eg, depression, seizure disorder, or anxiety disorder). A good resource when deciding which drug to use is provided by the Neurobehavioral Guidelines Working Group.7CHECKPOINTS?
In somebody with a severe psychotic illness that develops 3 months after a traumatic brain injury with no loss of consciousness, one can be fairly confident that the illness is not a direct consequence of the effects of the brain injury on delusion formation.? If posttraumatic amnesia (PTA) lasts less than 1 week, a reasonably good outcome is expected; if PTA lasts longer than 1 month, significant disability is likely.? In patients with severe brain injury, a typical clinical picture consists of significant cognitive impairment—particularly in the domains of attention and concentration, psychomotor speed, memory, and executive function—as well as fatigue and problems with motivation. Long-term functional deteriorationAfter a severe injury, most recovery occurs during the first 1 to 2 years. This is possibly a consequence of the patient’s greater awareness of his very disabled state. The optimism of the early phases of recovery begins to be replaced by the realization that the rate of improvement has slowed and full recovery is unlikely. In my experience, it is not easy to help such patients.• Psychosocial factors affect the patient’s recovery. Compared with patients who do well, patients who deteriorate—up to 10 years postinjury—tend to be more anxious and depressed. They have more problems with alcohol (both before and after the injury), have lower self-esteem, and are more likely to have been injured in an assault.10• Dementia may ensue. Patients who have sustained a TBI may be at increased risk for Alzheimer disease or other dementias, or for a lesser degree of decline in cognitive function.11,12Secondary complications of TBI need to be considered in any patient with functional deterioration (Table 1). Which medication to choose to reverse the deterioration may be a matter of trial and error. Antidepressants are a reasonable first-line therapy, particularly if there is evidence of depressive symptoms.
The brain injury may have direct effects on the control of facial expression so that the patient looks depressed, regardless of how he is feeling.
Brain injury can have direct effects on pathways involved in appetite, sleep, pleasure, and reward, resulting in biological symptoms of depression even though the patient may not be depressed. Symptoms of distress and sadness can be understood as a reasonable reaction to a desperate predicament. Other changes in mood control may be seen—most commonly lability of mood, but also alexithymia.The assessment depends on understanding the severity of the distress and the degree to which it is intractable and enduring. Feelings of worthlessness, hopelessness, or guilt suggest that clinical depression may be present.
Suicidal ideation is not uncommon, and rates of suicide after TBI are increased 2- to 3-fold.Psychotic symptomsOnce the patient emerges from coma after a severe brain injury, there may be days or weeks of delirium. When the patient emerges from the delirium, more discrete psychotic symptoms may become apparent. Confabulation, delusional disorientation, and delusional misidentification are characteristic of these early psychotic symptoms.Confabulations are often banal. For example, a patient may describe visits from friends or family when in fact there have been none. However, confabulations may be more bizarre, (eg, a patient recalls a helicopter evacuation from the ward next door at the same time that a gun battle took place on the hospital roof). Delusional disorientation is common and might typically involve descriptions of the ward as being the patient’s place of work. Or the patient may believe that he is on a ship at sea.Delusional disorientation may overlap with reduplicative paramnesia, so the patient may believe that he is in an annex of the hospital but in another part of the country.
Other delusional misidentifications may be seen, particularly the Fregoli syndrome: the patient is convinced, for example, that the old lady in the bed opposite him is his aunt.
Another delusion, also based on attributing familiarity when in fact there is none, is the patient’s belief that he has seen you, the treating doctor, before, whereas in fact you have never met.Remember that confabulatory delusional states are part of a resolving organic mental state.
Antipsychotic drugs may have little effect and, particularly if there are no concerns about patient safety because of delusions, are probably not needed.Psychotic states that develop in the longer term may still be directly attributable to the brain injury.
For example, depersonalization may result in nihilistic delusion, or poor memory may result in delusions of persecution. Morbid jealousy may be seen and is particularly dangerous in somebody who has suffered a brain injury, given the risk of violence and poor impulse control. For example, many patients are given phenytoin in the acute postinjury period, and months or years later are still taking the drug despite never having had a seizure.
There is good evidence that prophylactic anticonvulsants do not work and that phenytoin has adverse effects on cognition.The principles of drug treatment in somebody with a brain injury are outlined in Table 2. Therefore the best advice when treating mental symptoms in a patient with a brain injury is to be symptom-specific and to use the same drug you would for someone without a brain injury. However, the medications tend to be less effective and symptoms are more difficult to treat in brain-injured persons.17,18 Patients who have had a TBI are more vulnerable to adverse effects of medication and are less likely to show evidence of benefit. Furthermore, there may not be an indication for the symptom that the drug is being used for. It is prudent to continue drug treatment of behavioral, cognitive, and psychiatric symptoms after TBI only if there is good evidence that the patient is benefiting.

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