Unfortunately, the official test results that came back from the state lab found that the mixture in Hernandez's truck was actually cheese, shrimp, and tamale and tortilla dough, according to WRAL. Note: If you need help accessing information in different file formats, see Instructions for Downloading Viewers and Players. Vaccines Did Not Save Us – 2 Centuries Of Official Statistics This is the data the drug industry do not want you to see. A detailed Contents listing of this article with each category of disease and related graphs appears after the Introduction.
The main advances in combating disease over 200 years have been better food and clean drinking water. Improved sanitation, less overcrowded and better living conditions also contribute. Measles mortality graphs are enlightening [more below] and contradict the claims of Government health officials that vaccines have saved millions of lives. It is an unscientific claim which the data show is untrue. The success of the City of Leicester, England was remarkable in reducing smallpox mortality substantially compared to the rest of England and other countries by abandoning vaccination between 1882 and 1908 [see more below]. This contrasts how the drug industry has turned each child in the world into a human pin-cushion profit centre. To account for the fall in diphtheria mortality [blue graph line] one must look elsewhere for the cause.
We have compensated cases in which children exhibited an encephalopathy, or general brain disease. These third world children die because we have vaccines. In the 21st Century, despite all the claims made about modern science, we have no effective treatments for common basic childhood diseases.
It has been estimated vaccines prevent 25% of the deaths of these children, so 75% still die. If there were effective treatments we could save their lives.
We have no effective treatments because there is no incentive for the drug industry and every incentive for them not to develop them. The World Health Organisation and our health departments worldwide, in thrall to and under the influence of the drug industry, do nothing about it.
Examples of recent overdiagnoses of measles when there are measles “scares” are proportionately up to 74 times (or 7400% overdiagnosed). Figures and sources follow the next paragraph. What health officials are also doing is relying on very old and unreliable data which ignores that measles has become progressively milder so the risks of long term injury have diminished – (and death is the most extreme form of long term injury – shown here by official data to have diminished rapidly and substantially over the past 100 years without the risks posed to children’s health by vaccines).
To start you with something simple, Scurvy, Typhoid and Scarlet Fever are good examples to use as comparisons with “vaccinatable” diseases.
Medicine and especially drugs and vaccines played no part in the fall in Scurvy death rates and the same can be seen for other diseases. Typhoid and Scarlet Fever vanished without vaccines but with clean water, better nutrition, sanitation and living conditions. By 2007 the chance of anyone in England and Wales dying of measles if no one were vaccinated was less than 1 in 55 million.
Note that what seem large fluctuations after MMR vaccination was introduced in 1988 are not so large and are a feature of plotting the graph on a logarithmic scale. This can be seen in the following graph, plotted on an analog scale. The graph below is from a peer refereed medical paper: Englehandt SF, Halsey NA, Eddins DL, Hinman AR. Correspondingly, when vaccination was introduced, they will tend to follow the fashion of not diagnosing measles, where they believe it controlled by vaccination. It is not exaggeration but accurate to state that mumps vaccination takes the medical profession firmly into the territory of the criminal law and unethical medical treatment of children.
Providing treatment to a patient that is not clinically needed and misleading patients as to the clinical need for a treatment so as to vitiate their consent can mean the administration of the treatment is a criminal offence: Appleton v Garrett (1995) 34 BMLR 23. Doctors and nurses who fail to tell parents mumps vaccine in MMR is clinically unnecessary, of the exact risks of adverse reactions and then give the vaccine appear to be behaving unethically, potentially in contravention of the criminal law and liable to civil proceedings for damages. They are also unable to explain the exact risks because data on adverse reactions are not being collected properly or at all, and there is evidence showing adverse reaction data are suppressed. A consequence is that giving MMR vaccine to children cannot be justified on clinical or ethical grounds.
And one consequence of this unnecessary measure is that we are now putting young male adults at risk of orchitis and sterility because they did not catch natural mumps harmlessly when children and because MMR vaccination is not effective in conferring full or lasting immunity across an entire population. 1 in 4 males who has achieved puberty and has not achieved immunity to mumps runs the risk of orchitis. Orchitis (usually unilateral) has been reported as a complication in 20-30% of clinical mumps cases in postpubertal males. As with mumps, rubella vaccination again takes the medical profession into the territory of the criminal law and unethical treatment of children. Aside from a rash the adverse effects of rubella for children are minimal. Vaccination against rubella is of no clinical benefit to a child particularly when compared to the risks of adverse vaccine reactions. The following is the same USA graph as just above, but with Influenza and Tuberculosis Deaths included. And you can see that Influenza deaths were not prevented by a vaccine – because for most of the period covered, there was no vaccine available at all and when it became available, it was not freely available until the present day – when guess what – ‘flu mortality had already plummeted – and guess what else – it does not work particularly well either – in fact so badly it may well be best avoided. The following is the same graph as above but showing the full curve for influenza and pneumonia mortality.
It was not until 1946-7 – after the substantial fall in diphtheria mortality had taken place that a major effort was made to vaccinate the children who had been missed.
This graph demonstrates that the administration of tetanus vaccine is likely to be pointless and puts children especially at risk of adverse reactions to the vaccines. On any scientific analysis of the history and data, crediting smallpox vaccine for the decline in smallpox appears misplaced. The severity of the disease dimished with improved living standards and was not vanquished by vaccination, as the medical “consensus” view tells us. SMALLPOX FATALITY RATES, cases in vaccinated and re-vaccinated populations compared with “unprotected” Leicester – 1860 to 1908.
Biggs said “In this comparison, I have given the numbers of revaccinated cases, and deaths, and each fatality-rate separately and together, so that they may be compared either way with Leicester. It is certain beyond doubt that diptheria vaccine played no part in thesudden fall in diphtheria mortality from 1941 to 1946 [see graph] . The records show most children went unvaccinated until after the major fall. The Government should create, issue, and circulate all the currency and credits needed to satisfy the spending power of the Government and the buying power of consumers.
Kent Freedom Movement is a grass roots organisation of people who want to bring important information to the people of Kent. Insulin is one of the most important hormones in the human body, and yet most people don’t really understand why our bodies make it or how what we eat affects the levels of insulin we produce. All cells, from bacteria and fungi to us, take glucose and use it to generate ATP by a process called Oxidative Phosphorylation. It’s particularly important for our bodies to maintain glucose levels every time we eat. It turns out insulin in the brain has a lot of functions, very few of which we understand well. While insulin levels are mostly regulated by the amount of glucose in our blood, other things can stimulate its release. Once we start to eat, our bodies ramp up insulin secretion, in what is often called first phase insulin release. When your body stops producing insulin, injections are all that is left - credit, i5a, flickrDiabetes occurs when the body does not have the insulin signaling it should.
Everyone should think about insulin and blood glucose levels, not just people with diabetes or metabolic syndrome. Research into the glycemic index have found strong support of the idea that low GI foods are better for us. The GI or GL of a food isn’t the only thing you should consider when it comes to insulin and your diet. Thinking about how our diet affects insulin is especially key when trying to lose weight or maintain a healthy weight. Understanding how our bodies regulate insulin release also explains why certain foods are worse for us than we’d expect. High spikes in insulin lead to dramatic drops in blood glucose, which can cause your body to feel hungry sooner. My work on this whole project has slowed significantly over the summer as I pursued an interesting opportunity of sorts with the Rodale Institute up in Pennsylvania and as I work towards a masters in nutritional biochemistry. Both opportunities have opened a whole new series of doors that will only serve to strengthen this site going forward. During a traffic stop on May 1, a deputy used a portable drug-testing kit to test a mixture found in Hernandez's truck. Hernandez has been released from jail with a $400 check from the sheriff's office to cover the lost food.
A new generation of muckraking journalists and consumer protection organizations aided in pushing a reluctant Congress to sponsor a bill to replace the old law. Here you will also learn why vaccinations like mumps and rubella for children are medically unethical and can expose medical professionals to liability for criminal proceedings and civil damages for administering them. It was only after the large fall, that in 1946-47 there was a “catch-up” diphtheria vaccination campaign.
All this whilst we watch as childhood prevalence of asthma, allergies, autism, diabetes and more have increased exponentially as the vaccines have been introduced. This needs political commitment from western developed nations and the courage to stand up against vested commercial interests to develop effective treatments to save lives – children’s lives. This following of fashions has been seen in other areas, including Coroner diagnoses of causes of death. And as there is insufficient clinical benefit to children to introduce mass mumps vaccination, it cannot be justified as a general public health measure.
A graph for rubella mortality is not included because death from rubella over the last century was so rare the figures are insufficient to plot a graph of any note.
If a pregnant woman catches rubella infection during the first three months of pregnancy and the child survives, this poses a risk to the unborn child of being born with congenital rubella syndrome (CRS), involving multiple congenital abnormalities. In pro-vaccinist language, may I ask, if the excessive small-pox fatality of Japan, of the British Army, and of the Royal Navy, are not due to vaccination and revaccination, to what are they due?
You probably know that people with type 1 diabetes need to inject themselves with insulin to survive, and must constantly monitor the amount of sugar they eat.
More so than any other hormone, our diet is key in regulating insulin levels, and thus a number of biological processes. In cells, energy is stored and shuttled around using a molecule called Adenosine Tri-Phosphate, or ATP. First, glucose is converted to an intermediate molecule called pyruvate via a process called glycolosis.
Whenever we ingest food, our bodies have to quickly adjust to the sudden flood of glucose entering our systems as our meals are digested. Our bodies release insulin right before and during eating, and that insulin tells our bodies to start taking glucose out of the blood, thus lowering our blood glucose levels.
It’s main job is to signal the liver, muscle and fat tissues to take up glucose from the blood and store it as glycogen. Mice that lack insulin and leptin receptors in their brains, however, exhibit insulin resistance that is characteristic of diabetes.
Other molecules from digestion, like certain amino acids, proteins and lipids, can similarly stimulate insulin release. Insulin that was kept in storage while our blood glucose levels were normal is released all at once, leading to a dramatic increase in insulin levels. If the first pulse was enough, then they slowly take up the insulin they released, and store it for the next meal. As I just explained, insulin in the brain is particularly key, and can lead to neurological disorders. Prevalence rates of type 2 diabetes doubled between 1990 and 2005, causing the CDC to declare it an epidemic.
Metabolic syndrome, often called prediabetes, is poorly characterized, and even more poorly understood. What we eat, how much of it, and when can impact our insulin release, which in turn can have a big impact on our bodies and how we feel.
The glycemic index rates foods based on how much of an immediate impact they have on blood glucose per 50 grams of carbohydrate. People who eat less high GI foods have lower risks of developing both type 2 diabetes and heart disease. Glycemic indices aren’t the whole story since they are based on a per-carbohydrate basis. When different foods were tested for their GI values at different times of the day, for example, researchers found that the same food eaten for lunch instead of breakfast induced a lower glucose response. For example, foods that contain fiber, protein, or fat will generally reduce the GI of the meal as a whole. This is important in keeping healthy and reducing the risk of metabolic syndrome and type 2 diabetes.
Insulin actually triggers the storage of fats in adipose tissues, so sustained high levels of insulin promote weight gain! Sugary drinks are particularly bad for us, for example, even when we take into account their calorie and sugar content.
Effect of alcoholic beverages on postprandial glycemia and insulinemia in lean, young, healthy adults.
Consuming eggs for breakfast influences plasma glucose and ghrelin, while reducing energy intake during the next 24 hours in adult men. However, even I can see that the information in this website is better than anything those institutions have ever attempted at diagnosing, treating or curing any disease. I can attest from personal experience that intermittent fasting and very low carbohydrate consumption (below a total of 50-grams per day) has produced much better health and high energy levels. It changed colors, indicating a match for illegal drugs, which led to Hernandez's arrest. The FDA itself exemplified the state of affairs in the marketplace by assembling a collection of products that illustrated shortcomings in the 1906 law. Doctors substantially overdiagnose measles cases especially when they believe it is a possible diagnosis.Doctors were told the vaccine prevented children getting measles when introduced in the late 1960’s so after that time a substantial reduction in diagnoses would be expected. Poor nutrition, particularly a lack of fresh fruit and vegetables, can result in Scurvy. Mortality rates fell dramatically as living conditions improved.
Whenever the cell then has an energy-requiring reaction, enzymes can use the energy stored in ATP’s phosphate bonds to fuel it. As long as there is oxygen around, this pyruvate is further converted to Acetyl CoA, which enters a cycle of reactions called the Citric Acid Cycle.
Unlike plants, though, we cannot create our own glucose, so we rely on our diets to provide it for us. The amino acids that make up proteins can be converted to glucose via an enzymatic process called gluconeogenesis. It does this by both promoting uptake of glucose by cells and the storage of glucose within our cells. This creates an effective rise in tryptophan concentration in the blood, allowing it to pass through the blood brain barrier.
Strangely, though, they have a lot of reproductive deficiencies, too – the females have poor fertility, high testosterone levels and deformed ovaries, for example.
But most incredibly, our bodies begin releasing insulin before we even take a single bite of food. The amount of insulin secreted in the first phase response to a meal is determined by the amount of glucose encountered in the previous meal – the more you needed last time, the more is released in this first phase. If the blood glucose levels stay high, though, the ?-cells begin producing and releasing insulin in pulses every ten to twenty minutes. People with type 1 diabetes tend to realize their condition early in life, and must deal with it throughout their lifetime, while those with type 2 tend to develop symptoms later on. Insulin is meant to be a fast acting hormone – you release it when glucose levels are high, so that they drop. Symptoms include elevated blood pressure, high blood cholesterol, and increased waist circumference. There are enzymes in our saliva that begin carbohydrate breakdown before the foods even reach our stomachs!
If you picture the rise in blood glucose levels in response to a food on a graph over time, the glycemic index is a number that is related directly the area under a two-hour curve. Two foods that have the same GI can have dramatically different effects on blood glucose per serving if one has significantly higher carbohydrate content than the other.
This is why it may be particularly important to eat a protein-rich breakfast, like eggs, instead of high glycemic foods like white bread toast. But insulin affects so many other things in our bodies, from amino acid uptake to fat storage.
Furthermore, recall that our bodies don’t break down fat while insulin is circulating.
Low GI foods don’t cause dramatic drops in glucose levels, thus you tend eat less throughout the day. Very well written, comprehensible even by a person not from the medical background (like myself).
Unlike doctors who don’t explain bullshit to you and just tell you to do something because and expect you to pay huge amounts of money for just ordering you to do something without telling you the reason. Everything is detailed with references to original sources and is not simplified to the point where it becomes a fallacy (simplifications when wrongly used can, and most of the time, become fallacies). I bet the writer is a person of great experience or of sound practical intelligence coupled with a strong sense of empathy for the reader. And contrary to feeling like crap, I feel better than ever before with much more clarity of thought and high energy levels. This takes the carbon to carbon bonds and uses them to create high energy electrons, which are then passed down a chain of enzymes which use the electron’s energy to create a proton gradient, the force of which fuels ATP synthase, the enzyme which creates ATP from ADP. Fats, too, are converted to glucose or its derivatives; glycerol, for example, can be converted to glucose via gluconeogenesis, and fatty acids can be converted to Acetyl CoA via beta oxidation. You would think that since it’s so important, we would want a ton of glucose in our blood, but too much causes our blood to thicken, slowing it down and drawing fluid from our tissues to try and make it thin again. Without insulin, we would all go into hyperglycemic shock and die from something as common as a hamburger. If it drops too low, an antagonistic hormone, called glucagon, is released which does the opposite of insulin, stimulating cells to break down glycogen and release glucose. Instead, they respond to insulin by taking the fats that enter the blood stream and turning them into fatty acids, which they store in large vacuoles.
In the brain, tryptophan is converted to serotonin, a neurotransmitter whose primary purpose, in this case, is to reduce appetite. Why insulin has these effects on reproduction is unknown, but it just goes to show that insulin does a lot more than regulate blood sugar levels, and is far more important in our bodies than we once thought. When we think about, smell, or slightly taste foods, our brains trigger what is called Cephalic Phase Insulin Release.
In a healthy person, this first phase response peaks a few minutes after you’ve started your a meal. There is a special name for the series of diseases caused by impaired insulin signaling in the body. The main cause appears to be decreased response to insulin in certain tissues, specifically muscle and fat.
But more importantly, carbohydrates lead to immediate rises in blood glucose because they contain glucose. The higher the spike in blood glucose levels, the larger the area under the curve is, and thus the higher the glycemic index, which is somewhere on a scale of 1 (low) to 100 (high).
The affect a serving of food has on blood glucose is referred instead to its Glycemic Load. Morning is a special time for your body because you’ve just spent a while in a comatose state. Time of day has been found to have a larger effect on insulin responses in women than in men, though no one understands why.
Furthermore, many cultures eat high GI foods like potatoes or rices but have low occurrences of diabetes and obesity.
This means that if we eat foods with high GIs that produce sustained insulin levels, we’re shooting ourselves in the foot, even if we eat less calories overall.
Thousands of years of evolution led our brains to believe that drinks, overall, were low-cal things that mostly contain water. It’s thought that this effect, on top of the high-sensitivity of our bodies to high GI foods in the AM, is why eating eggs, a low GI food, instead of cereal or toast in the morning has been found to reduce overall food intake for the day by as much as 18%. For example, dieters often experience depression around 2 weeks after they begin cutting high-glycemic foods like carbohydrates out of their diet. Also, too many analogies (which when wrongly used too may become falasies) overloads the reader’s perception with useless information which leads the reader to a wrong understanding of concepts. No matter where it comes from, the glucose from our meals then ends up in our blood to travel around our bodies to the tissues that need it.
Too high of a blood glucose level, called hyperglycemia, can result in blurred vision, fatigue, dry mouth and heart problems that can sometimes be fatal. But serotonin has a lot of other effects, as those of you who have read the previous Understanding Our Bodies on Serotonin know well. A food’s color, appearance, flavor, aroma, and texture can all impact how our brains respond to the idea of eating it. The blood sugar rise caused by the meal peaks about half an hour after eating, and this, in turn, leads to a decrease in insulin production and release. If we see a tiger, for example, our stress hormones spike so we can be prepared to fight if it attacks or run like hell to get away. Meanwhile, reduced levels of insulin and its related proteins are linked to Alzheimer’s disease and other degenerative disorders. While both conditions involve problems with the insulin pathway, type 1 diabetes is caused by a lack of insulin, while type 2 is caused by chronically high levels. If you constantly eat too much or have a very sugary diet, you can end up with high insulin levels all the time. Other molecules must first be converted to glucose, but carbohydrates, which include sugars, just need to be hacked into pieces by our digestive enzymes. Mainly, the faster it’s churning, the more likely the machinery is to leak reactive oxygen species (ROS). Glycemic load is calculated by multiplying the weight (in grams) of carboydrate in a serving by the food’s overall GI and divided by 100.
The changes your body undergoes while you sleep can have a dramatic impact on how it responds to food.
Furthermore, studies have shown that the quality of sleep you get affects how strongly your body reacts to food. The truth is we have yet to tease out all the factors that lead to these conditions, and the GI level of our diets is likely only one of many related factors.
You can create lower spikes in insulin not only by avoiding sugary drinks and eating lower GL foods but also by eating smaller meals.
A Tennessee drug company marketed a form of the new sulfa wonder drug that would appeal to pediatric patients, Elixir Sulfanilamide. This means that if all the sugar from a single roll of those delicious candies were to enter my bloodstream at once and remain unchecked, it would raise by blood sugar by 8 mM! In general, increased serotonin leads to a feeling of happiness and calm, which is why we get such satisfaction when we eat. The goal is to prepare the body for what the brain thinks will be a sudden flood of glucose. Either way, we’ll need extra energy on hand to deal with the stressful situation, so stress hormones stop insulin from being released to ensure that a little extra glucose is in the bloodstream and able to reach whatever body parts need it most. Often, people with metabolic syndrome are overweight, and at higher risk for other, even more life threatening conditions like heart disease.
Different carbohydrates contain different amounts of the monosaccharides, like glucose or fructose.
There is another index, called the Insulin Index, that looks directly at rises in insulin levels. These oxygen radicals are highly reactive and tend to transform whatever they come in contact with, which can cause damage to proteins, membranes, or even our DNA. Insulin levels tend to be low in the early morning, for example, because your body releases stress hormones just before you wake up. A restless night can lead to higher glucose responses and larger spikes in insulin in response to food in the morning. Thus when we look at a soda or even begin to sip one, we don’t have the same level of cephalic phase insulin release or first phase insulin release that we would for a solid treat. This is because the amount of first phase insulin release is dependent on the amount of insulin needed for the previous meal. However, the solvent in this untested product was a highly toxic chemical analogue of antifreeze; over 100 people died, many of whom were children. This is about enough glucose to provide energy to the body for 20-30 minutes, so as we use up the glucose in our blood, our bodies constantly release more (hopefully) without overdoing it. Thus insulin is important not just when it comes to dealing with fats and sugars but in regulating our emotions, too! The sweeter and sugarier the brain thinks the meal will be, the more insulin it stimulates the pancreas to release before the food even enters the mouth.
Without the ability to produce enough of this vital hormone, they usually have consistently high blood glucose levels.
While some drugs can be prescribed to treat the symptoms like high blood pressure, the only long-term solution is to lower chronic blood glucose levels and restore insulin sensitivity, if, indeed, it can be restored at that point. While the glucose and insulin scores of most foods are related, high-protein foods and baked goods that are rich in fat and refined carbohydrates usually elicit much higher insulin responses than their glycemic index values would suggest.
By eating foods that increase glucose levels more slowly, we limit ROS bursts that can damage our cells.
Once you’re awake, though, your body ramps up insulin secretion to metabolize the high glucose levels and give your cells a little fuel to start the day with. So getting a good night’s sleep is also important in preventing the kinds of spikes which may be a major factor in type 2 diabetes. The end result of this is that our bodies are unprepared for the sudden sugar rush, and have to instead release a massive amount of insulin all at once to deal with what it considers an inexplicable rise in blood glucose.
The bigger meals are, the larger the spike at the beginning of every meal, and the bigger the drop in glucose afterward. Decreased insulin means decreased amino acid uptake, and because the level of other amino acids affects how well tryptophan crosses the blood brain barrier, decreased insulin means less serotonin which leads to, in layman’s terms, feeling like crap. The public outcry not only reshaped the drug provisions of the new law to prevent such an event from happening again, it propelled the bill itself through Congress. If you only get a little static every once in a while, you can hear the song fine, and understand what the artist is saying.
While you should monitor the GI of your meals to reduce insulin spikes, you shouldn’t go for rock bottom either. By carefully controlling their sugar intake and taking insulin when they need it, people with type 1 diabetes can regulate their blood glucose levels to being almost normal. But start having high static all the time and you can’t tell what song is playing anymore. But there’s a lot less carbohydrates per serving in the whole gain loaf, and thus its GL is only 9 while the GL of the french baguette is 27!
Moreover, it mandated pre-market approval of all new drugs, such that a manufacturer would have to prove to FDA that a drug were safe before it could be sold. It irrefutably prohibited false therapeutic claims for drugs, although a separate law granted the Federal Trade Commission jurisdiction over drug advertising.
Type 2 diabetes is that much more dangerous because the body will rarely respond to insulin treatment, meaning that drastic diet changes and exercise are the only ways to fight back.
The act also corrected abuses in food packaging and quality, and it mandated legally enforceable food standards. I will gladly encase my right foot in a natural rubber boot, and my left foot in a latex free piece of footwear (preferably something with a steel toecap). Blindfolded, I then propose to kick a series of anti-vaxxers in the booty, with one of my randomly selected booties. I also want to add that the use of modern medical technology will help further improve your health.
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