Pregnancy induced hypertension quizlet,when are you 8 weeks pregnant quiz,can i get pregnant at 46 years old jokes,fertility cycles and nutrition pdf book - New On 2016

Hypertension-or-High-Blood-PressureWhen arteries become narrow due to plaque deposition, the blood pressure increases against vascular walls.
Blood-CholesterolCholesterol Deposit on the Walls of the blood vessels, leads to the formation of plaque, that narrows the blood vessels resulting into less or no blood flow.
Renovascular-HypertensionWhen kidney problems combine with high blood pressure, it leads to renovascular hypertension.
Orthostatic HypertensionWithin three minutes of standing there is a sudden fall of systolic and diastolic pressure. Hypertension and ObesityObese people have 60% more chance of getting affected with hypertension. Pregnancy-Induced-Hypertension-This condition primarily strikes pregnant women in the 20th week of gestation.
Hypertensive-Retinopathy-Prolonged and untreated Hypertension may lead to Hypertensive Retinopathy. Lower Blood Pressure Naturallyreduce stress, exercise regularly, maintain a healthy lifestyle, follow dash diet.
Pregnancy induced hypertension is considered as a critical condition that usually occur to pregnant women. The mother will reveal a medical history of high blood pressure which is about (140 mmHg or more for systolic pressure and 30 mmHg or more for diastolic pressure).
Further laboratory and diagnostic evaluations may indicate a pregnant women with pregnancy induced hypertension.
If the pregnant mother’s blood pressure remains high and has not reduced despite of the treatment given, they will receive magnesium sulfate to enhance diuretic effect.
Pregnant woman who is diagnosed to have pregnancy induced hypertension are required for strict measuring of blood pressure. As a conclusion, pregnancy induced hypertension should be relieved with immediate medical response. In the earliest stage of hypertension (MAP 90 mm Hg in midpregnancy) , termed pre-eclampsia, in the absence of proteinuria is considered transient hypertension.
Clinical correlates usually include rapid weight gain from sodium and water retention, a rise in systolic and diastolic pressure, followed by proteinuria.
PIH may be triggered by a fall in vascular PGI2 (prostacyclin) synthesis and an increase in thromboxane production increasing platelet aggregation. In March 2001, the National Institutes of Health issued the following warning: "The number of Web sites offering health-related resources grows every day.
In addition to offering a structured and comprehensive bibliography, this medical reference on pregnancy-induced hypertension will quickly direct you to resources and reliable information on the Internet, from the essentials to the most advanced areas of research. Micrograph showing hypertrophic decidual vasculopathy, the histomorphologic correlate of gestational hypertension.
Gestational hypertension or pregnancy-induced hypertension (PIH) is defined as the development of new arterial hypertension in a pregnant woman after 20 weeks gestation without the presence of protein in the urine. This is when tonic-clonic seizures appear in a pregnant woman with high blood pressure and proteinuria.

This is a dangerous combination of three medical conditions: hemolytic anemia, elevated liver enzymes and low platelet count.
There is no specific treatment, but is monitored closely to rapidly identify pre-eclampsia and its life-threatening complications (HELLP syndrome and eclampsia).
Drug treatment options are limited, as many antihypertensives may negatively affect the fetus.
The fetus is at increased risk for a variety of life-threatening conditions, including pulmonary hypoplasia (immature lungs).
Burger D, Schock S, Thompson CS, Montezano AC, Hakim AM, Touyz RM.SourceKidney Research Centre, Ottawa Hospital Research Institute, University of Ottawa, Ottawa, Canada.
Choi S, Kim JA, Na HY, Kim JE, Park S, Han KH, Kim YJ, Suh SH.SourceDepartment of Physiology Medical School, Ewha Womans University, Seoul 158-710, Republic of Korea. Endothelial dysfunction is associated with K3.1 dysfunction and contributes to the development of hypertension in preeclampsia. MicroRNA-376c Impairs Transforming Growth Factor-? and Nodal Signaling to Promote Trophoblast Cell Proliferation and Invasion.
Fu G, Ye G, Nadeem L, Ji L, Manchanda T, Wang Y, Zhao Y, Qiao J, Wang YL, Lye S, Yang BB, Peng C.SourceDepartment of Biology, York University, 4700 Keele St, Toronto, Ontario, Canada. Preeclampsia is a major disorder of pregnancy and a leading cause of maternal and perinatal morbidity and mortality.
When this pressure goes beyond normal blood pressure, it is called high blood pressure or hypertension. Copying the lifestyle of hypertensive parents keep you at risk, but if a person practices healthier lifestyle, this factor will not take effect at all. Most of the time, they gain at least 3 or more pounds (1.4 kilograms) in a week until the 3rd trimester. These include edematous extremities (lower and upper legs, including the face), hyperreflexia (hyperactivity response to stimulus) for deep tendon reflexes. Additionally, it can reduce the blood pressure and prevent the occurrence of seizure attacks. These include the assessment of pulse rate, respiratory rate, fetal heart rate (condition of the fetus), level of consciousness and the reflexes of her deep tendon. Their only aim is to share their opinion on the concerned topic, and help the reader understand it better. When the condition occurs with proteinuria it is considered progressive toward eclampsia which is defined as hypertension with convulsions, vasospasms, and CNS hyperexcitability. Cerebral hemorrhage, acute cardiac failure with pulmonary edema and hepatic failure may lead to maternal death.
The changing ratio in production of these prostaglandins results in placental atherosis, decreased uteroplacental blood flow, leading to fetal distress. Many sites provide valuable information, while others may have information that is unreliable or misleading." Furthermore, because of the rapid increase in Internet-based information, many hours can be wasted searching, selecting, and printing. It gives a complete medical dictionary covering hundreds of terms and expressions relating to pregnancy-induced hypertension.

First, you will not waste time searching the Internet while missing a lot of relevant information. Methyldopa, hydralazine, and labetalol are most commonly used for severe pregnancy hypertension. If the dangerous complications appear after the fetus has reached a point of viability, even though still immature, then an early delivery may be warranted to save the lives of both mother and baby.
However, evidence of endothelial K3.1 dysfunction in the vascular system from women with preeclampsia is still lacking.
The pregnant mother will receive IV medication (magnesium sulfate) to prevent the occurrence of seizure.
Good or bad, Right or wrong is solely readers decision and should be taken under the guidance of a medical expert. Once established progressive PIH continues in a vicious cycle interrupted completely only after delivery of placenta.
Increased angiotensin II sensitivity (due to fall in PGI2) results in increased blood pressure with resulting edema and decreased plasma volume as fluid shift from the vascular space to the intercellular space.
Various abstracts are reproduced to give you some of the latest official information available to date. An appropriate plan for labor and delivery includes selection of a hospital with provisions for advanced life support of newborn babies. Currently, this condition can cause serious complications to both mother and her developing fetus.
Pregnant women may complain of oliguria (urine output of at least 400 ml in a day or lesser), blurring of vision (visual problem), pain at the epigastrium area, heartburn, and emotional anxiety that may lead to easy irritability.
Health care professionals such as nurse must explain this medication to pregnant women, to alleviate anxiety level.
This book was created for medical professionals, students, and members of the general public who want to conduct medical research using the most advanced tools available and spending the least amount of time doing so.
Finally, it provides information to users on how to update their knowledge using various Internet resources. Abundant guidance is given on how to obtain free-of-charge primary research results via the Internet.
The book is designed for physicians, medical students preparing for Board examinations, medical researchers, and patients who want to become familiar with research dedicated to pregnancy-induced hypertension.
For readers unfamiliar with the Internet, detailed instructions are offered on how to access electronic resources.
For readers without access to Internet resources, a directory of medical libraries, that have or can locate references cited here, is given. We hope these resources will prove useful to the widest possible audience seeking information on pregnancy-induced hypertension.

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