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The Web site for Thomas Jefferson University, its contents and programs, is provided for informational and educational purposes only and is not intended as medical advice nor, is it intended to create any physician-patient relationship. Diffuse interstitial lung disease refers to a group of lung disorders in which the deep lung tissues become inflamed. Causes The lungs contain tiny air sacs (alveoli), which is where oxygen is absorbed. Pulmonary edema is a type of myocardial damage that may cause excessive sweating and heart palpitations. An excessive amount of macrophages and low-density lipoproteins accumulate within arterial walls, forming plaque.
Atheriosclerosis is a chronic inflammatory disease that compromises arterial walls, the main function of which are to transport oxygen from the heart to the rest of the body via the circulatory system.
The mechanism behind atheriosclerosis being considered the most common myocardial infarction pathophysiology is actually two-fold. Although atheriosclerosis is the most common myocardial infarction pathophysiology, its development does not always trigger a sudden heart attack.
The likelihood of any of the above-mentioned conditions occurring depends on the location in which myocardial infarction pathophysiology takes place, as well as the size and nature of occlusions involved.
The primary symptom of myocardial infarction is chest pain, most often described as a feeling of heavy pressure or tightening of the chest.


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These air sacs open up or expand with each breath. The tissue around these air sacs is called the interstitium. The term specifically refers to an infarction of the myocardium, which simply translates to damage of the inner muscular wall of the heart.
Healthy arteries possess strong and elastic walls in which specialized white blood cells called macrophages remove excess fats and cholesterol. Either the condition is due to a complete occlusion, or blockage, of one or more arteries leading to the heart, or caused by pieces of arterial plaque rupturing and migrating away from the accumulation site and causing blot clots to develop elsewhere. For instance, if the arteries of the left side of the heart are affected, the patient is at risk for pulmonary congestion and edema. The views or opinions expressed in the resources provided do not necessarily reflect those of Thomas Jefferson University Hospitals, Thomas Jefferson University or their staffs. In people with interstitial lung disease, this tissue becomes stiff or scarred, and the air sacs are not able to expand as much. However, if an excessive amount of macrophages and low-density lipoproteins accumulate within arterial walls, a plaque forms by a process known as furring or hardening of the arteries.


The end and permanent result is the same in either event: The cells of the myocardial tissue die, leaving collagen scars behind. Secondary conditions related to myocardial dysfunction include pulmonary congestion and edema, dyspnea, tachycardia, and arrhythmias. This type of myocardial damage is also associated with excessive sweating, nausea, heart palpitations, and dyspnea, or shortness of breath. Should this function become impaired, ischemia will occur, a condition characterized by a lack of sufficient blood and oxygen supply to the heart. Experiencing any of these symptoms warrants immediate medical attention, particularly if there is a history of high blood pressure, elevated cholesterol, smoking, or diabetes. While there are numerous risk factors that can contribute to a diseased heart, the most common myocardial infarction pathophysiology is atheriosclerosis.
A respiratory therapist will help you set up oxygen for use in the home or outside of the home.



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