A toxin synthesized from the venom of a spider may offer an alternative to today's erectile dysfunction drugs, a new study suggests. The toxin, unpoetically named PnTx2-6, comes from the bite of the Brazilian wandering spider (Phoneutria nigriventer). The toxin has been shown to improve erections in rats with hypertension and diabetes; now, researchers have tested it in aging mice and found that the toxin is effective in reversing age-related ED as well.
Peyronie’s Disease is a rare form of penile condition, which causes progressive penile deformity and leads to erectile dysfunction.
Although the above indicated symptoms could, by themselves, determine the presence of the disease, there are also other tests, which could further verify their presence.
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To get an erection, a man's body must release nitric oxide, which relaxes the smooth muscle around the arteries of the penis, allowing for his blood vessels to dilate. Instead of affecting PDE5, the compound seems to trigger nitric oxide release, acting directly to relax the smooth muscles.
23 in the Journal of Sexual Medicine, Nunes and her colleagues injected aging and young rats with the toxin extracted from the spider venom. The next step, Nunes said, is to make sure that the compound doesn't have any nasty effects beyond its intended purpose.
She covers the world of human and animal behavior, as well as paleontology and other science topics.
This is due to the growth and accumulation of fibrous plaques in the tunica albuginea surrounding the corpora cavernosa following a trauma or an injury inside the soft tissues of the penis which caused an internal localized bleeding.


The primary symptom of Peyronie’s Disease is the formation of scar tissues referred herein as fibrous plaques.
During erection, this hardened area of the penis (or the location of the fibrous plaques) shortens, thereby causing the penis to bend. The disease could either cause flaccidity of the tip of the penis (or the end), or it could make achieving erection difficult. The best way is to consult your doctor as soon as possible in order to decide for the best possible course of treatment before everything turns awry. What's more, male victims may find themselves with priapism, or unrelenting and painful erection.
The nitric oxide is a first step in a series of chemical reactions that allow this muscle relaxation to take place. Because about 30 percent of patients don't respond to PDE5 inhibitors, the toxin could provide an alternative to ED treatments currently on the market, Nunes said.
They found that the toxin reversed age-related erectile dysfunction, offering hope that the toxin could eventually move out of animal testing and into human use.
Stephanie has a Bachelor of Arts in psychology from the University of South Carolina and a graduate certificate in science communication from the University of California, Santa Cruz.
Overtime, these fibrous plaques can either prevent someone from achieving erection or can cause painful erection. The direction at which the penis is bent is determined by the location of the hardened area. There he was involved in research in radiation biology and received the Huisking Scholarship.


It was this symptom, turning up in emergency rooms after spider bites in Brazil, that first alerted researchers to the potential of PnTx2-6 as an erectile dysfunction (ED) drug.
One step in the series is cGMP, a signaling molecule that acts to keep the muscles relaxed. It is therefore best to diagnose the presence of the disease and seek treatment as early as possible.
Normally, this hard area is found at the upper side but could also occur at the bottom or side. Stöppler's educational background includes a BA with Highest Distinction from the University of Virginia and an MD from the University of North Carolina. If it is at the bottom or at the side, it bends towards the bottom or towards the particular side at which the plaque is located, consequently. Louis University School of Medicine, he completed his Internal Medicine residency and Rheumatology fellowship at the University of California, Irvine. She completed residency training in Anatomic Pathology at Georgetown University followed by subspecialty fellowship training in molecular diagnostics and experimental pathology.



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