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Treatment of bed bug bites involves two factors namely identifying the insect and treating the infection. Staying in healthy atmosphere and following healthy lifestyle can prevent you from getting infested from bed bug bites. The mitral valve is made up of the annulus, anterior and posterior leaflets, and chordae, which attach the leaflets to their respective papillary muscles.
This chapter reviews three types of mitral valve disease: mitral stenosis (MS), mitral regurgitation (MR), and mitral valve prolapse (MVP).
MS refers to narrowing of the mitral valve orifice, resulting in impairment of filling of the left ventricle in diastole. Patients identified as having mild valve doming during diastole are considered at risk of MS (Stage A).
Although the incidence of rheumatic heart disease has steeply declined during the past 4 decades in the United States, it is still a major cause of cardiovascular disease in developing countries. Patients with MS may present with exertional dyspnea, fatigue, atrial arrhythmias, embolic events, angina-like chest pain, hemoptysis, or even right-sided HF.
The characteristic findings of MS on auscultation are an opening snap, a mid-diastolic rumble and an accentuated first heart sound. Echocardiography also allows assessment of pulmonary artery pressures, detection of other valve disease, visualization of left atrial thrombus, and identification of important differential diagnoses, such as left atrial myxoma. Transthoracic echocardiography is necessary to diagnose and determine the severity of mitral stenosis. Transesophageal echocardiography is indicated in patients before percutaneous mitral balloon valvotomy. Stress echocardiography and cardiac catheterization might be helpful in those cases in which there is a discrepancy between the severity of symptoms and baseline echocardiographic findings.
Medical therapy has no role in altering the natural history or delaying the need for surgery in patients with MS. Tachycardia is typically poorly tolerated in patients with MS and can lead to an acute deterioration as diastolic filling time may be inadequate. Three invasive options are available for patients with MS: PMBC, surgical mitral commissurotomy, and mitral valve replacement (MVR). PMBC is a catheter-based technique in which a balloon is inflated across the stenotic valve to split the fused commissures and increase the valve area.
Surgical mitral commissurotomy was first performed in 1925 as a closed technique (which does not necessitate the use of full cardiopulmonary bypass and is performed through an incision in the left atrial appendage) and is still widely used in many developing countries.
PMBC and surgical mitral commissurotomy are palliative procedures and, in most cases, further intervention is eventually required, usually in the form of a MVR. Medical therapy in patients with mitral stenosis includes diuretic therapy, heart rate or rhythm control, anticoagulation to prevent thromboembolism, and antibiotic prophylaxis against recurrent rheumatic carditis. Antibiotic therapy of group A streptococcal tonsillo-pharyngitis, even delayed 9 days after the onset of symptoms, can prevent rheumatic fever and rheumatic carditis.10 Antibiotic therapy also reduces transmission to contacts.
Patients with MS should at a minimum be followed-up with yearly history and physical examinations.
During pregnancy, women with MS should receive appropriate medical therapy, including beta-blockers (Class IIa) and in certain cases, diuretics (Class IIb), but never angiotensin-converting enzyme inhibitors or angiotensin II receptor blockers (Class III), due to their teratogenic potential. MR is leakage of blood from the left ventricle backwards into the left atrium during systole. Patients identified as having mild abnormalities of mitral valve structure or function (mild prolapse, thickening or leaflet restriction) are considered at risk of MR (Stage A).
Patients with coronary disease or cardiomyopathy with normal mitral valve anatomy are considered at risk of MR (Stage A).
Significant mitral valve regurgitation occurs in about 2% of the population with a similar prevalence in males and females.11 Myxomatous disease is the most common cause of primary MR in the United States (Figure 4).
Significant MR leads to volume overload of the left ventricle, because it has to accommodate both the stroke volume and regurgitant volume with each heartbeat. Patients with chronic, severe MR may remain asymptomatic for years because the regurgitant volume load is well tolerated as a result of compensatory ventricular and atrial dilation. The characteristic finding in a patient with MR is a blowing holosystolic murmur heard best at the cardiac apex. Determining the severity of mitral regurgitation requires knowledge regarding the underlying etiology of the mitral regurgitation and an integrated assessment of several echocardiographic parameters. Additional noninvasive and invasive testing is useful as part of a complete preoperative assessment of patients with severe mitral regurgitation. In patients with acute severe MR, afterload reduction with intravenous nitroprusside and nitroglycerin reduces the regurgitant fraction and pulmonary pressures.
In patients with chronic asymptomatic MR caused by primary valve disease, there is no evidence for the routine use of vasodilator therapy given normotension and normal systolic LV function.13 The management of these patients is focused on deciding the appropriate timing of surgery, before the development of irreversible LV dysfunction as discussed below.
In patients with ischemic heart disease or dilated cardiomyopathy, MR portends a poor prognosis.14 MR in these patients is called functional or secondary MR and is caused by global or regional changes in LV geometry as well as annular dilation.
In patients with primary mitral regurgitation, surgery is indicated in the presence of symptoms or, in asymptomatic patients, if there is evidence of secondary left ventricular dysfunction. Mitral valve repair is the procedure of choice for the surgical management of mitral regurgitation and is associated with lower mortality and better preservation of left ventricular function. Patients with established MR should at a minimum be followed-up with yearly history and physical examination.
Patients with MR generally tolerate pregnancy better than patients with MS do, because the decrease in after-load means that increased cardiac output does not necessarily cause a rise in ventricular filling pressures or pulmonary pressures. MVP is the systolic billowing of one or both mitral leaflets into the left atrium during systole.16 It may occur in the setting of myxomatous valve disease or in persons with normal mitral valve leaflets.
MVP is the most common valvular disorder in the United States, occurring in 2% to 3% of the general population. Many patients with MVP have normal mitral leaflets, with little or no MR, and a benign prognosis. The causes of myxomatous mitral valve disease are not certain, but appear to involve dysregulation of extracellular matrix proteins. Mitral valve prolapse is present if there is more than 2 mm displacement of the mitral valve leaflets into the left atrium during systole in a parasternal long-axis or apical three-chamber view on echocardiography.
Asymptomatic patients require no specific treatment and they should be reassured of their excellent prognosis.
In MVP patients with severe MR, the indications for mitral valve surgery are similar to those for patients with other primary causes of severe regurgitation. Mitral valve prolapse is a benign condition in most cases with similar surgical indications as those for patients with other causes of primary mitral regurgitation. Conoscere l’agenda dei nove mesi, ovvero tutto quello che succede al vostro corpo e al vostro bambino quando siete incinta, e davvero fondamentale, soprattutto per le donne che sono alle prese con la loro prima gravidanza.
Un tuffo al cuore: che tu l’abbia cercato o no, il test di gravidanza dice che sei incinta! Nel quarto mese il ginecologo prescrivera altri esami specifici come gli esami del sangue (con emocromocitometrico completo con conteggio delle piastrine), ricerca degli anticorpi anti-toxoplasma, ecografia e flussimetria doppler (ecografia sulle arterie uterine della donna). Riconoscera anche i rumori forti ai quali reagira coprendosi le orecchie e potrebbe anche cominciare a succhiarsi il pollice. Nel secondo trimestre di gestazione e importante pianificare l’ecografia morfologica, importantissima per approfondire tutti i controlli sul feto.
Ormai, se i genitori decidono di saperlo prima della nascita, potrete conoscere il sesso del bambino.
Ormai ci siamo quasi, mancano solo tre mesi e finalmente potrete conoscere il vostro piccolo! Modern Hand Reading Forum - Discover the language of your hands: palm reading & palmistry forum!Modern Hand Reading eg.
Il complesso articolare di Lisfranc  viene riferito all'articolazione tra I e II osso metatatarsale con l'osso cuneiforme mediale (I) ed intermedio (II)  (vedi schema anatomico sottostante). Visione dorsale dell'anatomia scheletrica del mesopiede: nel cerchio viene rappresentata   il complesso articolare di  Lisfranc. Il complesso legamentoso di  Lisfranc  e una  larga banda del tessuto collageno plantare che si espande ad unire l'articolazione tra  cuneiforme mediale e la base del secondo osso metatarsale; mentre il legamento trasverso unisce la porzione laterale della base  tra II e V osso metatarsale,  non esiste il legamento trasverso tra I e II osso metatarsale. Per la sua limitata mobilita, l'articolazione di Lisfranc  assicura uno stabile asse di rotazione  del piede, ed svolge un ruolo importante per la flessione e dorsiflessione del piede. FIGURA - Dislocazione dorsale della base del II metatarso (freccia piccola) quando il piede e sottoposto alla massima flessione plantare nel contempo ad una spinta assiale (freccia grande). When Lisfranc joint complex injury is suspected, palpation of the foot should begin distally and continue proximally to each tarsometatarsal articulation.

The initial radiographs of a suspected Lisfranc joint injury should include weight-bearing anteroposterior and lateral views, as well as a 30-degree oblique view.1,4,9,12 A weight-bearing radiograph is necessary, because a non–weight-bearing view may not reveal the injury.
The lateral radiographic view of the foot may show a diagnostic “step-off,” which means that the dorsal surface of the proximal second metatarsal is higher than the dorsal surface of the middle cuneiform(Figure 1). Computed tomographic (CT) scanning or bone scanning is helpful in diagnosing difficult cases of Lisfranc joint injury. Some investigators13 have suggested that radiographic displacement or flattening of the longitudinal arch of the foot is associated with a poor prognosis. Early diagnosis of a Lisfranc joint injury is imperative for proper management and the prevention of a poor functional outcome.3 With a general knowledge of both conservative and operative treatment options, the primary care physician can decide whether to treat the injury nonoperatively or refer the patient to an orthopedist.
If the clinical evaluation indicates the probability of a mild or moderate sprain and the radiograph shows no diastasis, immobilization is suggested. After the period of immobilization, ambulation and rehabilitation exercises should be progressive. According to some investigators,3,6 a displacement of more than 2 mm requires open reduction and internal fixation to avoid a poor outcome,3,6 especially in athletes.4 Others13 report no correlation between the degree of diastasis and the eventual functional outcome. If surgical repair is warranted, it should be done within the first 12 to 24 hours after the injury.
While some orthopedists3,4,6 prefer closed fixation with percutaneous K-wires (Kirshner wires), others5,11 report that this method does not hold anatomic reduction and fixation.
A normally functioning valve allows blood to flow unimpeded from the left atrium to the left ventricle during diastole and prevents regurgitation during systole. Those with more advanced (progressive) rheumatic valve changes such as commissural fusion and at least moderate diastolic doming of the mitral valve leaflets are defined as Stage B. Single or recurrent bouts of rheumatic carditis cause progressive thickening, scarring, and calcification of the mitral leaflets and chordae (Figure 1).
Previously asymptomatic or stable patients may decompensate acutely during exercise, emotional stress, pregnancy, infection, or with uncontrolled atrial fibrillation. The first heart sound may be diminished in intensity if the valve is heavily calcified, with limited mobility.
On chest radiography, characteristic findings of MS are enlargement of the left atrium without cardiomegaly, enlargement of the main pulmonary arteries, and pulmonary congestion (Figure 1). Medical treatment is directed toward alleviating pulmonary congestion with diuretics, treating atrial fibrillation, and anti-coagulating patients who are at increased risk of arterial embolic events.
Hemodynamic as well as clinical improvements may be seen immediately and the results are typically comparable with those achieved with open mitral commissurotomy, although less invasive and less costly.7,8 Mitral valve morphology is an important predictor of successful PMBC. Open surgical mitral commissurotomy involves the use of cardiopulmonary bypass and the surgical repair of a diseased mitral valve by direct visualization. In addition, MVR may be necessary as a first line procedure in patients with either heavily calcified valves, or significant MR. Routine screening or treatment of asymptomatic contacts of persons with group A streptococcal tonsillo-pharyngitis is not recommended. These features will tend to increase the transmitral gradient (sometimes to double that of baseline), leading to increased left atrial pressures and elevated pulmonary pressures, which can result in pulmonary edema.
It is caused by various mechanisms related to structural or functional abnormalities of the mitral apparatus (primary) or the left ventricle (secondary) (Figure 3).
Those with more severe valvular abnormalities (severe prolapse, rheumatic changes with loss of central coaptation, or prior infective endocarditis) are considered as having progressive MR (Stage B).
Those with regional wall motion abnormalities with mild mitral leaflet tethering or annular dilation with mild loss of central coaptation of the mitral leaflets are considered as having progressive MR (Stage B).
The rationale for this was largely based upon the clear adverse prognostic impact of even lesser degrees of secondary (vs.
When symptoms do develop, the most common are dyspnea, fatigue, orthopnea, paroxysmal nocturnal dyspnea, and palpitations caused by atrial fibrillation. When ventricular enlargement is present, the apical impulse may be diffuse and laterally displaced, and a third heart sound may be heard. Repeat echo should be considered every 3 to 5 years for those with mild MR and every 1 to 2 years for those with moderate MR.
However, patients with severe pre-existing regurgitation who are already symptom-limited, have a reduced LVEF or pulmonary hypertension may develop HF symptoms because of the volume load of pregnancy. Survival rates among affected patients are similar to those of age- and gender-matched individuals without MVP.18 In other patients, MVP is caused by myxomatous valve disease, with typical findings of elongated and thickened leaflets, interchordal hooding, and chordal elongation (Figure 6). Myxomatous mitral valve disease usually occurs sporadically, although there are well-described cases of familial clustering that involve an autosomal dominant mode of inheritance.16 Three genetic loci for autosomal dominant myxomatous mitral valve disease have been described, but the precise genes and mutations have not yet been identified. In the past, multiple nonspecific symptoms (atypical chest pain, dyspnea, palpitations, anxiety, and syncope) and clinical findings (low body weight, low blood pressure, and pectus excavatum) were associated with MVP and termed mitral valve prolapse syndrome. Because the mitral annulus is known to have a saddle shape, a normal mitral valve can appear to prolapse in certain echocardiographic views, most notably in the apical two- and four-chamber views. Although antibiotic prophylaxis for endocarditis was once advocated for certain patients with MVP, more recent guidelines do not recommend antibiotic prophylaxis in this group of patients.1,6 Beta blockers may be useful for alleviating symptoms of palpitations, anxiety, and chest pain in certain patients. Echocardiography should be performed if the patient has new cardiovascular symptoms or if the physical examination suggests that significant MR has developed.
Percutaneous balloon versus surgical closed and open mitral commissurotomy: seven-year follow-up results of a randomized trial.
Dilated cardiomyopathy with mitral regurgitation: Decreased survival despite a low frequency of left ventricular thrombus.
Sustained reverse left ventricular structural remodeling with cardiac resynchronization at one year is a function of etiology: quantitative Doppler echocardiographic evidence from the MulticenterInSync Randomized Clinical Evaluation (MIRACLE). I sentimenti in questi momenti cruciali per la vita di una donna sono infatti tantissimi e contrastanti: gioia immensa, soddisfazione ma anche un pizzico di comprensibile ansia per una situazione del tutto nuova. Gioia e paura si mescolano in un turbinio di emozioni che ti accompagneranno per i nove mesi piu importanti della tua vita.
Da questo momento e consigliabile porre un po’ di attenzione alla dieta: la stanchezza e le credenze popolari potrebbero indurti a mangiare per due! Ovviamente seguite scrupolosamente le indicazioni del vostro ginecologo che valutera caso per caso i controlli piu appropriati da eseguire durante la gravidanza. Il sesto mese e il momento ideale per cominciare a fare una lista del corredino per il piccolo. La capsula articolare ed il legamento dorsale formano soltanto il minimorinforzo dorsale di tale complesso articolare. The horizontal axis, about which plantar flexion and dorsiflexion occur, passa direttamente attraversola giunzione metafisi-diafisi della base del II ossometatarsale.
Questa dislocazione avviene perche la base del II metatarso si estende oltre l'asse orizzontale.
Treatment with a short-leg walking cast,6 a removable short-leg orthotic or a non–weight-bearing cast4 is continued for four to six weeks or until symptoms have resolved.
Some investigators5,6,11 believe that nonoperative management of fractures and fracture-dislocations is ineffective, because the reduction and alignment that occur with casting are lost when soft tissue swelling decreases.
All studies indicate that timely diagnosis facilitates treatment and decreases long-term disability. A person infected with bed bug bites may lead to various types of skin infections and sometimes may even cause blisters. Eradicate the bugs totally from the house using pesticide can help you to overcome bed bug bites.
Normal mitral valve function is dependent not only on the integrity of the underlying valvular structure, but on that of the adjacent myocardium as well. Less common causes include severe calcification of the mitral annulus, infective endocarditis, systemic lupus erythematosus, rheumatoid arthritis, and carcinoid heart disease.
Severe MS is now defined by a mitral valve area (MVA) of 1.5 cm2 or less (normal valve area 4-5 cm2) and is staged according to whether patients are asymptomatic (Stage C) or symptomatic (Stage D). If the patient is in sinus rhythm, there is presystolic accentuation of the murmur during atrial contraction. Transthoracic echocardiography is indicated for all patients with suspected MS to establish the diagnosis, quantify hemodynamic severity (mean pressure gradient, MVA, and pulmonary artery pressure), assess for concomitant valvular lesions, and demonstrate valve morphology to determine suitability for mitral commissurotomy (Class I).1 Characteristic findings of MS include valve thickening, restricted valve opening, anterior leaflet doming, and fusion of the leaflets at the commissures. An attempt to restore sinus rhythm with direct current electrical cardioversion or antiarrhythmic drugs may be considered. It may be considered in patients with MS if the valve anatomy is unsuitable for PMBC, in the presence of a left atrial thrombus or significant MR, or for patients that require surgery for other concomitant valvular disease or coronary artery disease.
Both mechanical and biological prostheses are used for MVR; the choice of valve often depends on factors such as age, need for concomitant anticoagulation, and left ventricular (LV) size. Increased left atrial pressures often lead to atrial arrhythmias (such as atrial fibrillation), which are not well-tolerated by patients with MS, frequently resulting in clinical decompensation.
The most common causes of MR in the United States are myxomatous degeneration, chordal rupture, rheumatic heart disease, infective endocarditis, coronary artery disease, and cardiomyopathy.

In acute severe MR, the left atrial and pulmonary venous pressures increase quickly, leading to pulmonary congestion and pulmonary edema.
Acute severe MR, as occurs with chordal rupture or papillary muscle rupture, is almost always symptomatic because the sudden regurgitant volume load in the nondilated left ventricle and atrium leads to pulmonary venous hypertension and congestion.
Patients with myxomatous MVP are at increased risk for cardiovascular complications, particularly when prolapse is associated with at least moderate MV or LV dysfunction.
Myxomatous MVP also may occur in conjunction with certain connective tissue disorders, such as Marfan syndrome and Ehlers-Danlos syndrome. Prospective testing has failed to confirm most of these associations.17 The classic findings of MVP on physical examination are a mid-systolic click, with a late systolic murmur from MR (Figure 7), heard best at the cardiac apex.
Therefore, the diagnosis of MVP should be based on a parasternal long-axis or apical three-chamber view. Patients with severe MR or high-risk features should be reviewed with an echocardiogram yearly or more often if their clinical condition warrants it. Guidelines on the management of valvular heart disease (version 12) [published online ahead of print August 24, 2012]. La prima cosa da fare e prenotare un appuntamento con il ginecologo per effettuare tutti gli esami necessari. Nulla di piu sbagliato considerato che il fabbisogno giornaliero e calcolato intorno alle 2000 calorie. Il settimo mese e il momento ideale per iniziare a seguire un corso pre parto: ci sono tantissime alternative, da quelli classici che tengono in ospedale, fino ai corsi che insegnano tecniche particolari di respirazione e di rilassamento per affrontare il momento del parto nel modo migliore. Most tarsometatarsal ligament injuries are grade I (pain at the joint, with minimal swelling and no instability) or grade II (increased pain and swelling at the joint, with mild laxity but no instability). Pertanto , con la perdita del supporto plantare e l'immobilita del II metatarso, mettendo il piede in estrema flessione plantare a causa di una spinta assiale si puo determinare lo stress che causa la lesione del complesso legamentoso di Lisfranc.
The potential for disability following a Lisfranc joint injury justifies the use of a non–weight-bearing cast.
This new valve staging classification (similar to how heart failure [HF] is classified) provides a means to integrate all forms of valve disease in a unified way. This obstruction results in the development of a pressure gradient across the valve in diastole and causes an elevation in left atrial and pulmonary venous pressures. With increasingly severe stenosis, the duration of the murmur increases and the opening snap occurs earlier during diastole as a result of higher left atrial pressure. The mean pressure gradient across the mitral valve on Doppler echocardiography (echo) in MS is at least 5 mm Hg; in severe stenosis, it is usually higher than 10 mm Hg. PMBC should also not be performed in patients who have left atrial thrombus or more than 2+ (moderate) MR, because the degree of MR usually increases following the procedure.
Surgical mitral commissurotomy (either open or closed) may be carried out through a median sternotomy or left thoracotomy incision. Morbidity and mortality are higher with prosthetic valve replacement than with surgical or balloon valvotomy. Indeed, patients with asymptomatic moderate to severe MS may decompensate during periods of increased physiologic stress, such as pregnancy or non-cardiac surgery. However, this may be a moot point as prognosis in secondary MR is more related to the underlying pathology than the degree of MRa€”typically prognosis alters little even if you fix secondary MR.
In chronic MR, a gradual increase in left atrial size and compliance compensate so that left atrial and pulmonary venous pressures do not increase until late in the course of the disease.
These patients require hemodynamic stabilization in the cardiac intensive care unit as emergent surgery is arranged. In patients with MVP, echocardiography is also useful in determining the presence and severity of MR and assessing left atrial and ventricular chamber size, LV function, and leaflet thickening and redundancy. Il tuo bambino nel primo mese e lungo pochissimi millimetri ed e formato da 40 paia di muscoli e 33 paia di vertebre. La pancia potrebbe cominciare a pesare ed e quindi consigliato iniziare con della ginnastica dolce o esercizi in acqua pensati per le donne in dolce attesa.
Interestingly, there are multiple signs in the hand which can signal 'anemia' - especially when the manifest together in some kind of combination!Anemia goes undetermined in many people, and symptoms can be minor or vague. TLa distrazione di III grado representa la  completa interruzione del complesso legamentoso, spesso accompagnata ad avulsione ossea della bratta inserzionale sulla II base metatarsale (vedi RM sottostante). The diastasis between the base of the first and second metatarsals or the medial and middle cuneiforms should be evaluated carefully and compared with the unaffected side. Elevated left atrial pressures lead to left atrial enlargement, predisposing the patient to atrial fibrillation and arterial thromboembolism. Because the gradient across the mitral valve is flow dependent, the severity of MS is more accurately defined by the MVA. Antibiotic therapy is important for the secondary prevention of rheumatic carditis, de-novo rheumatic valvular disease or worsened rheumatic valvular disease. A criticism of this guideline change has been that MR is now the only valve disease where you first need to describe the etiology before you can assess the severity.
Then, progressive LV dilation eventually leads to an increase in afterload, contractile dysfunction, and HF. Unless severe MR is present, findings on the chest radiograph and electrocardiogram are typically unremarkable. Il secondo mese e quello che segna per moltissime donne l’inizio del classici sintomi della gravidanza: avvertirete uno stimolo a fare pipi frequentemente, in alcuni casi delle vertigini, nausea e stanchezza. Nel quarto mese, il cuore del vostro piccolo e ormai sviluppato e ha un ritmo cardiaco da 120 a 60 battiti al minuto. Ora potrete anche pensare alla sua cameretta, ordinando i mobili, la culla e il fasciatoio. If flow across the mitral valve is reduced because of HF, pulmonary hypertension, or aortic stenosis, the murmur of MS may be reduced in intensity or may be inaudible.
The valve area may be measured by tracing the mitral valve opening in cross section by 2D or 3D echo. The new guidelines stress the importance of dedicated centers of excellence in which "Heart Valve Teams" of relevant specialists with expertise come together to guide complex decision making and to provide care particularly for high-risk patients. How widespread this change will be adopted is as yet unclear and it also remains to be seen whether the American Society of Echocardiography will also adopt this change in their echocardiographic definition of severe MR in their upcoming new valve guidelines. Left atrial enlargement predisposes the patient to atrial fibrillation and arterial thromboembolism. Pensate che dal quarto mese il feto reagira alle sostanze amare (ad esempio lo iodio) ingerite e immesse nel liquido amniotico, cessando di inghiottire e aggrottando la fronte.
Vi diamo un piccolo consiglio: parenti ed amici sicuramente faranno a gara per fare un bellissimo dono al piccolo e, dato che i bambini hanno bisogno di moltissime cose, l’idea potrebbe essere quella di fare una lista (proprio come quella per le nozze!) in un negozio di articoli per l’infanzia.
Long-term secondary prophylaxis, preferentially with penicillin, is therefore recommended for all patients with a history of rheumatic fever, rheumatic carditis or rheumatic valve disease. Niente paura pero se i sintomi sono blandi: ogni donna e una caso a parte e ci sono anche donne che, ad esempio, non soffrono di nausee o stanchezza. It is difficult to spot them and hence it can grow and multiply infecting people in the house. Il secondo mese e il momento ideale per cominciare anche ad utilizzare un buon idratante per ammorbidire la pelle e prevenire il rischio di smagliature. Nel secondo mese il piccolo comincia a formarsi: si sviluppano occhi, orecchie e naso, oltre alle tre parti principali del cervello. Si sviluppa anche il cuore, il tratto dell’apparato digestivo, la milza e il pancreas. Il terzo mese solitamente i sintomi piu fastidiosi della gravidanza si attenuano mentre cominceranno i cambiamenti piu visibili del vostro corpo, con un aumento di peso. L’aumento medio va da 7 etti a 2 chili e mezzo nel primo trimestre, fino ad arrivare a 3-5 etti alla settimana. La grandissima emozione di tutti i genitori, nel terzo mese di gestazione e quella di poter finalmente ascoltare il battito del cuore del vostro piccolo: un momento davvero indimenticabile!
Il bambino comincera a sviluppare i denti, le unghie di mani e piedi e i follicoli dei capelli. La pelle diviene piu spessa e gli organi genitali esterni cominciano a rendere visibile la differenza tra femmine e maschi. Per quanto riguarda i controlli, ogni mese va programmata una visita ginecologica ed esame delle urine, urinocoltura ed esame microbiologico vaginale.
E’ anche il momento di effettuare gli esami prenatali sulle malattie genetiche: amniocentesi, translucenza nucale del feto e tritest.

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