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17.12.2014

Treatments for herpes simplex 1, thyroid hormone alternative medicine - Review

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Manufactured by GlaxoSmithKline and marketed as Zovirax, it comes in liquid, capsule and tablet forms.
But acyclovir remains the oldest form of treatment for the herpes simplex 1 and simplex 2 viruses, or cold sores and genital herpes, which cause infections in those afflicted with HIV. When a virus takes over a living cell and reproduces itself, at the host cell’s expense, acyclovir is converted into an activated form by the same virus, enabling it to use acyclovir’s active form instead of the nucleoside which it normally uses in the manufacture of DNA; DNA is a crucial element in the replication of a virus.
Simply put, cells which have been virally infected tend to absorb more of the acyclovir than their normal counterparts; therefore there is more conversion of it into active forms, a condition which helps prolong the anti-viral activity. Acyclovir is not a curative treatment for viral infections but it can shorten the duration of the infections and lessen their possibility of developing into more serious medical conditions. In initial cases of genital herpes, acyclovir helps reduce lesions and decreases severity and frequency of infections which are recurrent. Depending on the severity of the genital herpes infection of an individual who has it for the first time, an oral dosage of acyclovir at 400mg thrice a day has been recommended by the CDCP or Center for Disease Control and Prevention; this dosage has to be ingested between seven and ten days. The patient may then choose to have daily or episodic therapy; the former is also known as suppressive therapy.
Herpes Simplex Virus 1, more commonly known as herpes, is an infection of the skin that includes watery blisters in the skin around the lips and mouth. Herpes is spread orally, through kissing or sharing of objects, such as toothbrushes or eating utensils.
Many patients do not require Chronic Suppression Therapy (CST), but those who experience six or more outbreaks a year with severe pain, may consider this treatment. The first infection of herpes simplex 1 usually occurs in childhood or adolescence, and can reoccur in adults. A person may also be infected with oral herpes by sharing towels, dishes, shaving razors, drinking straws, and other personal items with someone who has the virus.
Stress, hormone changes, or a high fever from a viral infection can trigger an attack of herpes simplex 1. The University of Maryland Medical Center states that many people mistake canker sores for herpes simplex 1.
Herpes simplex 1 causes ulcers or blisters in or around the mouth, most commonly on the outer edges of the lips. The immune-boosting herb echinacea purpurea has been clinically proven to exert an antiviral action against the herpes simplex 1 virus.
The scientists were able to demonstrate that echinacea does boost the immune system and does reduce the latency rate of the herpes simplex 1 virus. In an earlier experiment published in the September 2002 issue of Planta Medica, Canadian scientists found that echinacea purpurea root extract and echinacea pallida variation sanguinea root extract in a 70% alcohol base were most effective at inhibiting the viral activity in herpes simplex 1.
Tea tree oil, also known as Melaleuca alternifolia, was demonstrated to have antimicrobial properties in vitro (in a test tube) against the herpes simplex 1 virus. The scientists recommended a larger study to further study tea tree oil as a topical treatment for herpes simplex 1 virus.
Garlic has been a well-known anti-viral herb for centuries in the natural health community.
Another clinically proven herbal treatment for herpes simplex 1 is lemon balm oil, also called by its Latin name, Melissa officinalis. Non-toxic concentrations of lemon balm oil killed 98% of herpes simplex 1 cells, while higher concentrations of lemon balm abolished the virus completely.
Scientists found that topical pretreatment with lemon balm oil before eruptions was significantly more effective than treating the cells after they had become infected with herpes simplex 1 virus. Applying the propolis solution before or at the onset of the herpes outbreak created the most significant results. The Journal of Clinical Microbiology suggests zinc salts such as zinc gluconate and zinc lactate to reduce the symptoms of herpes simplex 1 virus.
A solution of 50 mm of either zinc gluconate or zinc lactate was able to 100% deactivate the herpes simplex 1 virus in vitro. Although not an oral dietary supplement, zinc in the form of zinc oxide cream, commonly used as a sun block, has been proven to have some benefit for herpes simplex 1.


L-lysine has also been clinically proven to regulate the immune system and help treat herpes simplex 1. Vitamin A-treated rabbits which were infected with herpes simplex 1 developed milder symptoms that rabbits which were left untreated in a 1979 clinical study published in Archives of Ophthalmology.
In 2008, the Journal of Nutrition reported that Vitamin E in the form of d-alpha tocopherol functions as an antioxidant and free radical scavenger. In this study, mice were placed on Vitamin E-deficient, adequate, or high-Vitamin E diets for four weeks.
The mice with adequate amounts of Vitamin E definitely limited the symptoms of herpes simplex-related encephalitis. Cellular and Molecular Life Sciences published a study in 2005 which was able to clinically demonstrate that the antimicrobial protein lactoferrin acts as a blocking agent to keep the herpes simplex 1 virus from entering a host cell.
Herdox is a natural approach to herpes management that helps limit the number and severity of breakouts.
The following recommendations are based on the 2009 guidelines for prevention and treatment of opportunistic infections[1].
Patients who have frequent or severe recurrences of HSV infections should be considered for suppressive therapy with valacyclovir, famciclovir, or acyclovir(Figure 4.
Acyclovir is a prescriptive medication that prevents the division and multiplication of viruses responsible for said infections. Taking acyclovir for treatment of genital herpes can help reduce its severity or prevent it from further recurrences. For shingles, acyclovir helps reduce pain, shorten the duration for healing, and halts the formation of more lesions and further spread of the virus. Acyclovir may be used intravenously for patients who have severe cases of genital herpes as well as those whose immune systems are compromised. Those who elected episodic therapy can choose to take acyclovir orally at 400mg thrice daily for a total of five days, 800mg thrice daily for a total of two days or 800mg twice a day for a total of five days.
For suppressive therapy, the CDCP recommends 400mg of oral acyclovir twice daily; those who are already infected with HIV should take it between 400mg and 800mg two or three times daily.
Even though a herpes infection may only have one outbreak of sores, it is highly likely that an individual will have multiple outbreaks in the near future. Since the virus will never leave the body, only the physical aspects of herpes will go away. Buka creates a treatment plan that will take between six months to a year of prescription medications. According to the New York Times, herpes simplex 1 is so common that up to 90% of adults will have had the virus at some point in their lives. The medical journal Intervirology published a clinical study performed in 2009 in which scientists at Golestan University of Medical Sciences and Health Care in Gorgan, Iran used echinacea purpurea to prevent the spread of the herpes simplex virus to the eyes during the latent stage. If echinacea purpurea is taken on a regular basis, the herb will reduce the effects of a herpes outbreak. A study published in the Journal of Antimicrobial Chemotherapy stated that a 6% solution of tea tree oil gel or a placebo was given to adults between the ages of 18-70 who had recurring bouts of herpes simplex 1 virus. Clinical science has been able to confirm garlic’s ability to kill viruses such as herpes simplex 1. Phytomedicine reported in 2008 that the essential oil of lemon balm inhibited the viral activity of herpes simplex 1 in monkeys.
The European Medicines Agency states that olive leaf extract acts as a virucide when applied to cell cultures in vitro one hour before infection with herpes simplex 1 virus. In vitro, scientists found that a 0.5% solution of propolis extract caused a 50% inhibition of herpes simplex 1 virus.
However, even when a 10% propolis solution was administered two hours after a herpes eruption, there was 80-85% protection. The medical journal Canadian Family Physician reported that if zinc oxide cream is applied to the skin before going out in the sun, it prevents herpes outbreaks triggered by sunlight to a degree.
The Journal of Medical Virology published a study in 1989 stating that guinea pigs treated with L-lysine stayed symptom-free, while the untreated control group were symptomatic for three days after all of the guinea pigs were inoculated with HSV-1.


Some of the rabbits showed no symptoms of herpes-related keratitis at all after taking Vitamin A.
The treatment of orolabial lesions and genital HSV infection (initial or recurrent) should consist of a 5 to 14 day course of valacyclovir (Valtrex), famciclovir (Famvir), or acyclovir (Zovirax) (Figure 4.
Guidelines for prevention and treatment of opportunistic infections in HIV-infected adults and adolescents: recommendations from CDC, the National Institutes of Health, and the HIV Medicine Association of the Infectious Diseases Society of America.
FDA (Food and Drug Administration) approved the drug for use against the virus known as varicella-zoster in the treatment of viral infections which caused shingles and chicken pox. A study published last year, however, revealed that herpes strains which are resistant to acyclovir could develop over a certain period of time.
There have been reports of seizures, hepatitis, agitation, hypersensitivity reaction, muscle pain, and anemia, all of which must be reported to the patient’s doctor for advisement.
Attacks of the herpes virus may occur and create unnecessary stress, fatigue, or severe irritation to the infected area. CST helps those who are emotionally and psychologically stressed from the appearance and pain of herpes.
For patients that follow the prescribed medication plan they experience a decrease or complete halt in outbreaks.  Set-Up an Appointment Today! Herpes simplex 1 can also lead to meningoencephalitis, a viral infection in the lining of the brain.
Over time, outbreaks of oral herpes become much less frequent, and eventually seem to disappear. Herpes simplex 1 virus was reduced by 97% in two hours after applying zinc lactate in vitro. Additional support for use of suppressive therapy in HIV-infected patients arose from studies that have shown HSV outbreaks can result in increased HIV transcription and increased genital and plasma HIV RNA levels[12,13]. Doctors have also prescribed acyclovir in the treatment of the Epstein-Barr virus which causes mononucleosis.
Buka will discuss and examine the severity of your symptoms, and provide a treatment plan best for your situation. In the United States, approximately 70% of HIV-infected adults have serologic evidence of established infection with HSV-2 infection and 95% are seropositive for either HSV-1 or HSV-2[1]. For patients with severe mucocutaneous HSV lesions, intravenous acyclovir is recommend for initial therapy, followed by oral therapy when the lesions start to resolve. These findings correspond with data that show certain HSV regulatory proteins (ICPO, ICP27 and VP16) can induce HIV replication and herpes simplex virions can increase HIV expression in macrophages[14]. For persons co-infected with HIV and HSV, most available data suggest HSV suppressive therapy has a favorable impact on genital and plasma HIV levels[15,16,17,18].
Although most HSV lesions require only 5 to 14 days of therapy, treatment of moderate-to-severe chronic ulcerative HSV lesions usually requires at least 14 days and patients should have close follow-up. In general, these patient should receive therapy for HSV until the lesions have completely healed[10]. In one study that involved HSV suppressive therapy for HIV and HSV co-infected women in Africa on highly active antiretroviral therapy, the women had reduced genital shedding of HSV, but no significant decrease in plasma HIV RNA levels. If the patient has received multiple courses of therapy for recurrent HSV infection, or is taking chronic HSV suppressive therapy when new lesions develop, greater consideration should be given to possible acyclovir-resistant HSV.
Two large clinical trials investigated the impact of suppressive herpes therapy on HIV acquisition in HIV-negative, HSV-2 co-infected individuals, but found no benefit of suppressive therapy[19,20]. Evaluation and treatment of acyclovir-resistant HSV infection is discussed in Case 2 in this Dermatologic Manifestations module.



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