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10.03.2014

Medicines for herpes simplex 2, treatment herpes simplex 1 - How to DIY

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The drug acyclovir (Zovirax) does not have activity against viral pathogens until it is converted to the active form acyclovir triphosphate. Cidofovir (Vistide), formerly HPMPC, is an antiviral compound that reaches the cell in a monophosphorylated form whereby it is activated by cellular kinases. The herpes thymidine kinase is a 376-amino-acid protein encoded by the UL23 gene and the herpes DNA polymerase is a larger protein (approximately 1,200-amino-acids long) and it is encoded by the UL30 gene[6].
Perioral Lesion with Extension to Face Caused by Acyclovir-Resistant Herpes Simplex Virus">Figure 4). If acyclovir resistance is suspected, testing for HSV should include a viral culture of the sample, since resistance testing will require isolation of virus.
Enzymes in the human cell add two more phosphates to eventually form the active drug acyclovir triphosphate. Guidelines for prevention and treatment of opportunistic infections in HIV-infected adults and adolescents: recommendations from CDC, the National Institutes of Health, and the HIV Medicine Association of the Infectious Diseases Society of America.


Oral ACV spreads widely throughout the body via bodily fluids, topical ACV works in the area of application but has little effect elsewhere in the body, and intravenous ACV is reserved for patients with compromised immune systems (such as those already suffering from HIV). This is where Valacyclovir provides an exciting and unexpected benefit: when taken in a daily suppressive regimen, Valacyclovir was found in an eight-month study to reduce Herpes transmission by 50% among susceptible partners. It must be stressed again that, while these drugs provide significant improvements in the lives of people infected with Herpes, there remains no cure.
Acyclovir triphosphate, the active form of acyclovir, is present in 40- to 100-fold higher concentrations in herpes simplex virus (HSV)-infected cells than in uninfected cells.
Laboratory testing for acyclovir resistance includes a variety of phenotypic assays, such as the plaque-reduction assay, dye-uptake assays, and viral DNA-inhibition assays[5]. The acyclovir triphosphate competes with 2-deoxyguanosine triphosphate (dGTP) as a substrate for viral DNA polymerase, as well as acting as a chain terminator. Valacyclovir is intended for use in the treatment of both genital and oral Herpes, helping to curb infections by reducing the frequency and severity of outbreaks.


Acyclovir triphosphate has a two-pronged mechanism of action: (1) it competes with 2-deoxyguanosine triphosphate (dGTP) as a substrate for viral DNA polymerase and (2) once it becomes incorporated into the replicating viral DNA, it acts as a chain terminator because it does not have a terminal 3' hydroxyl group.
It has been approved by the FDA to treat both HSV-1 and HSV-2 during initial outbreaks, recurrent outbreaks, and as a therapy to suppress the virus’ activity for extended periods of time.
It must be stressed, though, that while drugs to control the infection are available and effective, there currently is no cure for a Herpes infection.
For best results, an Acyclovir regimen should begin at the first sign of Herpes sores or lesions.
Using condoms will not only prevent pregnancy, but will also prevent against the transmission of various STIs (including Herpes).



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