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My speculation is that a ketogenic (or high-fat) diet helps in NBIA by allowing Coenzyme A, the crucial enzyme which is under-generated in NBIA, to be redistributed from organs like the liver and muscle, where it is manufactured in abundance, to the brain where it is most needed. An aside: I’ve been hearing recently from a number of people who experienced great benefits in neurological conditions – NBIA, epilepsy, migraines, and others – following the ketogenic version of our diet, and later transitioned to the regular version of PHD with more carbs and less fat, and continued to maintain all the neurological benefits they had first achieved on the ketogenic diet. Thank you for being part of the forces around his life who helped support him, love him, and provide him with the best life that was possible for him.
The weekend was happy for us, because a number of readers left word of health improvements. I too have had Raynaud’s all my life … In the past two months, I have modified my diet in line with Paul’s suggestions for Migraine.
Thanks for asking about the migraines.В  They are in fact vastly improved!В  Since they have been so intractable in the past, I guess I was waiting to make sure the effect was stable before I reported my results.
I think there is something to this, but no headache relief for me.  Do feel more at peace with myself. This is already a long blog post, so I won’t go into an analysis of why and how the ketogenic variant of the Perfect Health Diet can cure migraines, reduce anxiety, and improve sleep.
I realize that you’ve already tried those, Kate, and didn’t notice an effect, but you may notice a benefit now that your diet is better. I believe that fading benefits are likely a result of eating the wrong diet, and that on the ketogenic version of the Perfect Health Diet the good effects will prove permanent.
It’s a little hard to nail down the exact cause of the bone fractures and stunted growth because the diets were so atrocious.
For those keeping score, Ross Carbohydrate Free consists of soy protein isolate, high oleic safflower oil, soy oil, and coconut oil, plus vitamins and minerals. Purified diets without added ultratrace elements support growth and reproduction, but investigators have noted that animals exposed to stress, toxins, carcinogens or diet imbalances display more negative effects when fed purified diets than when fed cereal-based diets. The second problem, from my point of view, is that they made little use of short-chain fats and ketogenic amino acids to make the diet ketogenic.
All of the parents interviewed preferred the diet over medications; 12 cited fewer side effects (such as cognitive dulling, sedation, ataxia, and behavioral problems) from medications that were successfully discontinued, and 11 cited decreased seizure frequency over medications as their primary reason.
The ketogenic diet had more than 6 times the fat of the control diet, the same amount of protein, and no carbohydrate at all. The ketogenic diet also had lower micronutrient levels (“Ash” and “Vitamin”) than the control diet, and much higher omega-6 levels.
Rats were fed ad libitum, meaning they could eat as much as they liked; they chose to eat twice as many calories on the ketogenic diet.
When protein+carbohydrate intake is deficient, muscle will tend to be catabolized for protein. You can see in panel A that rats on the control diet weighed more than rats on the ketogenic diet. The paper did not measure length of the rats, but I would bet that the ketogenic diet rats were not only lighter, but shorter as well. Like the children on Johns Hopkins Hospital’s diet for epilepsy, these malnourished rats experienced stunted growth. As we discussed in the first post in this series, Ketogenic Diets, I: Ways to Make a Diet Ketogenic, there are 3 ways to make a diet ketogenic. Via Nigel Kinbrum comes an interesting paper [3] exploring the use of branched-chain amino acids as an adjunct to ketogenic diets for epileptic children. Regarding the improvement of seizures, we found 3 patients who had already achieved a reduction of seizures on the ketogenic diet to experience a complete cessation of seizures, while 2 other patients had a further reduction of seizures from 70% on ketogenic diet to 90%.
Lysine, the other purely ketogenic amino acid, is generally considered to have no significant toxicity.
I believe the extreme limits on carb and protein intake in conventional clinical ketogenic diets are responsible for their growth stunting, muscle destroying, fattening effects. In order to supply sufficient protein and carbs while maintaining ketosis, it is necessary to provide ketogenic short-chain fats and amino acids. Personally, I wouldn’t attempt a long-term ketogenic diet without the aid of coconut oil (or MCTs), lysine, and the branched chain amino acids. I was going to write a single post about how to implement a therapeutic ketogenic (ketone-generating) diet.


I soon realized that with so many factors affecting diet design, it would be hard to fit everything into a single post. Maybe we’ll look at some other diseases after that, or maybe I’ll just move on to the lemon juice series I’ve been planning.
The liver is responsible for making sure that the body (but especially the brain and heart) have access to a sufficient supply of energy from the blood.
While preparing this post, I was surprised at how long it took for doctors to appreciate that ketones are an acceptable alternative energy source for the brain.
The use of prolonged starvation for the treatment of obesity has posed a fascinating problem; namely, that man is capable of fasting for periods of time beyond which he would have utilized all of his carbohydrate resources and all of his proteins for gluconeogenesis in order to provide adequate calories as glucose for the central nervous system. This makes it a bit easier to understand why ketogenic diets have not yet become standard therapies for neurological diseases. If your diet could include only compounds found in the body – glucose, complete protein, and long-chain fats stored as triglycerides or phospholipids – then we can use the above numbers to estimate the “ketogenic potential” of the diet.
The ketogenic formula was originally developed by Wilder at the Mayo clinic in the 1920’s. This formula basically treats all fats as triglycerides of long-chain fatty acids, and protein as “complete” protein with a typical mix of amino acids. A mix of amino acids that is not “complete,” but is biased toward the ketogenic amino acids.
So if the diet is rich in leucine and lysine, but poor in glucogenic amino acids, then it will be highly ketogenic. This means that if you eat a lot of coconut oil (which is 58% short-chain fats), you deliver a lot of fat to the liver for disposal. Come to the Perfect Health RetreatCome join us for a week at the beach learning how to achieve a lifetime of great health! Buy our bookPerfect Health Diet: Regain Health and Lose Weight by Eating the Way You Were Meant to Eat is a great way to understand the dietary and nutritional practices that lead to optimal health.
I wrote about their case in Ketogenic Diet for NBIA (Neurodegeneration with Brain Iron Accumulation), February 22, 2011. Mathias and another boy who tried the diet, Zachary, regained control their limbs, and the spasms and pain went away.
I grip the steering wheel tightly, sit forward in the seat, and am generally hyper vigilant. The Four Steps of the Perfect Health Diet eliminate food toxins, optimize nutrition, and enhance immunity; therefore they remove most of the causes of disease and should render most diseases curable. Hopefully we can bring the good news to others, and gather more evidence to prove that diet is the best therapy – for migraines, and for many other diseases as well.
Effect of high-dose vitamins, coenzyme Q and high-fat diet in paediatric patients with mitochondrial diseases.
Our immediate goal is to help the NBIA kids, Zach and Matthias, but most of the ideas will be transferable to other conditions – and even to healthy people who engage in occasional or intermittent ketogenic dieting for disease prevention. Skeletal fractures occurred in 6 of 28 children following the ketogenic diet for 6 years; 4 children had fractures at separate locations and times.
First, purified diets are notoriously unhealthy; they are missing all kinds of helpful compounds found in real food.
Instead, they relied on protein and carb restriction and overall calorie restriction to force ketone production. Use of short-chain fats and ketogenic amino acids can trigger high ketone production even on a nourishing diet.
You don’t often see bodybuilders or Olympic weight lifters who eat a continuously ketogenic diet. Which brings us to a rat study [2] discussed by CarbSane in her post “Ketogenic Diet increases Fat Mass and Fat:Total Body Mass Ratio”.
Since protein has to be converted to glucose on zero-carb diets, this ketogenic diet is actually protein restricted.
One of them is severe protein+carb restriction, but the other two – short-chain fat consumption and supplementation of the ketogenic amino acids lysine and leucine – can generate ketosis even if substantial carbs and protein are eaten. It is remarkable that 1 of our children had improved so much that she is now applying to attend normal grade level for her age.


As far as we know, it is the first time branched chain amino acids have been used in patients with epilepsy and we had to be very cautious with their administration.
Providing exclusively leucine as an adjunctive treatment to ketogenic diet is impossible because it is toxic when consumed out of proportion to valine and isoleucine….
If we have to add other branched-chain amino acids to avoid leucine toxicity, it would be better to add isoleucine, which can be ketogenic, than valine which is only glucogenic.
Providing supplemental amino acids to epileptic children on ketogenic diets improved their health and allowed them to maintain ketosis with higher protein intake. Ketogenic diet-fed rats have increased fat mass and phosphoenolpyruvate carboxykinase activity.
Branched chain amino acids as adjunctive therapy to ketogenic diet in epilepsy: pilot study and hypothesis. There are various ways to make a diet ketogenic, and different ways are appropriate in different diseases. The lemon juice and acid-base balance issues will fit in nicely since kidney stones and acidosis are risks of ketogenic diets and lemon juice relieves those risks.
They diffuse throughout the body into cells, and are taken up by mitochondria and oxidized for energy. Epileptics caught a lucky break – the ketogenic diet was already in use for epilepsy in the 1920s.
Richard Bernstein in his Diabetes Solution to help people appraise the ketogenicity of a diet.
For instance, suppose you want to eat enough carbs to avoid “zero-carb dangers” such as mucus deficiency.
Short-chain fats do not appear in cell membranes and are not stored in adipose tissue (except for a little 12-carbon fatty acids). The disposal process for fat is conversion to acetyl CoA followed by either burning in the TCA cycle or conversion to ketones.
We have to look at diseases and diet risks to figure out which way of making the diet ketogenic is optimal.
Last month, when the sun came out and the temps got over 25, I enjoyed a successful 2-mile snow hike for the first time in three years.
Rob was willing to fast for 30 days (!); Kate read the book in January and was able immediately to make substantial diet, lifestyle, and supplement changes. The ketogenic diet rats had more fat mass but less body mass; they had obviously lost muscle and bone mass.
The ketogenic diet’s therapeutic potential for other neurological disorders probably couldn’t have been appreciated until after 1967, and by then medicine had turned its back on dietary therapy. Typically, 10-12% of energy from a triglyceride is in the form of glycerol (which has the potential to become glucose) and 88-90% is in the form of fatty acids (which have the potential to become ketones). Rather than being transported throughout the body, they are shunted to the liver for disposal. My Raynaud’s has further improved, as measured by the fact I sometimes forget to turn the heat up in the morning, and cold extremities don’t always alert me to my forgetfulness! For humans, we recommend a minimum protein+carb intake of 600 calories per day, which is about 30% of calories for a sedentary adult.
You can achieve both by eating carbs for glucose, but also eating short-chain fats and ketogenic amino acids to make ketones.
The plan is based on healthy whole foods, and includes an easy-to-use, step-by-step ketogenic food guide and the unique "KetoMeter" for quickly and easily choosing the right foods in the right amounts.
We’ll look at what they’re eating now, and consider ways they might be able to improve their diets further – and, hopefully, get further improvements in health, longevity, and function.




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Comments to “Ketogenic diet meal plan for epilepsy”

  1. IMPOSSIBLE_LIFE:
    Phytosterols, vitamin E, copper, manganese, selenium, various.
  2. EzoP:
    Flour can be used as a substitute almond flour.
  3. Qaqquli:
    Sunflower seeds are loaded with protein copper, manganese, selenium.