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A common eye problem with cats is called Cat Conjunctivitis, more commonly called pink eye in cats. Cat Conjunctivitis can sometimes be contagious to other cats, so it’s best to keep your cat away from others until the cat pink eye is gone.
Eye Easy for Cat Conjunctivitis from Pet Wellbeing is an all natural Herbal supplement for pink eye in cats. Pet Wellbeing is a well known and respected pet supplements and all natural pet treatment product company. So if you think your cat has pink eye, order a bottle of Eye Easy for Cat Conjunctivitis and start the healing process. Return to top of pageCopyright 2011 Orlando Pet Services • Orlando Pet Services has everything your looking for about pet products, pet services and more in the Orlando area.
Anatomical location of feline adrenal glandsCushing's syndrome in a cat, where fragile skin is easily torn during grooming.
Cats do not show as much drinking and urinating as dogs do, unless they have diabetes mellitus concurrently. Cats do not routinely show any specific changes on a regular blood panel or urinalysis, although it is common to see hypercholesterolemia, hyperglycaemia, mild leukocytosis and erythroid regeneration (nucleated RBCs).
The ACTH stimulation test is used, but two blood samples need to be analysed at 30 and 60 minutes, instead of the 1 sample at 2 hours for the dog.
Advantages - The ACTH stimulation test is the best screening test for distinguishing spontaneous from iatrogenic hyperadrenocorticism.
Disadvantages - The ACTH stimulation test does not reliably differentiate adrenal-dependent from pituitary-dependent hyperadrenocorticism. Interpretation - It is essential to use absolute values for pre- and post- ACTH plasma cortisol concentrations rather than a ratio or percentage increase in post-ACTH cortisol concentration over the basal concentration. The LDDS test is used but the dexamethasone that is injected needs to be given at a higher dose. Advantages - The low-dose dexamethasone suppression test is more reliable than the ACTH stimulation test in confirming hyperadrenocorticism, since the results are diagnostic in all adrenal-dependent cases and in 90 to 95 per cent of dogs with pituitary-dependent hyperadrenocorticism. Disadvantages - The low-dose dexamethasone suppression test is not as accurate as the ACTH stimulation test for the detection of iatrogenic hyperadrenocorticism. Interpretation - Interpretation of the results of a low-dose dexamethasone suppression test must be based on the laboratory's normal range for the dose and preparation of dexamethasone administered. The HDDS test to differentiate PD from AT has not been refined to the point that is of diagnostic value.
In general, results of these tests can be variable, and must be interpreted in conjunction with the history and clinical findings. If the above tests suggest Cushing's then radiology can be helpful since up to 30% of feline adrenal tumours are mineralised. Surgery is needed to remove one of the adrenal glands if the gland has a tumour, and both glands if the problem is PD.
Unfortunately, cats with Cushing's can be poor aesthetic risks due to diabetes mellitus and fragile skin.


The causes of pink eye in cats are many, but most commonly are because of eye infections, something in the eye, including eyelashes, and trauma to the eye, such as cat fights. Lucky pink eye in cats is easy to treat with the right Cat Conjunctivitis treatment products. Check out the Orlando pet events page, pet rescue groups, pet sitting and dog walking services, pet groomers, Central Florida veterinarians, doggy day care and pet boarding places.
Sigurdsson MD 41 Comments Recently, concerns have been raised about the increased risk of diabetes associated with statin therapy.However, it has been pointed out that the risk of diabetes is low in absolute terms and when compared with the reduction in cardiovascular events achieved by treatment.
One reason is because they tend to be more resistant to higher levels of cortisol, especially if given as a medication for other diseases such as skin allergies. Most cats are presented in a more advanced state of Cushing's disease because the early symptom of drinking and urinating are not observed. In spontaneous hyperadrenocorticism, the ACTH stimulation test reliably identifies more than 50 per cent of dogs with adrenal-dependent hyperadrenocorticism and about 85 per cent of dogs with pituitary-dependent hyperadrenocorticism.
A diagnosis of hyperadrenocorticism should not by excluded on the basis of a normal ACTH response if the clinical signs are compatible with the disease. This test, when used in conjunction with the ACTH stimulation test, is one of the best ways to diagnose Cushing's in the cat. The test is also affected by more variables than the ACTH stimulation test, takes 8 hours to complete and does not provide pre-treatment information that may used in monitoring the effects of mitotane therapy. If the dose of dexamethasone fails to adequately suppress circulating cortisol concentrations in a dog with compatible clinical signs, a diagnosis of hyperadrenocorticism is confirmed. In light of the fact that Cushing's is uncommon in cats, these tests need careful interpretation. If both glands are removed the cat has to be on supplemental cortisone and mineralocorticoids for the rest of its life. When this occurs we sometimes will use medical therapy to help control the problem and make our patient a better anaesthetic risk. In other words, the potential benefits to health are believed to outweigh potential risks. They might also have hepatomegaly, weight gain, pot-bellied appearance, and muscle wasting. It is a simple and quick test to perform and documents excessive production of glucocorticoids by the adrenal cortex in cases of hyperadrenocorticism. Occasionally, an animal under chronic stress may develop some degree of adrenal hyperplasia, which produces an abnormal ACTH response. The low-dose dexamethasone suppression test does not reliably differentiate pituitary-dependent from adrenal-dependent hyperadrenocorticism. While basal and 8-hour post-dexamethasone samples are most important for interpretation of the test, one or more samples taken at intermediate times (for example, 2, 4, or 6 hours) during the test period may also prove helpful. Ultrasonic evidence of an enlarged adrenal gland (especially if unilateral) or changes in internal adrenal architecture is strong evidence of an adrenal tumour (AT). No fall occurs in patients with ectopic adrenocorticotropic hormone (ACTH) syndrome, even with high doses of dexamethasone.


Some cats with concurrent diabetes mellitus will no longer have the disease when their adrenal tumour is removed. Of the 26 reported cases of feline Cushing's syndrome and one previously unreported case, 21 (78%) have been females.
Often the elevated alkaline phosphatase is due to liver changes from unregulated diabetes mellitus. The information gained is also useful in providing baseline information for monitoring mitotane therapy, although different criteria are used to interpret cortisol results during treatment. This may be seen for example with diabetes mellitus or pyometra and a normal cortisol response to ACTH stimulation will be obtained after treatment of the underlying disease in these cases. This contrasts with the responses seen in patients with pituitary-dependent disease in whom inadequate suppression after low-dose administration of dexamethasone, and at least 50% suppression after administration of a high dose of dexamethasone (2mg 6-hourly) are characteristic. This female sex predilection resembles the human syndrome and contrasts with canine hyperadrenocorticism, where no sex predilection occurs. The urine cortisol: creatine ratio test is helpful in cats, especially since it is a relatively stress free test compared to blood sampling.
It can affect the ability to control the blood sugar level in cats with diabetes mellitus concurrently. Cushing's may be caused by a pituitary tumour (90% are adenomas), pituitary hyperplasia, adrenal tumours, adrenal hyperplasia, by non-endocrine tumours (usually lung) or may be iatrogenic (external corticosteroid use). It the test is elevated it might be Cushing's, but there are also other situations that cause this elevation.
Millions of people are taking statin drugs; many of them will not derive much benefit, and some will be harmed. The LDL receptor then moves back to the cell surface where it can bind to additional LDL particles. The LDL receptor then moves back to the cell surface where it can bind to additional LDL particles. This is believed to be a key step for the lowering of blood levels of LDL cholesterol achieved by statin treatment.Familial Hypercholestrolemia and DiabetesFamilial hypercholesterolemia is a dominantly inherited disease characterized by high plasma levels of LDL cholesterol. This assumption is based on findings from a large observational study of men with metabolic syndrome.It is likely that the prevalence of diabetes among statin users varies according to the population being studied. Individuals with metabolic syndrome usually have insulin resistance and are at increased risk of developing diabetes. Lipid Biomarkers (cholesterol, lipoproteins) and Cancer Risk Ferritin – High and Low Ferritin Explained Familial Hypercholesterolemia (FH) is Not Detected by Measuring Cholesterol Cardiovascular Risk Factors and Age: Is it all about the cholesterol?



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