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Diabetes chart- convert hba1c to equivalent blood glucose, Free printable charts and tools to better understand, track and manage your blood glucose.. 1Department of Dermato-Allergology, Gentofte Hospital, University of Copenhagen, Niels Andersens Vej 65, DK-2900 Copenhagen, and 2Department of Virology, Statens Serum Institut, Copenhagen, Denmark. Hand, foot, and mouth disease (HFMD), as the name indicates, affects the hands, feet and mouth, with haemorrhagic and papulo-vesicular lesions (1). HFMD is generally a mild and self-limited disease caused by enteroviruses, primarily strains of coxsackieviruses, A10 and A16, and enterovirus 71. HFMD is a highly contagious disease occurring most commonly in children below the age of 5 years (1, 3). A 49-year-old man with a 2-day history of multiple painful haemorrhagic papules and vesicles in the palms, soles, face and scalp (Fig. A 37-year-old woman presented with multiple vesicles on the palms, soles, scalp and genital area. A widespread vesicular eruption on the scalp was initially interpreted as secondary bacterial infection and the patient was treated with oral antibiotics.
Histological examination of a 3-mm punch biopsy suggested an enterovirus infection, and enterovirus RNA was identified in vesicular fluid by real-time PCR. CV A6 is not one of the usual causes of HFMD; the serotype is more often associated with herpangina, in which the ulcerative lesions are predominantly located on posterior oropharyngeal structures (1, 3, 4).

HFMD spreads through contact with salvia, respiratory secretions, fluid in vesicles, and faeces. Differential diagnosis of HFMD in the 2 cases described here included drug eruptions, erythema multiforme, vasculitis, dermatitis, bacterial infections, herpes, and varicella. 5 foot 8 inches, 230 pound man with Type 2 diabetes mellitus for 7 years Fasting blood sugar  Range averages 110-150 ???? Enteroviruses are small, non-enveloped, single-stranded RNA viruses that belong to the family Picornaviridae (2). Small epidemics of HFMDs are seen in nursery schools or kindergartens, usually during the summer and autumn months. In addition to presenting typical lesions on the hands and feet, both cases had involvement of the scalp, which is unusual, and no lesions in the oral mucosa where lesions are most commonly found. Enterovirus RNA was detected by real-time PCR, and coxsackievirus A6 (CV A6) subtype was identified by semi-nested reverse transcriptase (RT) PCR and sequencing. The subtype CV A6 was subsequently identified by sequencing the semi-nested RT-PCR product. Firstly, the causative agent was CV A6, a virus that recently has only been associated with HFMD in Scandinavia (5, 6). Patients do not need to have manifestations in all 3 areas to be classified as having HFMD. Single cases of both immunocompetent and immunocompromised adults with HFMD caused by CV A6 have been described (3, 11, 12 ).
Awareness, that papulo-vesicular scalp lesions may be part of the clinical presentation of HFMD might improve future diagnosis and surveillance of HFMD.
Several major outbreaks have been described throughout the world, especially in Asia (2, 4).

Histological examination from a 3-mm punch biopsy of another vesicle was consistent with post-viral dermatosis, confirming the diagnosis of HFMD. Secondly, the location of the cutaneous lesions to the scalp has to our knowledge not been described previously in HFMD.
Cases with symptoms elsewhere, primarily on the buttocks and limbs, have also been described (1, 3, 4, 7). Furthermore, within the last 2 years outbreaks of HFMD caused by CV A6 have been reported in the USA, Japan and Europe (5a€“7, 9, 10).
For diagnosis of enterovirus the timesaving and sensitive PCR technique is preferred to viral culture (2, 8).
Other typical clinical findings are fever, malaise, abdominal pain and upper respiratory tract symptoms (1, 4, 8, 9).
Clinical and molecular characterizations of hand-foot-and-mouth disease in Thailand, 2008-2009. Co-circulation of coxsackieviruses A6 and A10 in hand, foot and mouth disease outbreak in Finland. Comparison of enterovirus 71 and coxsackievirus A16 clinical illnesses during the taiwan enterovirus epidemic, 1998. Notes from the field: severe hand, foot, and mouth disease associated with coxsackievirus A6 a€“ Alabama, Connecticut, California, and Nevada, November 2011 a€“ February 2012.

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