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You have probably heard some of the stories about Green Tea and Weight Loss, but there are so many other benefits that most people ignore, or that they are just completely unaware of. This is a condition where the artery walls thicken from the accumulation of fats such as cholesterol.  Clinical studies indicate that the antioxidant properties of green tea may help prevent Atherosclerosis, particularly Coronary Artery Disease. Several studies have shown that both green and black teas helps boost the immune system, helping  protect against cancer. Green tea may help reduce inflammation associated with Crohn's Disease and Ulcerative Colitis, the two types of IBD.
A few small clinical studies have found that daily supplements of green tea extract powder lowered the hemoglobin A1c level in individuals with borderline diabetes.
Studies have shown that men who drink more than 10 cups of green tea per day are less likely to develop disorders of the liver. Results from several animal and human studies suggest that one of the polyphenols present in green tea, "catechin", may help treat viral hepatitis (inflammation of the liver from a virus).
So by all accounts, the value of utilizing Green Tea in your daily routine far out way any reason for not drinking it.
Honey has different enzymes & antioxidants in it that make it therapeutic and not like other items with sugar. While I've lost a considerable amount of weight in the past 6 months, I have to say that I really need to start working out more. DisclaimerThis site is designed for educational purposes only and is not engaged in rendering medical advice, legal advice or professional services.
If you were of the opinion that a cucumber was purely for use in a salad, then think again. Sudden and frequent cravings for sweet things or food in general – usually linked with type 2 diabetes and unstable blood sugar levels. Much slower ability to metabolize fats – small fatty tumors just under the skin, bloating, weight gain and hypertension.
Problems in the digestive system – irritable bowel syndrome, inability to properly manage food and drink, constipation, indigestion and hemorrhoids. Your liver is one of the vital organs of your body which constantly works on the improvement of your overall health. Throw Away Your Glasses By Using Only THIS One Ingredient to Increase Your Vision Up To 97%! The pancreas has many islets that contain insulin-producing beta cells and glucagon-producing alpha cells. Since diabetes is a disease that affects your body's ability to use glucose, let's start by looking at what glucose is and how your body controls it.
When you eat food, glucose gets absorbed from your intestines and distributed by the bloodstream to all of the cells in your body.
To maintain a constant blood-glucose level, your body relies on two hormones produced in the pancreas that have opposite actions: insulin and glucagon. Insulin is made and secreted by the beta cells of the pancreatic islets, small islands of endocrine cells in the pancreas.
As such, insulin stores nutrients right after a meal by reducing the concentrations of glucose, fatty acids and amino acids in the bloodstream.
Metformin is the least expensive, most effective and most common medication that is prescribed by doctors. Not only for this type 2 diabetics condition, doctors also prescribe this medication for people who experience fasting blood sugars that are more than normal but are not in the range of diabetic.
Metformin is available in the form of tablet, as a liquid form and extended release tablet. Metformin is safe for majority of people who have normal liver, kidney and lung function, but all medicines have side effects.
Being on the Mito Registry will ensure patients have the earliest possible opportunity to enter any relevant study or clinical trial.
Find support from someone who understands what it is like to live with mitochondrial disease. Mitochondria are the power houses of the cell providing the body with over 90% of the energy it needs to sustain life. The following information is intended as a general guide to help you understand the symptoms of mitochondrial disease and where to go for support and help.
Until about five years ago, mitochondrial disease (the name, not the disease), did not seem to exist.
However, type ‘Mitochondrial Disease’ into Google and faces appear of people who have it, many are children.
Many experts refer to mitochondrial disease as the ‘notorious masquerader’ because it mimics so many different illnesses, affecting both children and adults.
Recent research demonstrates that mitochondrial mutations are present in at least 1 in 200 people and that at least 1 in 5,000 will develop serious illness. Like the small light that hangs off a key-ring, or the huge emergency torch that saves us in a blackout, each organ requires different amounts of energy.
Hence, high energy organs that do a lot of work, such as the brain, contain cells with 1,000s of mitochondria (like the many batteries in the large torch). Since the high energy organs require so many mitochondria within their cells, they are usually the first to be affected in mitochondrial disease.
Mitochondria are extremely complex little organelles and each one requires over 1,400 genes to create it. Mitochondrial disease (‘mito disease’ for short) is due to a fault in one or more of the genes that make up the mitochondria. The ratio of healthy to unhealthy mitochondria can also vary greatly from cell to cell and organ to organ.
Mitochondrial disease can have many presentations because mitochondria are located everywhere within the body.
Some mitochondrial disorders might only affect a single organ, such as the eye in Leber hereditary optic neuropathy (LHON), but most involve multiple organs, with the nerves and muscles being affected the most. Many affected individuals will present with a number of features that fall into a discrete subgroup, such as the Kearns-Sayre syndrome (KSS), chronic progressive external ophthalmoplegia (CPEO), mitochondrial encephalomyopathy with lactic acidosis and stroke-like episodes (MELAS), myoclonic epilepsy with ragged-red fibres (MERRF), neurogenic weakness with ataxia and retinitis pigmentosa (NARP), or Leigh syndrome (LS). Others subgroups may be named according to the actual defect occurring in the mitochondria, such as COX deficiency or complex I and IV deficiency.
When we think of the symptoms that might present in mitochondrial disease, we need to think of the organ systems that work the hardest and the longest and so require the most energy.
Symptoms may be absent in healthy people who have silent genetic defects, or can be complex and multiple in a person with advanced disease.
Therefore, with so many illnesses looking like mitochondrial disease and mitochondrial disease looking like so many illnesses, how do we know which is which?
These energy level fluctuations can make it difficult for a person to present their case to a practitioner. Symptoms relating to muscles and nerves include weakness (may be intermittent), altered nerve sensation, fainting, temperature regulation problems, low muscle tone, muscle cramping or pain, and recurrent rhabdomyolysis (rapid muscle breakdown).
As our ears and eyes rarely rest, visual loss or blindness, and deafness (often intermittent) are another common feature of mitochondrial disease. The bowel can show symptoms such as gastro-oesophageal reflux (indigestion), delayed gastric emptying (feeling full a lot), constipation, pseudo-obstruction, chronic or recurrent vomiting, and sometimes difficulties in swallowing.
Mitochondrial Disease DiagramThe kidney, heart and liver are often the forgotten high energy organs, but are very significant because their deterioration plays vital roles in the patient’s prognosis.

Diabetes, short stature, an underactive thyroid or parathyroid, excess body hair, exercise intolerance not in proportion to weakness, hypersensitivity to general anaesthetics, and symmetrical fatty lumps in the skin are other symptoms to alert someone of possible mitochondrial disease. To prove the existence of mitochondrial disease in a newly presenting patient is a bit like doing a jigsaw puzzle. Mitochondrial disease is a multi-system illness, so a thorough history and examination of all the organs mentioned above can be done by a GP. Basic investigations such as hearing tests, eye tests, blood tests, etc., can be done straight away.
As mentioned earlier, mitochondrial disease is often referred to as the ‘notorious masquerader’.
Given its elusive nature, mitochondrial disease should be considered in all patients with an atypical presentation, particularly those suggested in the diagram below.
Also, some overlap is beginning to emerge with mainstream illnesses, for example, there is strong evidence that impaired mitochondrial function is important in Parkinson’s disease, diabetes and diabetic complications.
The secondary type is always related to another illness, an illness that has caused the mitochondria to malfunction despite the normal formation of mitochondria initially. Therefore, due to the broad range of presentations and genetic defects of this illness, it can originate at any point in time. Due to the illusive nature of the illness, it is difficult to know the exact incidence of mitochondrial disease.
Not all of these people will develop the illness, and many may develop subtle symptoms that go unnoticed during their lifetime… BUT the risk of developing serious illness in the general population is about 1 in 5,000.
The prognosis and pathway for any mitochondrial patient is hard to predict as it is an illness that can strike many parts of the body to varying degrees. As mentioned earlier, combinations of symptoms differ greatly even in those with exactly the same genetic defect. For children to develop the illness, it means they have a high load of abnormal mitochondria. The general principals rely on the ‘energy balance equation’, that is, to minimise energy losses and to optimise energy gains. The best way to manage ‘energy loss’ is to have adequate rest and to avoid physical stresses such as infections and big temperature changes, as well as emotional stresses.
Optimising ‘energy in’ will require adequate sleep and nutrition, and a balanced and regular exercise program.
To optimise the mitochondrial environment, we often use what is called a ‘mitochondrial cocktail’ which involves a variety of vitamin and cofactor supplementation. Regular visits to a specialist will monitor any progression or changes in the illness and if found early enough, each complication or development can be better managed. Many patients in earlier days were diagnosed with ‘mitochondrial myopathy’ and were sent home without treatment or support.
Doing nothing can worsen the symptoms, increase the level of disability, decrease independency and worsen the prognosis. Because mitochondrial disease is often ‘invisible’, the biggest challenge the patient faces is convincing the doctor there is something wrong.
If a diagnosis is not established it can be difficult for a patient to obtain adequate medical care, healthcare supports and disability services. Mitochondrial disease affects not only the person with the illness but carers and family as well. Mitochondrial symptoms are very often atypical and bizarre and challenge even the best specialist to ‘think outside of the box’.
Mitochondrial disease is one of the most rapidly growing areas of interest in the world of research. There is a great need to raise the profile of mitochondrial disease within the medical and the general community, and this task is a priority for the AMDF. One study found that people who drink green tea, are more likely to have lower total cholesterol than those who do not drink it.
Because green tea proves to be helpful for preventing colon cancer, this would be an added benefit for those with IBD because they are at risk for colon cancer. It is full of useful tips for those who are definitely interested in this specific subject, particularly this very post. If you feel that you have a medical problem, you should seek the advice of your Physician or health care Practitioner.
If the disease has progressed very far, it is very difficult (maybe impossible) to heal it.
The fast food you consume when you don’t have enough time (and this is something that happens quite often these days) has a strong negative impact on the liver. Your body tries to keep a constant supply of glucose for your cells by maintaining a constant glucose concentration in your blood -- otherwise, your cells would have more than enough glucose right after a meal and starve in between meals and overnight. It is a member of the category of diabetes medications called biguanides, medicines that reduce the blood sugar level by lowering the glucose produced by the liver.
It also has been proposed for women who experience gestational diabetes, a condition where level of blood sugar are more than normal at the time of pregnancy. Mitochondria take in sugars and proteins from the food we eat and produce energy called ATP that our bodies use to function properly.
It is an illness that seems to have suddenly appeared and one that most GPs don’t know about. Although medicine continues to advance rapidly, why is this illness not being considered, and why do most sufferers remain undiagnosed or misdiagnosed?
Due to its widespread variety and severity of symptoms, diagnosing mitochondrial disease can be extremely difficult.
The low energy organs might only contain a few mitochondria in their cells, such as the platelets in our blood (like the single battery in the small torch).
These include the brain (using 20% of our total energy), nerves, muscles, eyes, ears, heart, bowels, liver, kidney and pancreas.
Our genes are like the blueprints of our body, dictating exactly how we will be made and how we function. This means a hiccup will occur in the production of mitochondria from the time of conception. This is the main reason why it is so difficult to know where and when and how severely mitochondrial disease can strike each person.
The severity of damage, location, and number of mitochondria affected all have an affect on body functioning. Also, the complex specialisation of mitochondria within each cell causes a dizzying array of symptoms that mitochondrial specialists have come to recognise as characteristic of this illness.
Many combinations of symptoms are also possible, even in those people with exactly the same genetic defect! Overall, like most mitochondrial symptoms, the fatigue will progress over time, although at vastly different rates for different people.
The brain works constantly in the background, monitoring and controlling most of our bodily functions to the point where it requires 20% of our total energy input. Weak eye muscles may present as droopy eyelids (ptosis), and difficulty in moving the eyes together. Nephrotic syndrome, the loss of important electrolytes in ‘leaky kidneys’, heart arrhythmias, heart blocks, cardiomyopathy (large heart), and unexplained liver abnormalities are some of the earlier signs that could lead to the events of kidney, heart or liver failure. Over time as we study and further understand this illusive illness, we will be better equipped to diagnose it.

The collection of pieces and putting them into place begins with the family doctor and progresses to the mitochondrial specialist. The next and more specialised investigations are best done by a mitochondrial specialist or a neurologist with knowledge of the illness, or in the case of children, a metabolic geneticist. Results from the classical muscle biopsy may be misleading so a number of different tissues are now being used as samples. Unfortunately, due to 1,400 genes making up a single mitochondrion and our ability to test only one at a time, this is often unlikely, especially in the lesser known genetic mutations of mitochondrial disease. Therefore, other illnesses are usually investigated long before mitochondrial disease is considered. Disorders such as Alzheimer’s, autism, cardiovascular disease and some forms of cancer have also been associated with mitochondrial dysfunction, as has the process of ageing. Generally, when we talk of mitochondrial diseases, we are usually talking about the primary type.
Worldwide statistics have varied considerably over time, but conservatively, from recent Australian studies, about 1 in 200 people will carry a mitochondrial genetic defect – that equates to nearly 120,000 Australians.
Hence, the illness tends to progress more quickly than in adults and with a poorer prognosis.
Poor mobility, poor coordination, weakness and fatigue can sometimes create a need for a walking frame or a wheelchair. There is also no single treatment for mitochondrial disease and treatment will need to be tailored for each individual.
This reduces stress on the mitochondria and creates the optimal environment for the mitochondria to function.
Now we know a great deal more and the appropriate management of the illness may not only improve the prognosis of the individual, it can also greatly improve their quality of life. There is a medical void that currently exists regarding this illness, partly due to the illusive nature of the illness and partly due to it being relatively new. This can contribute to a progression of symptoms and a risk of inappropriate treatments and management. So carers and family may even be in need of their own support, support they might have to continuously fight for given the poor education about this disease. The AMDF is currently building a collection of resources, including articles, lifestyle advice and details on community and allied health services that can assist people with these daily challenges, big and small. Not just because of the illness itself, but the mitochondria has now been shown to play vital roles in many other illnesses.
This year the AMDF is developing an accurate Australasian-wide patient registry and is coordinating it with other genetic disease patient registries here and around the world. Results from one study suggests that Polyphenols in green tea may block the intestinal absorption of cholesterol and promote it's excretion from the body. Your also very sweet and thoughtful of others who read your website posts which are fantastic. The liver has an average weight of 3,5 pounds and it is 6,4 inches long, 8 inches wide and about 4,5 inches thick which means that this is the largest organ in the system.
For example, when you consume fatty food in large quantities, you also increase the levels of alanine aminotransferase.
The cells take in glucose from the blood and break it down for energy (some cells, like brain cells and red blood cells, rely solely on glucose for fuel).
So, when you have an oversupply of glucose, your body stores the excess in the liver and muscles by making glycogen, long chains of glucose. Insulin is required by almost all of the body's cells, but its major targets are liver cells, fat cells and muscle cells. Either body doesn’t generate plenty of insulin or the cells just ignore insulin in type 2 diabetes. It is also prescribed often for treating a condition associated with insulin resistance or metabolic syndrome.
Mitochondrial disease (mito) is a debilitating and potentially fatal disease that reduces the ability of the mitochondria to produce this energy. As the foetus grows, some mitochondria can divide and grow as normal whilst others will divide and grow abnormally.
So far we know of at least 100 different types of mitochondrial disease with the potential for 100s more. The main neurological symptoms of mitochondrial disease include developmental delays, mental retardation or regression, dementia, seizures (especially atypical or refractory ones), coma, neuro-psychiatric disturbances, atypical cerebral palsy, myoclonus (brief, involuntary muscle twitching), movement disorders, ataxia (poor coordination), migraines and strokes. After considering the possibility of mitochondrial disease it’s then a matter of putting the pieces on the table and turning them up the right way to see them all together! Mitochondrial disease can therefore become a clinical diagnosis (that is, a diagnosis based on symptoms and signs and basic investigations) in the hands of the right specialist. This means that people with mitochondrial disease are often ‘hidden’ in other areas of medicine and this creates frustration and confusion for patients, practitioners and specialists. The older a person is when symptoms occur, the slower the progression and the better the prognosis. A health professional such as a social worker, occupational therapist, speech therapist, and physiotherapist, with their access to a variety of equipment, home aids and services, can help the mitochondrial patient cope better with daily life.
On bad days the person can be extremely ill, making it difficult to return to the original baseline. This disease can be life-threatening and it can seriously affect the health condition of any individual.
This organ can be separated into two basic elements – a small left lobe and a larger right lobe. This is an enzyme that shows liver damage and increases the chances for developing hepatitis C and type 2 diabetes.However, it is not only the poor eating habits that causes problems in the liver – drinking alcohol and other potentially dangerous beverages can also have negative impact on the liver. There are many treatments you can select that depend on your requirements and the level of your disease.
Unlike many diabetes medications, it doesn’t cause weight gain, but it can cause weight loss in patients of type 2 diabetes who are obese or overweight.
When the mitochondria are not working properly, cells begin to die until eventually whole organ systems fail and the patient’s life itself is compromised. Often when mitochondria are well rested, a person can achieve what they want and appear ‘normal’. Because mitochondrial disease is relatively ‘new’ it is unlikely that there will be a clear and distinct family history of the illness.
People who develop symptoms late in life are often not diagnosed at all and will pass away from other unrelated illnesses. There are several liver diseases related to poor drinking habits and one of the deadliest ones is cirrhosis. Allergies with this medicine are also possible, so if you observe any type of reaction, you should call your doctor immediately. However, in times of excess energy loss, high physical stresses or poor energy input, the person may feel extremely fatigued and often struggles to simply get out of bed.
Throat swelling, lip swelling, tongue swelling, face swelling are some of the other serious side effects that should be informed to your doctor.

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    The modern era is getting more and more concerned.


  2. sladkaya

    Their blood glucose levels are are keeping me from really eat nothing that.