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An interpretation of the continuous relationship between LDL-C and CVD Therefore, many men and most women with heart disease have lipid problems other than high total or LDL cholesterol that put them at risk for heart disease. 7.3 160 Relationship between cholesterol and CVD mortality with and without diabetes Statin (LIPID, HPS, CARDS) ? This 56 year-old lady, who recently suffered a TIA, has history of SVT, type 2 diabetes diagnosed 4 years ago, and hypertension treated from then.
An interpretation of the continuous relationship between LDL-C and CVD Therefore, many men and most women with heart. That there is no cut-off cholesterol number below which coronary heart disease cannot develop.
TG and HDL-C levels are inversely correlated –2) TG exchange causes (pro- atherogenic) smaller, denser LDL.


85 CVD events in 35 conventional patients (44%) versus 33 CVD events in 19 intensive patients (24%) FIELD event rate: No longer ”coronary equivalent”, or only if prolonged? This 56 year-old lady, who recently suffered a TIA, has a history of SVT, type 2 diabetes diagnosed 4 years ago, and hypertension treated from that point in time. Urinary ACR is 4.2 HbA1C is 7.3% Which aspect of her risk factor profile is of the greatest concern? A) BP and ACRB) Weight and BMIC) Total and LDL-C D) TG and HDL-CE) Plasma glucose and HbA1C Which aspect of her risk factor profile is most amenable to intervention?
A) BP and ACRB) Weight and BMIC) Total and LDL-C D) TG and HDL-CE) Plasma glucose and Hb A1C Which aspect of her risk factor profile is most amenable to intervention? You replace her Enalapril and Indapamide with a higher dose combination agent and replace her simvastatin with a tolerable dose of the atorvastatin felodipine combination.


A) BP and ACRB) Weight and BMIC) Total and LDL-C D) TG and HDL-CE) Plasma glucose and Hb A1C If refusal to take more than 1 extra tablet was a limitation, what would you do?
Niacin: Efficacy includes LDL-C reduction In patients with diabetes and mixed dyslipidaemia, Niacin has been shown to Increase HDL-C levels 15%-30% Decrease TG levels 15%-50% Have dose-dependent effects on LDL-C levels (up to 40%) Decrease lipoprotein(a) levels by 25% Decrease fibrinogen levels by 14% Decrease Lp-PLA2 by an additional 20% when added to statin therapy Chesney C et al.



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