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Dulaglutide does not increase the risk of major cardiovascular (CV) events in people with type 2 diabetes, research has found.
The meta-analysis evaluated the CV risk in people with the condition who were treated with Dulaglutide in nine randomised safety and efficacy trials. Dulaglutide is a once-weekly glucagon-like peptide-1 receptor agonist that is approved for treatment of type 2 diabetes. Of the 6,010 people who took part in the research, 3,885 were taking Dulaglutide and the remaining 2,125 participants were given placebos. The demographic and baseline CV disease characteristics were similar across groups, according to the paper.
The ongoing CV outcomes study, Researching CV Events with a Weekly Incretin in Diabetes (REWIND), will further assess CV safety of Dulaglutide. The Diabetes Forum - find support, ask questions and share your experiences with 209,001 people.
Long-acting insulin does not increase the risk of acute myocardial infarction - or heart attack - in people with type 2 diabetes, according to new research. The study, conducted by researchers from the Leibniz Institute for Prevention Research and Epidemiology in Bremen, Germany, compared the impact of long-acting insulin analogs with other basal insulin therapies on the risk of heart attack in people with type 2 diabetes. The researchers examined data from 21,501 type 2 diabetes patients who had recently begun to take insulin; some were treated with premixed insulin, some with analog insulin, and some with human neutral protamine Hagedorn (NPH) insulin. The researchers observed no significant increase in heart attack risk from the use of either type of insulin. Find support, ask questions and share your experiences with 209,001 members of the diabetes community. 10 week (free) low-carb education program developed with the help of 20,000 people with T2D and based on the latest research.
The first comprehensive, free and open to all online step-by-step guide to improving hypo awareness. Please note that we are unable to respond back directly to your questions or provide medical advice.

As the fastest growing consumer health information site a€” with 65 million monthly visitors a€” Healthlinea€™s mission is to be your most trusted ally in your pursuit of health and well-being. There is growing research showing our “gut microbiome,” or the bacteria residing in our intestinal tract, influences metabolism, including evidence of contrasting bacterial profiles in obese individuals versus lean. A case-control study, by design, compares a group of patients who have a disease (in this study, the type 2 diabetics) to a group who does not have the disease (the controls, who do not have diabetes).
It must be noted that case-control studies are not only retrospective, but also observational and have many limitations. The Upstream blog is managed by students in the Interdisciplinary Health Communication (IHC) program at the University of North Carolina at Chapel Hill.
Type 2 diabetes, or non-insulin-dependent diabetes as it was formerly known, is characterized by insulin resistance; that is, although a sufferer may produce enough insulin, body cells are resistant to its effects. The development of type 2 diabetes starts with a fully compensated insulin-resistant state, which then progresses to imaired glucose tolerance (IGT) and then to full-blown type 2 diabetes(Figure 1). Obesity and type 2 diabetes are closely linked; many patients with type 2 diabetes are obese.
When combined with poor circulation, neuropathy can result in diabetic foot ulcers and leg infections.
Type 2 diabetes is associated with clustering of coronary risk factors and 60% to 80% of patients have hypertension. Diabetes is associated with damage to the small blood vessels in the retina leading to vision loss. Both of which will support, guide, and inspire you toward the best possible health outcomes for you and your family. Now, a recent study in the Journal of Endocrinology and Metabolism examines the association between antibiotic use and type 2 diabetes in a Danish population-based case-control study. Looking back, researchers compare the frequency of exposure to a risk factor (like the use of antibiotics) to determine the relationship and estimate the “odds” of getting the disease based on exposure. The association between antibiotic use and development of type 2 diabetes may “represent an increased demand for antibiotics from increased risk of infections in patient with yet-undiagnosed diabetes.” However, these preliminary findings call for future investigations regarding the long-term effects of antibiotic use on the gut microbiome, metabolism, and risk of type 2 diabetes.

Antibiotics have been related diverse adverse effects, and I hope the relationship between antibiotics and type 2 diabetes will be clarified in the near future. However, we reserve the right to delete any comments that threaten, demean, or decrease the civility of discussion. The most common type of diabetes, it is most frequently found in people over the age of 40, although the incidence among youth is rising dramatically. Even if a patient is not obese as such, type 2 diabetes patients generally have an increased amount of visceral fat. Increased incidence of non-insulin dependent diabetes mellitus among Japanese school children correlates with an increased intake of animal protein and fat.
This study in Denmark found that the odds ratio associating type 2 diabetes with exposure (or use of) antibiotics was 1.53, suggesting patients with type 2 diabetes are overexposed to antibiotics prior to their diabetes diagnosis (compared to control subjects without diabetes).
Prescribing antibiotics too often has also been a critical problem in quite many countries.
The Pima tribe in Arizona has an incidence of type 2 diabetes that is 19 times higher than that of the white population. Obesity is thought to exacerbate insulin resistance, and over the last few years it has been suggested that cytokines could play a role in the pathophysiology of obesity and insulin resistance.
More accessible information and education is needed to make a good medicine be helpful, not harmful.
In addition, defects in genes that reduce the activity of β-3-adrenergic receptor, which is found in visceral fat cells, have been found. This defect results in a slowdown in metabolism and an increase in obesity and has a very high incidence in type 2 diabetes and obesity. Additional gene defects considered to be significant include those affecting the FABP2 gene and the lipoprotein lipase.

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