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01.10.2013

Subcutaneous fat function, quick workouts for abs - Test Out

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Regardless of gender, diet alone results in more internal fat loss (and less surface fat loss). Hopefully, these basic facts about fat have given you a better idea of what body fat is and how you can lose body fat – but not necessarily have any immediate change in appearance. Endocrine function of the adipocyte can be divided into proteins and enzymes involved in steroid metabolism. The adipocyte-derived proteins with endocrine function are summarized in the figure to the left.
With the combination of specific receptors and production of endocrine and steriodogenic enzymes, the adipocyte performs specific functions in metabolism. Different adipose deposits may function differently as to substances produced and affect on various tissue. The primary function of leptin is to increase satiety and energy expenditure through action on the hypothalamus.
TNF alpha is secreted more from the subcutaneous adipose deposits than from the visceral deposits and may be dependent on regional fat mass. IL-6 primary function is to increase insulin resistance at the insulin receptor or insulin signaling pathway in hepatic, muscle and adipose tissue. Adiponectin is found more in the subcutaneous adipose deposits than in the visceral deposits. Adipsin is found more in the subcutaneous adipose deposits than in the visceral deposits. Resistin is 15 times greater in the visceral adipose deposits than in the subcutaneous deposits.


When normal-weight people gain modest amounts of fat, they do so by enlarging the fat cells in their upper body subcutaneous fat depot. But in the last fifteen years or so, scientists are discovering that fat is much more complicated (and clever) than we tend to believe.  Fat is not a passive repository but an active organ that performs a variety of essential functions, functions we are just beginning to understand. Endocrinologists recently discovered that fat is the largest endocrine organ in the body.  Like the pituitary gland and the thyroid gland, fat is responsible for sending and receiving certain key hormones.
But cellulite differs from normal fat in other ways, too.  In our interview with world-renowned lymphatic specialist Dr. You have two types of fat storage, one is subcutaneous fat (beneath the skin) and the other one is internal fat (also known as visceral fat) which mains function is to protect your internal organs.
For instance, if the last place you see fat is your chest, then it will be the first to go when you burn fat. For example, leptin is produced more in the subcutaneous fat than in the visceral fat; whereas, IL-6 is produced more in the visceral fat than in the subcutaneous. Visceral adipose tissue secretes endocrine substances into the portal system providing direct access to the liver; whereas subcutaneous adipose secretes substances directly into the systemic circulation.
Jensen explains: "For example, men and women with small visceral fat adipocytes have a much greater tendency to store fat in their visceral depot, probably because the levels of proteins and enzymes related to fat storage are quite high in these fat cells. In contrast, gain of lower body fat is largely the result of increasing the number of fat cells. Jensen adds: "One of the recent hypotheses regarding why some people become metabolically ill when they become obese and others do not relates to dysfunctional storage and release of fatty acids by adipose tissue. Oz describes cellulite as marshmallowy fat cells that swell together and bulge up underneath the skin, creating that unsightly dimpling effect.  With this understanding of cellulite, the obvious treatment option is to decrease the size of the fat cells by eating less or burning more.


These hormones work to relay important messages between the fat and other organs, including the brain.  Clearly our perception of fat needs to be adjusted. For more than 20 years, we've been aware that persons with a preponderance of visceral (omental and mesenteric) fat and upper body fat are at greater risk for the metabolic complications of obesity. One possible explanation is that those persons with excess visceral fat also take up and release too many fatty acids from the visceral fat.
However, as omental and mesenteric fat cells (and thus visceral fat mass) become larger, most of the proteins and enzymes related to fat storage are dramatically suppressed and the cells become resistant to fat storage.
When adipose tissue releases excessive amounts of fatty acids or cannot store them effectively, the fatty acids accumulate in so-called ectopic sites, such as visceral fat, muscle, liver, heart and islet cells. In these tissues, fatty acids can accumulate to excessive amounts in ceramides, diacylglycerols and long-chain acyl coenzyme A's. Excess FFA release from visceral fat could disproportionately affect hepatic metabolic function because the venous drainage of omental and mesenteric fat is into the portal vein — the so-called portal hypothesis. This finding refuted the long-held belief that adult humans do not develop new fat cells but are instead stuck with those that developed in their adolescence. These fatty acid-containing molecules can serve a signaling function within cells and as such may cause insulin resistance and tissue dysfunction. We recently reported that adults do not lose these new leg fat cells when they lose weight.



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Comments to “Subcutaneous fat function”

  1. PLAGIAT_HOSE:
    Rank among the least important idea that fat is going to make.
  2. HULIGANKA:
    You are trying to burn the and that's what was causing her days before.